| 31. |
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| 32. |
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| 33. |
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| 34. |
- Malmgren, Helge, 1945
(författare)
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Immediate Knowledge. A study in G.E. Moore's epistemology
- 1983
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Bok (övrigt vetenskapligt/konstnärligt)abstract
- In G.E. Moore's lectures from 1910–11, "Some Main Problems of Philosophy", it is suggested that we sometimes know immediately such facts as, e.g., 'This pencil exists'. In the present book, the author traces the fate of that idea in Moore's well-known essays "A Defence of Common Sense" and "Proof of an External World". In parallell with this interpretative work, a number of distinctions of importance for the contemporary discussion about immediate knowledge are also put forward.
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| 35. |
- Malmgren, Helge, 1945
(författare)
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Immediate knowledge of other minds
- 1976
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Ingår i: Theoria. - 0495-4548. ; 42, s. 189-205
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Tidskriftsartikel (övrigt vetenskapligt/konstnärligt)
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| 36. |
- Malmgren, Helge, 1945
(författare)
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In What Sense, if Any, do Hippocampal “Time Cells” Represent or Encode Time?
- 2015
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Ingår i: The Brain's Networks: Which are they, and what do they do? September 18-20, 2015, Gothenburg, Sweden.
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Konferensbidrag (övrigt vetenskapligt/konstnärligt)abstract
- The first part of this poster is a conceptual discussion of the notion of a time cell. It is argued that some neural structures must encode time in a more fundamental way than the time cells do. I introduce the term “i-timecode” for this fundamental representation. The second part places the conceptual discussion in a context of certain wellknown empirical findings in humans. The third and last part presents two main alternative approaches when formulating a theory of the i-timecode. These are: mapping time to space, and transforming time to the frequency domain (or a mixed domain). I also present a version, in gross outlines, of the second approach. In it, neural oscillations are given a role that is quite different from those which they, in the contemporary discussion of time cells in the hippocampus, have been hypothesized to play.
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| 37. |
- Malmgren, Helge, 1945
(författare)
-
Inlärning och minne i neurala nätverk
- 2007
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Bok (övrigt vetenskapligt/konstnärligt)abstract
- Bokens fokus är teorin om artificiella neurala nätverk (ANN), men den redogör också för relevanta psykologiska och neurovetenskapliga fakta och för den filosofiska diskussionen om begreppen information och kognition.
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| 38. |
- Malmgren, Helge, 1945
(författare)
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Inlärning och minne i neurala nätverk
- 2020
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Bok (övrigt vetenskapligt/konstnärligt)abstract
- Detta är den andra, fortfarande preliminära, upplagan av en bok som gavs ut 2007. Dess fokus är teorin om artificiella neurala nätverk (ANN), men den redogör också för relevanta psykologiska och neurovetenskapliga fakta och för den filosofiska diskussionen om begreppen information och kognition.
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| 39. |
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| 40. |
- Malmgren, Helge, 1945
(författare)
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Is schizophrenia a brain disease? : Är schizofreni en hjärnsjukdom?
- 2006
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Ingår i: Philosophy, Psychiatry and the Neurosciences: 9th International Conference on Philosophy, Psychiatry and Psychopathology. Leiden, June 29, 2006.
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Konferensbidrag (övrigt vetenskapligt/konstnärligt)abstract
- Recent advances in brain imaging techniques have made it possible to discern changes in brain morphology and function in several different psychiatric disorders. Should such results be taken as evidence for an “organic” etiology of these disorders? A well-known objection says that the visible physical changes could be effects instead of causes. An even more important possibility, which is usually overlooked, is that the observable anatomic or physiological changes may be the immediate biological substrate of the psychiatric condition. Even the paradigmatically “psychogenic” condition which consists in the possession of learned knowledge has such a substrate. This substrate has been observed by imaging methods, but no one (I hope) would draw the conclusion that the possession of learned knowledge is not a mental state, or that it is not mentally caused. Similarly, the observation that, e.g., schizophrenia is accompanied by observable changes of the brain must not be taken as proof that schizophrenia is not a mental disorder, or that the non-hereditary etiological component in schizophrenia (which accounts for some 50% of the variance) is “organic” in nature. For the latter conclusion, one would need support from independent findings of such exogenic or endogenic biological and physical events in the patient’s history as could lead to organic lesions. Notably, the evidence for an overrepresentation of such events in the history of schizophrenic patients is scarce. My conclusion is that the total evidence is quite compatible with the possibility that schizophrenia is a mental disorder with an strong psychogenic component.
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