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21.
  • Svedendahl, Mikael, et al. (författare)
  • Complete light annihilation in an ultrathin layer of gold nanoparticles.
  • 2013
  • Ingår i: Nano letters (Print). - American Chemical Society (ACS). - 1530-6984. ; 13:7, s. 3053-3058
  • Tidskriftsartikel (refereegranskat)abstract
    • We experimentally demonstrate that an incident light beam can be completely annihilated in a single layer of randomly distributed, widely spaced gold nanoparticle antennas. Under certain conditions, each antenna dissipates more than 10 times the number of photons that enter its geometric cross-sectional area. The underlying physics can be understood in terms of a critical coupling to localized plasmons in the nanoparticles or, equivalently, in terms of destructive optical Fano interference and so-called coherent absorption.
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22.
  • Trovik, Clement, et al. (författare)
  • The Scandinavian Sarcoma Group Central Register : : 6,000 patients after 25 years of monitoring of referral and treatment of extremity and trunk wall soft-tissue sarcoma
  • 2017
  • Ingår i: Acta Orthopaedica. - Taylor & Francis. - 1745-3674. ; s. 1-7
  • Tidskriftsartikel (refereegranskat)abstract
    • Purpose — We wanted to examine the potential of the Scandinavian Sarcoma Group (SSG) Central Register, and evaluate referral and treatment practice for soft-tissue sarcomas in the extremities and trunk wall (STS) in the Nordic countries. Background — Based on incidence rates from the literature, 8,150 (7,000–9,300) cases of STS of the extremity and trunk wall should have been diagnosed in Norway, Finland, Iceland, and Sweden from 1987 through 2011. The SSG Register has 6,027 cases registered from this period, with 5,837 having complete registration of key variables. 10 centers have been reporting to the Register. The 5 centers that consistently report treat approximately 90% of the cases in their respective regions. The remaining centers have reported all the patients who were treated during certain time periods, but not for the entire 25-year period. Results — 59% of patients were referred to a sarcoma center untouched, i.e. before any attempt at open biopsy. There was an improvement from 52% during the first 5 years to 70% during the last 5 years. 50% had wide or better margins at surgery. Wide margins are now achieved less often than 20 years ago, in parallel with an increase in the use of radiotherapy. For the centers that consistently report, 97% of surviving patients are followed for more than 4 years. Metastasis-free survival (MFS) increased from 67% to 73% during the 25-year period. Interpretation — The Register is considered to be representative of extremity and trunk wall sarcoma disease in the population of Scandinavia, treated at the reporting centers. There were no clinically significant differences in treatment results at these centers.
23.
  • Wannberg, Gudmund, et al. (författare)
  • EISCAT_3D - a next-generation European radar system for upper atmosphere and geospace research
  • 2010
  • Ingår i: Radio Science Bulletin. - 1024-4530. ; :332, s. 75-88
  • Tidskriftsartikel (refereegranskat)abstract
    • The EISCAT Scientifi c Association, together with a number of collaborating institutions, has recently completed a feasibility and design study for an enhanced performance research radar facility to replace the existing EISCAT UHF and VHF systems. This study was supported by EU Sixth-Framework funding. The new radar retains the powerful multi-static geometry of the EISCAT UHF, but will employ phased arrays, direct-sampling receivers, and digital beamforming and beam steering. Design goals include, inter alia, a tenfold improvement in temporal and spatial resolution, an extension of the instantaneous measurement of full-vector ionospheric drift velocities from a single point to the entire altitude range of the radar, and an imaging capability to resolve small-scale structures. Prototype receivers and beamformers are currently being tested on a 48-element, 224 MHz array (the "Demonstrator") erected at the Kiruna EISCAT site, using the EISCAT VHF transmitter as an illuminator.
24.
  • Allard, Christina, et al. (författare)
  • Rasbiologiskt språkbruk i statens rättsprocess mot sameby
  • 2015
  • Ingår i: Dagens Nyheter. - 1101-2447.
