| 14411. |
- Lindström, Peter, 1970-
(författare)
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Prästval och politisk kultur 1650-1800
- 2003
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Doktorsavhandling (övrigt vetenskapligt/konstnärligt)abstract
- The present dissertation focuses on the appointment of clergy in the rural areas of Swedish province of Hälsingland during the period 1650-1800. The aim has been to analyse the practical process of clergy appointments as well as to discuss the development of the political culture of the said period in the way that it was reflected through the actions of the actors involved.The discussion concentrates on two aspects of the political culture, firstly on what kind of influence the parishes had in regard to the clergy appointments, secondly what the actions of the various actors in this context tell us about the local political culture. The study shows that the parishes did indeed assert their rights in connection with the clergy appointments. It is also shown that the parishes exerted a certain influence in terms of which clergyman to promote, but that this influence seems to have been limited to conditions dictated by the authorities.The clergy election reforms effected in the 1730's brought a significant change to the local political culture. Whereas the earlier legislation assumed that decisions were made unanimously, the elective reforms of the 1730's inaugurated the majority principle as election method, with the result that each voting parish member was now regarded as an independent actor. The investigation of the practice of clergy appointment in Hälsingland shows that the opinions reflected in the unanimous parish demands raised prior to the 1730's reforms primarily belonged to the most leading actors of the community. However, through the formalising of the decision-making procedure, the reforms made way for a broader participation in that process.All farm owners were entitled to vote in the clergy elections, which means that also women farm owners - primarily widows - had the right to vote. Nevertheless, as shown in the present study, the political culture was not as gender neutral as the legislation, seeing that, relatively speaking, the widows voted through proxies to a considerably larger extent than did the men.
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| 14412. |
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| 14413. |
- Lindström, Sten
(författare)
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A Note on Physicalism and Supervenience
- 2002
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Ingår i: Physicalism, Consciousness, and Modality. - : Department of Philosophy and Linguistics, Umeå. ; , s. 125-132
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Bokkapitel (övrigt vetenskapligt/konstnärligt)
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| 14414. |
- Lindström, Sten, 1945-
(författare)
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Challenges to Neologicism
- 2007
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Konferensbidrag (övrigt vetenskapligt/konstnärligt)abstract
- The period in the foundations of mathematics that started in 1879 with the publication of Frege's Begriffsschrift and ended in 1931 with Gödel's Über formal unentscheidbare Sätze der Principia Mathematica und verwandter Systeme can reasonably be called the classical period. It saw the development of three major foundational programmes: the logicism ofFrege, Russell and Whitehead, the intuitionism of Brouwer, and Hilbert's formalist and prooftheoretic programme. In this period, there were also lively exchanges between the various schools culminating in the famous Hilbert-Brouwer controversy in the 1920s. The state of the foundational programmes at the end of the classical period is reported in the papers byCarnap, Heyting and von Neumann (Cf. Benacerraf and Putnam (1983)) from the Conference on Epistemology of the Exact Sciences in Königsberg 1930. This was the very same symposium at which Gödel announced his First Incompleteness Theorem.In this presentation I will concentrate on various varieties of Logicism inspired by Frege's original version which was outlined informally in his Grundlagen der Arithmetik (1884) and presented in formal detail in his Grundgesetze der Arithmetik (1893/1903). I will discuss the motivation behind the logicist programme and the severe difficulties that it faces. In view of these difficulties it is tempting to pronounce the "Death of Logicism"; and obviously the programme cannot be pursued in anything like its original form. The original goal of showing that substantial mathematical theories can be based conceptually, epistemologically, andontologically solely on something that deserves the label “logic” cannot—as far as I can see—be achieved. However, there are close connections between logic and mathematics that should be explored. Much can be learned both philosophically and technically from consideringvarious logicist and neologicist programmes. Moreover, Logicism very much resembles the boar Särimner that according to Nordic mythology was slaughtered every night and eaten—just to be brought back to life the next day.
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| 14415. |
- Lindström, Sten, 1945-
(författare)
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Church-Fitchs argument än en gång, eller: vem är rädd för vetbarhetsparadoxen?
