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Sökning: WFRF:(Johansson Evelina)

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  • [1]234567...8Nästa
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1.
  • Lilja, Mona, et al. (författare)
  • Feminism as Power and Resistance : An Inquiry into Different Forms of Swedish Feminist Resistance and Anti-Genderist Reactions
  • 2018
  • Ingår i: Social Inclusion. - Lisbon, Portugal : Cogitatio Press. - 2183-2803 .- 2183-2803. ; 6:4, s. 82-94
  • Tidskriftsartikel (refereegranskat)abstract
    • This article explores how resistance and power are intertwined within the field of mainstream Swedish feminism, by analyzing some of its more visible expressions and strategies. These feminist resistance strategies could be described as circulating resistance (e.g., the #metoo campaign), public assemblies, the more subtle “disciplinary resistance”, and state feminism. The article illustrates how these different forms of resistance fuel different reactions from movements that reiterate different discourses of “anti-genderism”. In addition, some forms of feminism (state feminism and feminist disciplinary resistance) sometimes develop into, or overlap with, different technologies of power.
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2.
  • Lilja, Mona, 1971, et al. (författare)
  • Understanding Power and Performing Resistance: Swedish Feminists, Civil Society Voices, Biopolitics and “Angry” Men
  • 2013
  • Ingår i: NORA - Nordic Journal of Feminist and Gender Research. - : Routledge. - 0803-8740 .- 1502-394X. ; 21:4, s. 264-279
  • Tidskriftsartikel (refereegranskat)abstract
    • This article discusses feminist resistance in relation to different concepts of power. In particular, it analyzes current debates within Swedish feminism in order to understand what perceptions of power and resistance are being harboured within these discussions. Within the Swedish feminist debate it has been suggested that the equality debate must be radicalized, creating real change instead of policy reports and political party programmes. Others, however, have argued that we must not abandon equality politics for “conflict, struggle and revolt”. This debate is discussed from different theories of power and resistance and we argue that different forms of power become entangled with different forms of resistance, thus creating manifold and messy forms of resistance.
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4.
  • Carvalho-Queiroz, Claudia, et al. (författare)
  • Associations between EBV and CMV Seropositivity, Early Exposures, and Gut Microbiota in a Prospective Birth Cohort : A 10-Year Follow-up
  • 2016
  • Ingår i: Frontiers in Pediatrics. - 2296-2360. ; 4
  • Tidskriftsartikel (refereegranskat)abstract
    • Early-life infections with persistent Epstein-Barr virus (EBV) and cytomegalovirus (CMV) are delayed in affluent countries, probably due to alterations in early environmental exposures, such as maternal age, siblings, and day-care attendance. We have previously reported that the timing of EBV and CMV contraction is related both to allergic sensitization and changes in functional competence of immune cells, while the presence/absence of lactobacilli [Lactobacillus (L.) casei, L. paracasei, and L. rhamnosus] or Staphylococcus (S.) aureus in feces is related to the risk for allergy. Here, we used the same prospective longitudinal birth cohort of children to investigate early-life environmental exposures and their influence on EBV and CMV contraction over time. Since gut microbes also belong to this category of early exposures, we investigated their association with herpesvirus contraction. Our results show that these two viruses are acquired with different kinetics and that EBV and CMV seroprevalence at 10 years of age was 47 and 57%, respectively. We also observed that a delayed EBV or CMV infection was associated with older maternal age [time ratio (TR) 1.14, 95% confidence interval (CI) 1.07-1.21, P-adj < 0.001 and TR 1.09, CI 1.03-1.16, P-adj = 0.008, respectively]. Further, we present the novel finding that S. aureus colonization reduced the time to CMV acquisition (TR 0.21, CI 0.06-0.78, = 0.02). Together, these findings suggest that there is a relationship between timing of herpesvirus acquisition and early-life immune modulating exposures, which interestingly also includes the early infant gut microbiota.
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5.
