| 1. |
|
|
| 2. |
- Karlberg, Mikael, et al.
(författare)
-
Acute vestibular neuritis visualized by 3-T magnetic resonance imaging with high-dose gadolinium.
- 2004
-
Ingår i: Archives of Otolaryngology - Head and Neck Surgery. - 1538-361X. ; 130:2, s. 229-232
-
Tidskriftsartikel (refereegranskat)abstract
- Sudden idiopathic unilateral loss of vestibular function without other signs or symptoms is called acute vestibular neuritis. It has been suggested that reactivation of human herpes simplex virus 1 could cause vestibular neuritis, Bell palsy, and sudden unilateral hearing loss. Enhancement of the facial nerve on gadolinium-enhanced magnetic resonance imaging (MRI) is a common finding in Bell palsy, but enhancement of the vestibular nerve has never been reported in acute vestibular neuritis. We present 2 consecutive cases of acute vestibular neuritis where high-field-strength MRI (3.0 T) with high-dose (0.3 mmol/kg of body weight) gadolinium–pentetic acid showed isolated enhancement of the vestibular nerve on the affected side only. These findings support the hypothesis of a viral and inflammatory cause of acute vestibular neuritis and might have implications for its treatment.
|
|
| 3. |
- Karlberg, Mikael, et al.
(författare)
-
Mondini-like malformation mimicking otosclerosis and superior semicircular canal dehiscence.
- 2006
-
Ingår i: Journal of Laryngology and Otology. - 1748-5460. ; 120:5, s. 419-422
-
Tidskriftsartikel (refereegranskat)abstract
- In 2003, it was reported that superior semicircular canal dehiscence can mimic otosclerosis because of low-frequency bone conduction hearing gain and dissipation of air-conducted acoustic energy through the dehiscence. We report the case of a 17-year-old girl with left-sided combined hearing loss thought to be due to otosclerosis. Bone conduction thresholds were -10 dB at 250 and 500 Hz and she had a 40 dB air bone gap at 250 Hz. When a tuning fork was placed at her ankle she heard it in her left ear. Acoustic reflexes and vestibular evoked myogenic potentials could be elicited bilaterally. Imaging of the temporal bones showed no otosclerosis, superior semicircular canal dehiscence or large vestibular aqueduct, but a left-sided, Mondini-like dysplasia of the cochlea with a modiolar deficiency could be seen. Mondini-like cochlear dysplasia should be added to the causes of inner-ear conductive hearing loss.
|
|
