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Common origin of the Val30Met mutation responsible for the amyloidogenic transthyretin type of familial amyloidotic polyneuropathy.
- Ohmori, H (author)
- Ando, Y (author)
- Makita, Y (author)
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- Onouchi, Y (author)
- Nakajima, T (author)
- Saraiva, MJM (author)
- Terazaki, H (author)
- Sobue, G (author)
- Nakamura, M (author)
- Yamaizumi, M (author)
- Munar-Ques, M (author)
- Inoue, I (author)
- Uchino, M (author)
- Hata, A (author)
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- (creator_code:org_t)
- 2004
- 2004
- English.
- In: Journal of medical genetics. - 1468-6244. ; 41:4, s. e51-
- Journal article (peer-reviewed)
Subject headings
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Keyword
- Amino Acid Substitution
- Amyloid/*genetics
- Amyloid Neuropathies; Familial/*genetics
- Europe
- Founder Effect
- Gene Frequency
- Haplotypes
- Humans
- Japan
- Microsatellite Repeats
- Mutation; Missense
- Polymorphism; Single Nucleotide
- Prealbumin/*genetics
Publication and Content Type
- ref (subject category)
- art (subject category)
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- Ohmori, H
- Ando, Y
- Makita, Y
- Onouchi, Y
- Nakajima, T
- Saraiva, MJM
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- Terazaki, H
- Suhr, Ole B
- Sobue, G
- Nakamura, M
- Yamaizumi, M
- Munar-Ques, M
- Inoue, I
- Uchino, M
- Hata, A
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- Journal of medic ...
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- Umeå University
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