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41471.
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41472.
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41473.
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41474.
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41475.
  • Laitinen, Kerstin, 1930- (creator_code:aut_t)
  • Begärets irrvägar : existentiell tematik i Stig Dagermans texter
  • 1986
  • swepub:Mat_doctoralthesis_t (swepub:level_scientificother_t)abstract
    • The aim of this s tudy is to investigate the three great existential questions—anxiety, love and death—in the works of Stig Dagerman. Emphasis is placed on the novels Ormen [The Snake] (1945), De dömdas ö [The Island of the Doomed ] (1946), Bränt barn [A Burnt Child] (1948), and Bröllopsbesvär [Wedding Worries] (1949), as well as the play Den dödsdömde [The Condemned] (1948). To this end, interpretation is used in the sense given to it by Paul Ricoeur—interpretation that leads to an understanding of a double meaning. In the present case, this means that the texts' symbolic level is revealed and accorded as great an importance as the story at the surface, manifest level .The theme of anxiety is most apparent in the first novel, while the question of eroticism is central to De dömdas ö and Bränt barn. Each novel expresses a pessimistic view of erotic love as a realizable possibility. Death, which has concrete, motivational and abstract aspects , occurs in every text. Murder and suicide interact with inner, spiritual deadness. This state means acquiring a lowered or threatened inner vitality in which the psyche becomes "icebound". Inner deadness is a form of anxiety.Analysis reveals that anxiety, the complications of love and the problem of death are related to a mother figure. The mother figures in the texts are alternately the objects of destructive hatred and excessive love. Behind these mother figures is concealed the experience of a mother who has betrayed, and who is there fore the object of both aggression and longing. This longing, according to Freudian-Lacanic theory, is expressed in a desire for the mother's body, i.e. a longing fo r the lost unity with the mother in original symbiosis. Thus, the mother figure in the texts also has mythical dimensions—Mother and death are in a sense equivalent. To die is to return to the mother, but death, or the proximity of death thus also provides the opportunity for a symbolic rebirth. Both the proximity of concrete death—as in thwarted suicide attempts—and a symbolic death — as in the form of extreme self-degradation—give rise to an increased feeling of life, if only for a moment.The psychologist Robert Jay Lifton has shown that murder can be a conscious step in a process of self- destruction. Murder is in this case a phase in a search for revitaliza-tion, which is thus the ultimate aim of suicide. This phenomenon appears on the symbolic level in Dagermans works, especially in the play Den dödsdömde. The interpretation reveals that the existential question that dominates Dagerman's texts is the struggle against the threat of inner deadness. The fixation with death in the texts is, in the final analysis , an expression of a desire to live.
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41476.
  • Laitinen, Teresa, et al. (creator_code:aut_t)
  • A Key Role for Apoplastic H2O2 in Norway Spruce Phenolic Metabolism
  • 2017
  • record:In_t: Plant Physiology. - : American Society of Plant Biologists. - 0032-0889 .- 1532-2548. ; 174:3, s. 1449-1475
  • swepub:Mat_article_t (swepub:level_refereed_t)abstract
    • Apoplastic events such as monolignol oxidation and lignin polymerization are difficult to study in intact trees. To investigate the role of apoplastic hydrogen peroxide (H2O2) in gymnosperm phenolic metabolism, an extracellular lignin-forming cell culture of Norway spruce (Picea abies) was used as a research model. Scavenging of apoplastic H2O2 by potassium iodide repressed lignin formation, in line with peroxidases activating monolignols for lignin polymerization. Time-course analyses coupled to candidate substrate-product pair network propagation revealed differential accumulation of low-molecular-weight phenolics, including (glycosylated) oligolignols, (glycosylated) flavonoids, and proanthocyanidins, in lignin-forming and H2O2-scavenging cultures and supported that monolignols are oxidatively coupled not only in the cell wall but also in the cytoplasm, where they are coupled to other monolignols and proanthocyanidins. Dilignol glycoconjugates with reduced structures were found in the culture medium, suggesting that cells are able to transport glycosylated dilignols to the apoplast. Transcriptomic analyses revealed that scavenging of apoplastic H2O2 resulted in remodulation of the transcriptome, with reduced carbon flux into the shikimate pathway propagating down to monolignol biosynthesis. Aggregated coexpression network analysis identified candidate enzymes and transcription factors for monolignol oxidation and apoplastic H2O2 production in addition to potential H2O2 receptors. The results presented indicate that the redox state of the apoplast has a profound influence on cellular metabolism.
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41477.