  • Tidskriftsartikel (populärvet., debatt m.m.)abstract
    • Statens hantering av forskningsresultat i rättsprocessen med Girjas sameby utgör ett hot mot Sverige som rättsstat och kunskapsnation. Åratal av svensk och internationell forskning underkänns och man använder ett språkbruk som skulle kunna vara hämtat från rasbiologins tid. Nu måste staten ta sitt ansvar och börja agera som en demokratisk rättsstat, skriver 59 forskare.
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25.
  • Aminian, Behdad, et al. (författare)
  • GISOO : A virtual testbed for wireless cyber-physical systems
  • 2013
  • Ingår i: Industrial Electronics Society, IECON 2013 - 39th Annual Conference of the IEEE. - IEEE. - 978-147990224-8 ; s. 5588-5593
  • Konferensbidrag (refereegranskat)abstract
    • The increasing demand for wireless cyber-physical systems requires correct design, implementation and validation of computation, communication and control methods. Traditional simulation tools, which focus on either computation, communication or control, are insufficient when the three aspects interact. Efforts to extend the traditional tools to cover multiple domains, e.g., from simulating only control aspects to simulating both control and communication, often rely on simplistic models of a small subset of possible communication solutions. We introduce GISOO, a virtual testbed for simulation of wireless cyber-physical systems that integrates two state-of-the art simulators, Simulink and COOJA. GISOO enables users to evaluate actual embedded code for the wireless nodes in realistic cyber-physical experiments, observing the effects of both the control and communication components. In this way, a wide range of communication solutions can be evaluated without developing abstract models of their control-relevant aspects, and changes made to the networking code in simulations is guaranteed to be translated into production code without errors. A double-tank laboratory experimental setup controlled over a multi-hop relay wireless network is used to validate GISOO and demonstrate its features.
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26.
  • Anantharaman, Devasena, et al. (författare)
  • No causal association identified for human papillomavirus infections in lung cancer
  • 2014
  • Ingår i: Cancer Research. - American Association for Cancer Research. - 0008-5472. ; 74:13, s. 3525-3534
  • Tidskriftsartikel (refereegranskat)abstract
    • Human papillomavirus (HPV) infections have been implicated in lung carcinogenesis, but causal associations remain uncertain. We evaluated a potential causal role for HPV infections in lung cancer through an analysis involving serology, tumor DNA, RNA, and p16 protein expression. Association between type-specific HPV antibodies and risk of lung cancer was examined among 3,083 cases and 4,328 controls in two case-control studies (retrospective) and one nested case-control study (prospective design). Three hundred and thirty-four available tumors were subjected to pathologic evaluation and subsequent HPV genotyping following stringent conditions to detect all high-risk and two low-risk HPV types. All HPV DNA-positive tumors were further tested for the expression of p16 protein and type-specific HPV mRNA. On the basis of the consistency of the results, although HPV11 and HPV31 E6 antibodies were associated with lung cancer risk in the retrospective study, no association was observed in the prospective design. Presence of type-specific antibodies correlated poorly with the presence of the corresponding HPV DNA in the tumor. Although nearly 10% of the lung tumors were positive for any HPV DNA (7% for HPV16 DNA), none expressed the viral oncogenes. No association was observed between HPV antibodies or DNA and lung cancer survival. In conclusion, we found no supportive evidence for the hypothesized causal association between HPV infections and lung cancer. (C) 2014 AACR.
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27.
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28.