- 2017
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Ingår i: Från Skaradjäkne till Uppsalaprofessor. - Uppsala : Department of Philosophy, Uppsala University. - 9789150626421 ; , s. 160-171
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Bokkapitel (övrigt vetenskapligt/konstnärligt)abstract
- Enligt ett realistiskt synsätt kan ett påstående vara sant trots att det inte ens i princip är möjligt att veta att det är sant. En sanningsteoretisk antirealist kan inte godta denna möjlighet utan att accepterar en eller annan version av Dummetts vetbarhetsprincip:(K) Om ett påstående är sant, så måste det i princip vara möjligt att veta att det är sant.Det kan dock förefalla rimligt, även för en antirealist, att gå med på att det kan finnas sanningar som ingen faktiskt vet (har vetat, eller kommer att veta) är sanna. Man kan därför tänka sig att en antirealist skulle acceptera principen (K) utan att därför gå med på den till synes starkare principen:(SK) Om ett påstående är sant, så måste det faktiskt finnas någon som vet att det är sant.Ett mycket omdiskuterat argument – som ytterst går tillbaka till Alonzo Church, men som först publicerades i en uppsats av Frederic Fitch i Journal of Symbolic Logic 1963 – tycks emellertid visa att principen (K) implicerar principen (SK).I uppsatsen diskuterar jag några olika sätt att undgå Church-Fitch paradoxala slutsats. Ett tillvägagångssätt är att ersätta kunskapsoperatorn med en hierarki av kunskapspredikat. Ett annat är baserat på distinktionen mellan faktisk och potentiell kunskap och ett förkastande av den vanliga modallogiska formaliseringen av principen (K). Den senare typen av lösning betraktas både från ett realistiskt och ett icke-realistiskt perspektiv. Utifrån denna analys kommer jag fram till slutsatsen att vi, vare sig vi är realister eller antirealister rörande sanning, kan sluta oroa oss för vetbarhetsparadoxen och ändå uppskatta Church-Fitchs argument.
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| 14416. |
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| 14417. |
- Lindström, Sten
(författare)
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Frege’s paradise and the paradoxes
- 2003
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Ingår i: A Philosophical Smorgasbord. - : Department of Philosophy, Uppsala. ; , s. 362-
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Bokkapitel (övrigt vetenskapligt/konstnärligt)
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| 14418. |
- Lindström, Susanne, 1962-
(författare)
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Försonad mångfald
- 2005
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Ingår i: Olikhetens paradigm. - Lund : Studentlitteratur. - 9144043848 - 9789144043845
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Bokkapitel (övrigt vetenskapligt/konstnärligt)
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| 14419. |
- Lindström, Sara, 1979-
(författare)
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Genetic variation and prostate cancer : population-based association studies in Sweden
- 2007
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Doktorsavhandling (övrigt vetenskapligt/konstnärligt)abstract
- Prostate cancer constitutes the most common malignancy and the most common cause of cancer‐related death in Swedish men. A large body of evidence suggests that inherited genetic variants contribute to both development and progression of prostate cancer. The aim of this thesis is to identify genetic variants that alter prostate cancer risk and progression. All papers included in this thesis are based on a Swedish population‐based case‐control study (CAPS) comprising 2,965 incident prostate cancer cases and 1,823 controls.In paper I, we investigated if genetic variants in the E‐cadherin gene altered prostate cancer risk. Seven haplotype tagging SNPs(tagSNPs) were selected and genotyped in CAPS and families with hereditary prostate cancer. We confirmed association of a promoter SNP rs16260 previously reported to increase risk of hereditary prostate cancer (OR: 2.6; 95% CI: 1.6‐4.3) for homozygous ‘A’ carriers.In paper II, we assessed 46 polymorphisms earlier reported to be associated with prostate cancer risk. Six polymorphisms in five different genes were replicated. Interestingly, three of these genes were involved in the androgen biosynthesis.In paper III, we followed up on the results from paper II by genotyping 23 tagSNPs located in the hormone regulating genes AR, CYP17 and SRD5A2. Multiple SNPs and haplotypes were associated withprostate cancer risk, especially in the AR gene. Combining risk alleles from all genes revealed a substantial risk increase for each additional allele carried (OR: 1.12; 95% CI: 1.1‐1.2, P=0.00009).In paper IV, we collected information about cause of death for all case patients in CAPS. At time of follow‐up 300 study subjects were deceased from prostate cancer. We assessed AR, CYP17 and SRD5A2 variants for association with lethal prostate cancer and found overall no association. However, one AR promoter SNP was associated with an increased risk of dying from prostate cancer amongst men who received palliative hormonal therapy as primary treatment.In paper V, we assessed common genetic variation at the ERG locus for association between prostate cancer risk and survival. ERG is recognized as a protooncogene frequently overexpressed in prostate cancer. A total of 21 tagSNPs in the 5’ region of ERG were genotyped. There was no correlation between ERG SNPs and prostate cancer risk but common genetic variation located approximately 100,000 basepairs upstream of ERG was significantly associated with prostate cancer specific survival.In summary, our results suggest that common genetic variation in Ecadherin alters prostate cancer risk in Swedish men with a positive family history of prostate cancer. Moreover, common genetic variation in the androgen‐related genes AR, CYP17 and SRD5A2 affects the risk of developing prostate cancer but is unlikely to alter prostate cancer progression. However, genetic variants in AR may affect hormonal therapy response. Finally, ERG polymorphisms are associated with prostate cancer‐specific death but are not likely to play a role in prostate cancer development.
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| 14420. |
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