241. |
- Sulymenko, O., et al.
(författare)
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Ultra-fast logic devices using artificial "neurons" based on antiferromagnetic pulse generators
- 2018
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Ingår i: Journal of Applied Physics. - : AIP Publishing. - 0021-8979 .- 1089-7550. ; 124:15
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Tidskriftsartikel (refereegranskat)abstract
- It has been shown previously that spin-Hall oscillators based on current-driven bi-layered film structures containing an antiferromagnet (AFM) and a normal metal can generate ultra-short (similar to 2 ps) "spike-like" pulses in response to an external current stimulus of a sufficient amplitude, thus operating as ultra-fast artificial "neurons." Here, we report the results of numerical simulations demonstrating that a single AFM "neuron" can perform the logic functions of OR, AND, MAJORITY, or Q-gates, while a circuit consisting of a small number n < 5 of AFM "neurons" can function as a FULL-ADDER or as a dynamic memory loop with variable clock frequency. The clock frequencies of such AFM-based logic devices could reach tens of GHz, which make them promising as base elements of future ultra-fast high-efficiency neuromorphic computing. Published by AIP Publishing.
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242. |
- Voight, Benjamin F., et al.
(författare)
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Twelve type 2 diabetes susceptibility loci identified through large-scale association analysis
- 2010
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Ingår i: Nature Genetics. - : Springer Science and Business Media LLC. - 1061-4036 .- 1546-1718. ; 42:7, s. 579-589
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Tidskriftsartikel (refereegranskat)abstract
- By combining genome-wide association data from 8,130 individuals with type 2 diabetes (T2D) and 38,987 controls of European descent and following up previously unidentified meta-analysis signals in a further 34,412 cases and 59,925 controls, we identified 12 new T2D association signals with combined P < 5 x 10(-8). These include a second independent signal at the KCNQ1 locus; the first report, to our knowledge, of an X-chromosomal association (near DUSP9); and a further instance of overlap between loci implicated in monogenic and multifactorial forms of diabetes (at HNF1A). The identified loci affect both beta-cell function and insulin action, and, overall, T2D association signals show evidence of enrichment for genes involved in cell cycle regulation. We also show that a high proportion of T2D susceptibility loci harbor independent association signals influencing apparently unrelated complex traits.
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