| 1. |
- Molassiotis, Alex, et al.
(författare)
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Nausea and vomiting
- 2006. - 1
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Ingår i: Nursing patients with cancer. - : Elsevier Churchill Livingstone. - 9780443072888 - 0443072884 ; , s. 415-437
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Bokkapitel (övrigt vetenskapligt/konstnärligt)abstract
- This title is directed primarily towards health care professionals outside of the United States. Nurses are in the vanguard of the international fight against cancer. National policies on reducing or eradicating cancer highlight the vital contribution nurses make in its prevention, identification, treatment and management. This book, which is based on the internationally acclaimed Core Curriculum for a Post-Registration Course in Cancer Nursing developed by the European Oncology Nursing Society (EONS), covers all the key areas nurses caring for patients in this demanding specialty will need to consider. Focusing on the needs of the patient, it provides the essential scientific, psychological and sociological information necessary for nurses to provide care that minimises the trauma of cancer, and maximises outcomes for
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| 2. |
- Högberg, Thomas, et al.
(författare)
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Gynekologisk onkologi
- 2008. - 2
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Ingår i: Onkologi. - Stockholm : Liber. - 9789147084012 ; , s. 488-533
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Bokkapitel (övrigt vetenskapligt/konstnärligt)
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| 3. |
- Alao, John Patrick, 1973
(författare)
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G1 Phase cyclins in cancer development and progression
- 2009
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Ingår i: Checkpoint controls and targets in cancer therapy. Cancer drug discovery and development. (Zahid H. Siddik, editor). - New York : Humana Press. - 9781607611776 ; , s. 123-153
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Bokkapitel (övrigt vetenskapligt/konstnärligt)abstract
- Cyclin D1 and cyclin E are rate limiting for progression through G1 and entry into S phase during the cell cycle. Cyclin D1 plays a crucial role in linking favourable environmental conditions with cell cycle progression. Cyclin D1 indirectly induces cyclin E expression and activation which is required for entry into S phase. The deregulated expression of either cyclin would thus greatly impact on a cell’s ability to effectively regulate cell cycle progression. It perhaps not surprising therefore, that both cyclin D1 and cyclin E play important roles in the development and progression of cancer. This chapter explores the roles of these cyclins in cancer. For clarity, the major focus will be on the well characterized roles of cyclin D1 and cyclin E and thus to a lesser extent on other G1 cyclins such as cyclins D2 and D3. This chapter summarizes the evidence linking cyclin D1 and cyclin E to cancer development and progression. The cellular pathways that underlie the deregulated expression of the G1 cyclins in cancer, as well as their oncogenic roles are also discussed. Lastly, this chapter explores the potential of these cyclins to serve as therapeutic targets in cancer therapy.
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| 4. |
- Toma-Dasu, Iuliana, et al.
(författare)
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Influence of acute tumour hypoxia on radiation therapy outcome
- 2005
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Ingår i: AAPM Symposium Proceedings No. 14: Physical, Chemical and Biological Targeting in Radiation Oncology. - Madison, Wisconsin : Medical Physics Publishing. - 9781888340532 ; , s. 111-117
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Bokkapitel (refereegranskat)abstract
- One of the most important factors that influence the outcome of cancer treatment is tumor hypoxia that is caused by a deficient vascular network and an increased interstitial pressure. Tumor hypoxia can be divided into two types depending on the underlying mechanism. Thus, chronic hypoxia is caused mainly by the limited diffusion of oxygen into tissue due to cellular consumption and might also be influenced by a high interstitial pressure, and changes very slowly in time. Acute hypoxia might also appear in tumors due to transient perfusion-related events such as the temporary closure of blood vessels and has a finite lifetime ranging from minutes to hours. The temporal variation of acute hypoxia influences the treatment outcome in a more complex way than the chronic hypoxia. The aim of this study is to simulate the influences of various amounts of acute hypoxia on the treatment outcome. The present model for tumor oxygenation allows the simulation of the two forms of tumor hypoxia. We have studied the reduced tumor oxygenations resulting from the temporary shutting down of a given fraction of tumor blood vessels at every radiation fraction throughout the treatment. The particular effect of acute hypoxia on the treatment outcome was evaluated by calculating the effective tumor control probability with or without acute hypoxia. The results suggest that the presence of acute hypoxia during every treatment fraction reduces the tumor control probability significantly. As consequence, higher and higher doses are needed to achieve a certain level of local control in tumors when the fraction of closed vessels increases. These results therefore show that acute hypoxia has to be taken into consideration for accurate simulations of radiation treatments and that an individual approach for each patient might be desirable for maximum improvement of outcome.
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| 5. |
- Granberg, Dan, et al.
(författare)
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Neuroendocrine tumours
- 2007
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Ingår i: Update on Cancer Therapeutics 2. - : Elsevier, Oxford. ; , s. 41-52
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Bokkapitel (refereegranskat)
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| 6. |
- Granberg, Dan, et al.
(författare)
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Neuroendocrine tumours
- 2006
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Ingår i: Update on Cancer Therapeutics I. - : Elsevier, Oxford. ; , s. 75-84
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Bokkapitel (refereegranskat)
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| 7. |
- Andersson-Engels, Stefan, et al.
(författare)
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Clinical fluorescence imaging
- 2008
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Ingår i: Lasers in Medicine.
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Bokkapitel (övrigt vetenskapligt/konstnärligt)
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| 8. |
- Belting, Mattias, et al.
(författare)
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Proteoglycans as endocytosis receptors for CPPs
- 2007
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Ingår i: Handbook of Cell-Penetrating Peptides, Second Edition. - 9780849350900 - 0849350905 ; , s. 219-219
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Bokkapitel (övrigt vetenskapligt/konstnärligt)
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| 9. |
- Brown, KM, et al.
(författare)
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cDNA Microarrays in Cancer Research.
- 2006
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Ingår i: Cancer: Principles and Practise of Oncology. - 0781774330 ; , s. 13-25
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Bokkapitel (övrigt vetenskapligt/konstnärligt)
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| 10. |
- Börjeson, Sussanne, et al.
(författare)
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Onkologisk omvårdnad
- 2008. - 2. uppl.
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Ingår i: Onkologi. - Stockholm : Liber. - 9789147084012 - 9147084014 ; , s. 232-242
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Bokkapitel (övrigt vetenskapligt/konstnärligt)
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