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Sökning: WFRF:(Liang Jinlong)

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1.
  • Liang, Shuai, et al. (författare)
  • PAHs and soot formation in laminar partially premixed co-flow flames fuelled by PRFs at elevated pressures
  • 2019
  • Ingår i: Combustion and Flame. - : Elsevier BV. - 0010-2180. ; 206, s. 363-378
  • Tidskriftsartikel (refereegranskat)abstract
    • This study investigated polycyclic aromatic hydrocarbons (PAHs)and soot formation characteristics in laminar jet flames fuelled by primary reference fuels (PRFs)at elevated pressures. Qualitative PAHs and quantitative soot profiles were acquired by using laser-induced fluorescence and laser-induced incandescence, respectively. The backpressure of flames ranged from 1 bar to 5 bar. Proper flames with the volume fraction of iso-octane in PRFs varying between 0% and 100% and flame equivalent ratio varying between 3.0 and 11.4 were stabilised in a pressurised chamber. The effects of backpressure, equivalent ratio and iso-octane ratio on PAHs and soot formation were evaluated. PAHs and soot formation can be promoted by increasing iso-octane ratio, equivalent ratio and backpressure. The data suggest that PAHs with large molecular size are more sensitive to the increase of backpressure compared with those with small molecular size. Backpressure played a positive role in the growth of PAHs size. The averaged soot volume fraction showed an approximate power-law relation with pressure. The measured averaged soot volume fraction was proportional to pn. Pressure exponent n was 1.34–2.17, 1.41–2.12 and 1.56–2.20 at equivalent ratios of 6.2, 8.5 and 11.4, respectively.
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2.
  • Sen, Partha, et al. (författare)
  • Novel FOXF1 Mutations in Sporadic and Familial Cases of Alveolar Capillary Dysplasia with Misaligned Pulmonary Veins Imply a Role for its DNA Binding Domain
  • 2013
  • Ingår i: Human Mutation. - : Hindawi Limited. - 1059-7794. ; 34:6, s. 801-811
  • Tidskriftsartikel (refereegranskat)abstract
    • Alveolar capillary dysplasia with misalignment of pulmonary veins (ACD/MPV) is a rare and lethal developmental disorder of the lung defined by a constellation of characteristic histopathological features. Nonpulmonary anomalies involving organs of gastrointestinal, cardiovascular, and genitourinary systems have been identified in approximately 80% of patients with ACD/MPV. We have collected DNA and pathological samples from more than 90 infants with ACD/MPV and their family members. Since the publication of our initial report of four point mutations and 10 deletions, we have identified an additional 38 novel nonsynonymous mutations of FOXF1 (nine nonsense, seven frameshift, one inframe deletion, 20 missense, and one no stop). This report represents an up to date list of all known FOXF1 mutations to the best of our knowledge. Majority of the cases are sporadic. We report four familial cases of which three show maternal inheritance, consistent with paternal imprinting of the gene. Twenty five mutations (60%) are located within the putative DNA-binding domain, indicating its plausible role in FOXF1 function. Five mutations map to the second exon. We identified two additional genic and eight genomic deletions upstream to FOXF1. These results corroborate and extend our previous observations and further establish involvement of FOXF1 in ACD/MPV and lung organogenesis.
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