  • Thul, Peter J., et al. (författare)
  • A subcellular map of the human proteome
  • 2017
  • Ingår i: Science. - : American Association for the Advancement of Science. - 0036-8075 .- 1095-9203. ; 356:6340
  • Tidskriftsartikel (refereegranskat)abstract
    • Resolving the spatial distribution of the human proteome at a subcellular level can greatly increase our understanding of human biology and disease. Here we present a comprehensive image-based map of subcellular protein distribution, the Cell Atlas, built by integrating transcriptomics and antibody-based immunofluorescence microscopy with validation by mass spectrometry. Mapping the in situ localization of 12,003 human proteins at a single-cell level to 30 subcellular structures enabled the definition of the proteomes of 13 major organelles. Exploration of the proteomes revealed single-cell variations in abundance or spatial distribution and localization of about half of the proteins to multiple compartments. This subcellular map can be used to refine existing protein-protein interaction networks and provides an important resource to deconvolute the highly complex architecture of the human cell.
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6.
  • Uhlén, Mathias, et al. (författare)
  • Tissue-based map of the human proteome
  • 2015
  • Ingår i: Science. - 0036-8075 .- 1095-9203. ; 347:6220
  • Tidskriftsartikel (refereegranskat)abstract
    • Resolving the molecular details of proteome variation in the different tissues and organs of the human body will greatly increase our knowledge of human biology and disease. Here, we present a map of the human tissue proteome based on an integrated omics approach that involves quantitative transcriptomics at the tissue and organ level, combined with tissue microarray-based immunohistochemistry, to achieve spatial localization of proteins down to the single-cell level. Our tissue-based analysis detected more than 90% of the putative protein-coding genes. We used this approach to explore the human secretome, the membrane proteome, the druggable proteome, the cancer proteome, and the metabolic functions in 32 different tissues and organs. All the data are integrated in an interactive Web-based database that allows exploration of individual proteins, as well as navigation of global expression patterns, in all major tissues and organs in the human body.
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7.
  • Zandian, A., et al. (författare)
  • Untargeted screening for novel autoantibodies with prognostic value in first-episode psychosis
  • 2017
  • Ingår i: Translational Psychiatry. - : Nature Publishing Group. - 2158-3188 .- 2158-3188. ; 7
  • Tidskriftsartikel (refereegranskat)abstract
    • Immunological and inflammatory reactions have been suggested to have a role in the development of schizophrenia, a hypothesis that has recently been supported by genetic data. The aim of our study was to perform an unbiased search for autoantibodies in patients with a first psychotic episode, and to explore the association between any seroreactivity and the development of a Diagnostic and Statistical Manual of Mental Disorders, fourth edition (DSM-IV) disorder characterized by chronic or relapsing psychotic symptoms. We collected plasma samples from 53 patients when they were treated for their first-episode psychosis, and 41 non-psychotic controls, after which the patients were followed for a mean duration of 7 years. Thirty patients were diagnosed with schizophrenia, delusional disorder, schizoaffective disorder, bipolar disorder or a long-term unspecified nonorganic psychosis during follow-up, whereas 23 patients achieved complete remission. At the end of follow-up, plasma samples were analyzed for IgG reactivity to 2304 fragments of human proteins using a multiplexed affinity proteomic technique. Eight patient samples showed autoreactivity to the N-terminal fragment of the PAGE (P antigen) protein family (PAGE2B/PAGE2/PAGE5), whereas no such autoreactivity was seen among the controls. PAGE autoreactivity was associated with a significantly increased risk of being diagnosed with schizophrenia during follow-up (odds ratio 6.7, relative risk 4.6). An immunohistochemistry analysis using antisera raised against the N-terminal fragment stained an unknown extracellular target in human cortical brain tissue. Our findings suggest that autoreactivity to the N-terminal portion of the PAGE protein family is associated with schizophrenia in a subset of patients with first-episode psychosis.
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8.
