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Toomadj, Farshad, et al.
(author)
Sensing application of nanocellulose modified with double-walled carbon nanotube and graphitized carbon nanopowder
2011
In: Proceedings of the 22n Micromechanics and Microsystems Technology Europe Workshop, 19-22 June 2011, Tönsberg, Norway. ; , s. B33-
Conference paper (peer-reviewed) abstract
Electrically conductive nanocellulose films were prepared by immersion in well dispersed solutions of double walled carbon nanotubes or graphitized nanopow¬der. These solutions were prepared by using the surfactant cetyltrimethylammonium bromide (CTAB) under heating and stirring conditions which were followed by a short time sonication process. Electrospun cellulose and bacterial cellulose were used as a backbone of these films. Controllable modification of cellulose with conductive agents was performed by treatment with their dispersions for 24 – 72 hours. Electrical conductivities of the resulting films are from 0,009 S cm-1 to 0,395 S cm-1 which is higher or comparable to previously reported results.Some methods have been employed to study the electrical conductivity behavior of samples. The electrical conductance of the films displays a high sensitivity to strain when tensile stress is applied.
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van der Wijngaart, Wouter, et al.
(author)
Valve-less diffuser fluid micropump
1999
In: Gordon Research Conference 1999, Analytical Chemistry, New Hampshire, USA.
Conference paper (peer-reviewed)
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Sörensen Ristinmaa, Amanda, 1992, et al.
(author)
Biochemical characterization of three tannases from Clostridium butyricum
2021
Conference paper (other academic/artistic) abstract
Bark is the outer tissue of the tree, shielding the wood from microbial attack. The compounds present in bark which gives the bark this ability to hamper microbial attack are the secondary metabolites called extractives, such as tannins. Biochemical degradation of tannins is known to be conducted by tannase enzymes (EC 3.1.1.20), which are serine-esterases able to separate gallic acid from the aromatic-sugar complexes. In this study we investigated three tannases from the bacterium Clostridium butyricum, named CbTan1-3, both on model substrates but also on oak bark extract. The enzymes were tested on a range of galloyl ester model substrates and display different substrate preferences, with CbTan1-3 preferring galloyl moiety linked to glucose and CbTan2 not displaying preference towards any model substrate tested in this study. On oak bark extract, the three enzymes act on different molecules, with CbTan2 degrading all identified substrates and CbTan3 and CbTan1 able to degrade similar substrates, however, CbTan3 was also able to liberate additional glucose from the oak bark extract.
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Hogmalm, Anna, 1981, et al.
(author)
Postnatal lung inflammation leads to abnormal lung structure and function in adulthood
2015
In: Pediatric Academic Societies Annual Meeting.
Conference paper (other academic/artistic) abstract
Background: Exposure to chorioamnionitis, mechanical ventilation, and the use of oxygen therapy cause inflammation in the preterm lung. The long-term consequences of neonatal pulmonary inflammation on lung structure and function have been incompletely characterized. We have previously shown that postnatal pulmonary IL-1b expression during the late saccular to alveolar stage of lung development causes lung inflammation and a mild increase in alveolar size and septal wall thickness in infant mice. Objective: To study the consequences of postnatal IL-1b expression in the newborn lung on lung structure and function in adulthood. Methods: To induce IL-1b production in the pulmonary epithelium of transgenic mice during the late saccular to alveolar stage of lung development, doxycycline (0.15 mg) was administered via intraperitoneal injections to transgenic pups and their littermate controls on postnatal days (PN) 0, 0.5, and 1. Lung inflammation, structure and airway reactivity (assessed by flexiVentTM at methacholine concentrations 0-200 mg/ml) were studied on PN 21 and 42. Results: IL-1b expression in the infant mice caused lack of alveolar septation in the lungs of young (PN 21) and adult (PN 42) mice (Fig. 2) and increased airway reactivity in the mice (Fig. 3). Conclusions: Postnatal lung inflammation in newborn mice caused abnormal lung structure and function in adulthood. Lack of alveolar septation in the newborn persisted after IL-1b production was stopped. Neonatal pulmonary inflammation thus has long-term consequences on the lung and may cause respiratory impairment in adulthood.
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