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Träfflista för sökning "WFRF:(Ekman M.) ;pers:(Ekman A)"

Sökning: WFRF:(Ekman M.) > Ekman A

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  • Amerl, M., et al. (författare)
  • Search for nonresonant pair production of Higgs bosons in the Formula Presented final state in pp collisions at Formula Presented with the ATLAS detector
  • 2023
  • Ingår i: Physical Review D. - : American Physical Society. - 2470-0010 .- 2470-0029. ; 108:5
  • Tidskriftsartikel (refereegranskat)abstract
    • A search for nonresonant Higgs boson pair production in the Formula Presented final state is presented. The analysis uses Formula Presented of Formula Presented collision data at Formula Presented collected with the ATLAS detector at the Large Hadron Collider, and targets both the gluon-gluon fusion and vector-boson fusion production modes. No evidence of the signal is found and the observed (expected) upper limit on the cross section for nonresonant Higgs boson pair production is determined to be 5.4 (8.1) times the Standard Model predicted cross section at 95% confidence level. Constraints are placed on modifiers to the Formula Presented and Formula Presented couplings. The observed (expected) Formula Presented constraints on the Formula Presented coupling modifier, Formula Presented, are determined to be Formula Presented (Formula Presented), while the corresponding constraints for the Formula Presented coupling modifier, Formula Presented, are Formula Presented (Formula Presented). In addition, constraints on relevant coefficients are derived in the context of the Standard Model effective field theory and Higgs effective field theory, and upper limits on the Formula Presented production cross section are placed in seven Higgs effective field theory benchmark scenarios. © 2023 CERN, for the ATLAS Collaboration.
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  • Ekman, A. -K., et al. (författare)
  • Systemic Up-Regulation of TLR4 Causes Lipopolysaccharide-Induced Augmentation of Nasal Cytokine Release in Allergic Rhinitis
  • 2012
  • Ingår i: International Archives of Allergy and Immunology. - : S. Karger AG. - 1423-0097 .- 1018-2438. ; 159:1, s. 6-14
  • Tidskriftsartikel (refereegranskat)abstract
    • Background: Allergic rhinitis is a systemic disorder, and it is clinically well recognized that it can be aggravated by infection. Activation of the innate immune system constitutes a critical element in the process. Toll-like receptors (TLRs) comprise a part of the innate immune system, and lipopolysaccharide (LPS)-induced activation of TLR4 represents bacterial-induced interactions in various model systems. The present study examines how TLR2 and TLR4 expression is affected by symptomatic allergic rhinitis, and if LPS added upon allergen affects nasal cytokine release. Methods: In patients with pollen-induced allergic rhinitis and healthy non-allergic volunteers, nasal lavage (NAL), peripheral blood and bone marrow were sampled before and during the pollen season. TLR2 and TLR4 expression was determined flow cytometrically. Changes in the TLR receptor expression pattern were evaluated by a nasal challenge with allergen followed by LPS, or vice versa. Symptoms along with cells and cytokines in NAL were analyzed. Results: TLR4 expression increased in leukocytes in NAL, peripheral blood and bone marrow during symptomatic allergic rhinitis. A similar increase was seen for TLR2 in neutrophils in blood. Nasal challenge with allergen followed by LPS augmented the release of IL-4, IL-5, IL-10, IL-13, IFN-gamma and TNF-alpha. Conclusion: A systemic up-regulation of TLR4 in symptomatic allergic rhinitis may explain why LPS preceded by allergen increases nasal cytokine release. Copyright (C) 2012 S. Karger AG, Basel
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