  • Bernberg, Evelina, 1981, et al. (författare)
  • Effects of social isolation and environmental enrichment on atherosclerosis in ApoE-/- mice
  • 2008
  • Ingår i: Stress. - 1607-8888. ; 11:5, s. 381-9
  • Tidskriftsartikel (refereegranskat)abstract
    • Social support and a stimulating environment have been suggested to reduce stress reactions and cardiovascular risk. The aim of this study was to assess the role of environmental enrichment and social interaction for development of atherosclerosis in atherosclerosis prone mice. Male ApoE-/- mice were divided into four groups and followed during 20 weeks: (i) enriched environment (E, n=12), (ii) deprived environment (ED, n=12), (iii) enriched environment with exercise (E-Ex, n=12) and (iv) socially deprived by individual housing (SD, n=10). Plasma lipid and cytokine concentrations were measured. Atherosclerosis was quantified in cross-sections of innominate artery and en face in thoracic aorta. Plaque area was significantly increased in SD mice in the innominate artery (P<0.05 vs. all other groups), but not in the thoracic aorta. Plasma lipids were increased in SD mice (P<0.001 vs. all for total cholesterol, P<0.05 vs. E and P<0.01 vs. ED for triglycerides). Plasma concentration of granulocyte-colony stimulating factor (G-CSF) was decreased in SD mice compared to E mice (P<0.05). Thus, social isolation increased atherosclerosis and plasma lipids in ApoE-/- mice. Reduction in plasma G-CSF levels may hamper endothelial regeneration in the atherosclerotic process. While environmental enrichment did not affect atherosclerosis, social isolation accelerated atherosclerosis.
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9.
  • Bernberg, Evelina, 1981, et al. (författare)
  • Repeated exposure to stressors do not accelerate atherosclerosis in ApoE-/- mice
  • 2009
  • Ingår i: Atherosclerosis. - 0021-9150 .- 1879-1484. ; 204:1, s. 90-95
  • Tidskriftsartikel (refereegranskat)abstract
    • Psychosocial stress is suggested to play a significant role in development of cardiovascular disease. To evaluate the effects of repeated exposure to stress on atherosclerosis in atherosclerosis-prone ApoE(-/-) mice we used five different stressors. We further sought to determine whether stress combined with high salt diet induces dysfunctional neurohormonal regulation and impaired salt excretion, thus amplifying the atherogenic potential of salt. The five stressors were evaluated in male C57BL/6 mice and ApoE(-/-) mice (studies I and II) and then used in female ApoE(-/-) mice to study their effect on atherosclerosis (study III). The mice in study III received standard or high salt diet (8%) alone or in combination with stress for 12 weeks. Urine and plasma were collected for corticosterone and lipid analysis, respectively. Acute blood pressure (BP) and heart rate (HR) responses to stress were measured using telemetry. Plaque burden was assessed in the thoracic aorta and aortic root. Plaque morphology was investigated regarding macrophages and collagen content. Urinary corticosterone chronically increased in stressed mice (P<0.05 control vs. stress, P<0.05 control salt vs. stress salt). BP and HR increased acutely during all stressors (P<0.05). Body weight gain decreased significantly in the stress group (P<0.05 vs. control). However, stress did not alter plasma lipid levels, plaque area or plaque morphology. Increased BP and HR suggest an acute stress-related response in ApoE(-/-) mice. Furthermore, stress chronically decreased body weight gain and increased urinary corticosterone levels. Notably, despite an apparent stress effect, stress affected neither atherogenesis nor plaque morphology.
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10.
  • Bernberg, Evelina, 1981, et al. (författare)
  • Social disruption stress increases IL-6 levels and accelerates atherosclerosis in ApoE-/- mice.
  • 2012
  • Ingår i: Atherosclerosis. - 0021-9150 .- 1879-1484. ; 221:2, s. 359-65
  • Tidskriftsartikel (refereegranskat)abstract
    • We have previously shown that different forms of stress have distinctive effects on atherogenesis in mice. We showed that social stress increase atherosclerosis in ApoE(-/-) mice, while more physical forms of stress do not. Here we evaluated the effect of social disruption (SDR) stress on atherogenesis and evaluated cytokine release after SDR-stress and five more physical stressors.
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