  • Baglietto, Laura, et al. (författare)
  • DNA methylation changes measured in pre-diagnostic peripheral blood samples are associated with smoking and lung cancer risk
  • 2017
  • Ingår i: International Journal of Cancer. - 0020-7136. ; 140:1, s. 50-61
  • Tidskriftsartikel (refereegranskat)abstract
    • DNA methylation changes are associated with cigarette smoking. We used the Illumina Infinium HumanMethylation450 array to determine whether methylation in DNA from pre-diagnostic, peripheral blood samples is associated with lung cancer risk. We used a case-control study nested within the EPIC-Italy cohort and a study within the MCCS cohort as discovery sets (a total of 552 case-control pairs). We validated the top signals in 429 case-control pairs from another 3 studies. We identified six CpGs for which hypomethylation was associated with lung cancer risk: cg05575921 in the AHRR gene (p-valuepooled  = 4 × 10(-17) ), cg03636183 in the F2RL3 gene (p-valuepooled  = 2 × 10 (- 13) ), cg21566642 and cg05951221 in 2q37.1 (p-valuepooled  = 7 × 10(-16) and 1 × 10(-11) respectively), cg06126421 in 6p21.33 (p-valuepooled  = 2 × 10(-15) ) and cg23387569 in 12q14.1 (p-valuepooled  = 5 × 10(-7) ). For cg05951221 and cg23387569 the strength of association was virtually identical in never and current smokers. For all these CpGs except for cg23387569, the methylation levels were different across smoking categories in controls (p-valuesheterogeneity  ≤ 1.8 x10 (- 7) ), were lowest for current smokers and increased with time since quitting for former smokers. We observed a gain in discrimination between cases and controls measured by the area under the ROC curve of at least 8% (p-values ≥ 0.003) in former smokers by adding methylation at the 6 CpGs into risk prediction models including smoking status and number of pack-years. Our findings provide convincing evidence that smoking and possibly other factors lead to DNA methylation changes measurable in peripheral blood that may improve prediction of lung cancer risk.
29.
  • Battram, Thomas, et al. (författare)
  • Appraising the causal relevance of DNA methylation for risk of lung cancer
  • 2019
  • Ingår i: International Journal of Epidemiology. - Oxford University Press. - 0300-5771. ; 48:5, s. 1493-1504
  • Tidskriftsartikel (refereegranskat)abstract
    • Background: DNA methylation changes in peripheral blood have recently been identified in relation to lung cancer risk. Some of these changes have been suggested to mediate part of the effect of smoking on lung cancer. However, limitations with conventional mediation analyses mean that the causal nature of these methylation changes has yet to be fully elucidated.Methods: We first performed a meta-analysis of four epigenome-wide association studies (EWAS) of lung cancer (918 cases, 918 controls). Next, we conducted a two-sample Mendelian randomization analysis, using genetic instruments for methylation at CpG sites identified in the EWAS meta-analysis, and 29 863 cases and 55 586 controls from the TRICL-ILCCO lung cancer consortium, to appraise the possible causal role of methylation at these sites on lung cancer.Results: Sixteen CpG sites were identified from the EWAS meta-analysis [false discovery rate (FDR) < 0.05], for 14 of which we could identify genetic instruments. Mendelian randomization provided little evidence that DNA methylation in peripheral blood at the 14 CpG sites plays a causal role in lung cancer development (FDR > 0.05), including for cg05575921-AHRR where methylation is strongly associated with both smoke exposure and lung cancer risk.Conclusions: The results contrast with previous observational and mediation analysis, which have made strong claims regarding the causal role of DNA methylation. Thus, previous suggestions of a mediating role of methylation at sites identified in peripheral blood, such as cg05575921-AHRR, could be unfounded. However, this study does not preclude the possibility that differential DNA methylation at other sites is causally involved in lung cancer development, especially within lung tissue.
30.
  • Bjernestedt, Amanda, et al. (författare)
  • Pupil dilation reflects interference during memory retrieval
  • 2016
  • Konferensbidrag (refereegranskat)abstract
    • We demonstrate that pupillometry can be used to track memory interference independent of explicit responses. Pupil diameter was recordedthroughout encoding and retrieval of words from the same category over 4 trials, causing buildup of proactive interference (PI). In a contrastingcondition, the category was switched on the 4th trial, causing release from interference (RI). Pupil dilation systematically increased for bothconditions as interference built up and retrieval performance declined. Critically, in trial 4 the RI condition resulted in improved retrievalperformance, with significantly smaller pupil dilation than in the PI condition, where performance continued to decline. Principal component analysisrevealed an early dilation peak possibly related to control of interference, and a later component possibly linked to memory search.
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