  • Laitman, Yael, et al. (creator_code:aut_t)
  • Haplotype analysis of the 185delAG BRCA1 mutation in ethnically diverse populations
  • 2013
  • record:In_t: European Journal of Human Genetics. - London : Nature Publishing Group. - 1018-4813 .- 1476-5438. ; 21:2, s. 212-216
  • swepub:Mat_article_t (swepub:level_refereed_t)abstract
    • The 185delAG* BRCA1 mutation is encountered primarily in Jewish Ashkenazi and Iraqi individuals, and sporadically in non-Jews. Previous studies estimated that this is a founder mutation in Jewish mutation carriers that arose before the dispersion of Jews in the Diaspora ~2500 years ago. The aim of this study was to assess the haplotype in ethnically diverse 185delAG* BRCA1 mutation carriers, and to estimate the age at which the mutation arose. Ethnically diverse Jewish and non-Jewish 185delAG*BRCA1 mutation carriers and their relatives were genotyped using 15 microsatellite markers and three SNPs spanning 12.5 MB, encompassing the BRCA1 gene locus. Estimation of mutation age was based on a subset of 11 markers spanning a region of ~5 MB, using a previously developed algorithm applying the maximum likelihood method. Overall, 188 participants (154 carriers and 34 noncarriers) from 115 families were included: Ashkenazi, Iraq, Kuchin-Indians, Syria, Turkey, Iran, Tunisia, Bulgaria, non-Jewish English, non-Jewish Malaysian, and Hispanics. Haplotype analysis indicated that the 185delAG mutation arose 750-1500 years ago. In Ashkenazim, it is a founder mutation that arose 61 generations ago, and with a small group of founder mutations was introduced into the Hispanic population (conversos) ~650 years ago, and into the Iraqi-Jewish community ~450 years ago. The 185delAG mutation in the non-Jewish populations in Malaysia and the UK arose at least twice independently. We conclude that the 185delAG* BRCA1 mutation resides on a common haplotype among Ashkenazi Jews, and arose about 61 generations ago and arose independently at least twice in non-Jews.
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41478.
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41479.
  • Lajus, D., et al. (creator_code:aut_t)
  • The implementation of Marine Stewardship Council (MSC) certification in Russia : achievements and considerations
  • 2018
  • record:In_t: Marine Policy. - : Elsevier. - 0308-597X .- 1872-9460. ; 90, s. 105-114
  • swepub:Mat_article_t (swepub:level_refereed_t)abstract
    • The Marine Stewardship Council (MSC) certification program in Russia is now well established and, in addition to fishery clients and stakeholders, involves environmental NGOs and experts familiar with the local management system. The present study aims to analyze the current status of the program and constitutes the first study covering all Russian MSC certifications. Based on certification reports and twenty semi-structured interviews with stakeholders, it was shown that problems with certification vary among fisheries. The most advanced in terms of management are the Barents Sea codfish fisheries, which are co-managed by Russia and Norway. The main concern of these fisheries is the use of bottom trawls, which may seriously affect bottom communities. The Alaska pollock fishery in the Sea of Okhotsk experienced serious pressure from rival fisheries during the certification process. In the Far East, interviewees dealing with the salmon fisheries note a high level of illegal, unreported and unregulated (IUU) fishing and insufficient scientific data for comprehensive stock assessment. For small-scale inland perch fisheries from the central part of the country, recreational and illegal fishing are important problems that are difficult to quantify. Many interviewees repeatedly mentioned communication issues, difficulties with access to scientific and management information, and the overall complexity of the MSC certification process. The study shows that important preconditions to expanding certification are making the process manageable for export-oriented companies and developing a national market for sustainable seafood.
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41480.
  • Lakehal, Abdellah, et al. (creator_code:aut_t)
  • A DAO1-Mediated Circuit Controls Auxin and Jasmonate Crosstalk Robustness during Adventitious Root Initiation in Arabidopsis
  • 2019
  • record:In_t: International Journal of Molecular Sciences. - : MDPI. - 1661-6596 .- 1422-0067. ; 20:18
  • swepub:Mat_article_t (swepub:level_refereed_t)abstract
    • Adventitious rooting is a post-embryonic developmental program governed by a multitude of endogenous and environmental cues. Auxin, along with other phytohormones, integrates and translates these cues into precise molecular signatures to provide a coherent developmental output. Auxin signaling guides every step of adventitious root (AR) development from the early event of cell reprogramming and identity transitions until emergence. We have previously shown that auxin signaling controls the early events of AR initiation (ARI) by modulating the homeostasis of the negative regulator jasmonate (JA). Although considerable knowledge has been acquired about the role of auxin and JA in ARI, the genetic components acting downstream of JA signaling and the mechanistic basis controlling the interaction between these two hormones are not well understood. Here we provide evidence that COI1-dependent JA signaling controls the expression of DAO1 and its closely related paralog DAO2. In addition, we show that the dao1-1 loss of function mutant produces more ARs than the wild type, probably due to its deficiency in accumulating JA and its bioactive metabolite JA-Ile. Together, our data indicate that DAO1 controls a sensitive feedback circuit that stabilizes the auxin and JA crosstalk during ARI.
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