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Sökning: WFRF:(Källén Anna)

  • Resultat 1-10 av 53
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  • Greenlees, Ruth, et al. (författare)
  • Paper 6: EUROCAT Member Registries: Organization and Activities
  • 2011
  • Ingår i: Birth Defects Research Part C: Embryo Today: Reviews. - Wiley-Blackwell. - 1542-975X. ; 91:Suppl. 1, s. 51-100
  • Forskningsöversikt (refereegranskat)abstract
    • BACKGROUND: EUROCAT is a network of population-based congenital anomaly registries providing standardized epidemiologic information on congenital anomalies in Europe. There are three types of EUROCAT membership: full, associate, or affiliate. Full member registries send individual records of all congenital anomalies covered by their region. Associate members transmit aggregate case counts for each EUROCAT anomaly subgroup by year and by type of birth. This article describes the organization and activities of each of the current 29 full member and 6 associate member registries of EUROCAT. METHODS: Each registry description provides information on the history and funding of the registry, population coverage including any changes in coverage over time, sources for ascertaining cases of congenital anomalies, and upper age limit for registering cases of congenital anomalies. It also details the legal requirements relating to termination of pregnancy for fetal anomalies, the definition of stillbirths and fetal deaths, and the prenatal screening policy within the registry. Information on availability of exposure information and denominators is provided. The registry description describes how each registry conforms to the laws and guidelines regarding ethics, consent, and confidentiality issues within their own jurisdiction. Finally, information on electronic and web-based data capture, recent registry activities, and publications relating to congenital anomalies, along with the contact details of the registry leader, are provided. CONCLUSIONS: The registry description gives a detailed account of the organizational and operational aspects of each registry and is an invaluable resource that aids interpretation and evaluation of registry prevalence data. Birth Defects Research (Part A) 91: S51-S100, 2011. (C) 2011 Wiley-Liss, Inc.
  • Gudmundsdottir, Anna, et al. (författare)
  • Timing of Pharmacological Treatment for Patent Ductus Arteriosus and Risk of Secondary Surgery, Death or Bronchopulmonary Dysplasia: A Population-Based Cohort Study of Extremely Preterm Infants
  • 2015
  • Ingår i: Neonatology2007-01-01+01:00. - Karger. - 1661-7800. ; 107:2, s. 87-92
  • Tidskriftsartikel (refereegranskat)abstract
    • Background: The optimal timing of pharmacological treatment for patent ductus arteriosus (PDA) in extremely pre-term infants is unknown. Objective: To investigate whether timing of pharmacological PDA treatment is associated with a risk of secondary PDA surgery or death before 3 months of age, or bronchopulmonary dysplasia (BPD) in extremely preterm infants. Methods: In this population-based cohort of infants born before 27 gestational weeks in Sweden in 2004-2007, 290/585 infants (50%) received pharmacological PDA treatment. Cox proportional hazards regression estimated the hazard ratio (HR, with 95% confidence interval, Cl) of secondary PDA surgery or death as a composite outcome in relation to postnatal age at the start of pharmacological treatment: early (0-2 days); intermediate (3-6 days); late (>= 7 days). Furthermore, the odds ratio (OR, with 95% Cl) of BPD was estimated in relation to postnatal age at PDA treatment by conditional logistic regression. Results: The median postnatal age at the start of pharmacological PDA treatment was 4 days. 102 infants had secondary PDA surgery. Timing of PDA treatment was not associated with risk of PDA surgery or death; adjusted HRs were 0.89 (95% CI 0.57-1.39) after an intermediate start and 1.10(95% CI 0.53-2.28) after a late start, compared to an early start of treatment. Compared to the early start of PDA treatment, the intermediate start was not associated with any risk of BPD, while late PDA treatment was associated with a lower BPD risk; adjusted ORs were 0.83 (95% CI 0.42-1.64) and 0.28 (95% CI 0.13-0.61), respectively. Conclusion: Timing of pharmacological PDA treatment after extremely preterm birth is not associated with the risk of secondary PDA surgery or death. Moreover, expectant PDA management is not associated with an increased risk of BPD. (C) 2014 S. Karger AG, Basel
  • Lundgren, Pia, et al. (författare)
  • WINROP identifies severe retinopathy of prematurity at an early stage in a nation-based cohort of extremely preterm infants
  • 2013
  • Ingår i: PLoS ONE. - 1932-6203. ; 8:9
  • Tidskriftsartikel (refereegranskat)abstract
    • Objective: To evaluate the ability of a postnatal weight-gain algorithm (WINROP) to identify sight-threatening retinopathy of prematurity (ROP type 1) in a nation-based extremely preterm infant cohort. Methods: This study enrolled all 707 live-born extremely preterm (gestational age [GA] <27 weeks) infants, born 2004-2007 in Sweden; the Extremely preterm Infants in Sweden Study (EXPRESS). WINROP analysis was performed retrospectively in 407 of the infants using weekly weight gain to assess the preterm infant's risk of developing ROP type 1 requiring treatment. GA, birthweight (BW), and weekly postnatal weight measurements were entered into WINROP. WINROP signals with an alarm to indicate if the preterm infant is at risk for ROP type 1. Results: In this extremely preterm population, WINROP correctly identified 96% (45/47) of the infants who required treatment for ROP type 1. The median time from alarm to treatment was 9 weeks (range, 4-20 weeks). Conclusions: WINROP, an online surveillance system using weekly weight gain, identified extremely preterm infants at risk for ROP type 1 requiring treatment at an early stage and with high sensitivity in a Swedish nation-based cohort.
  • Jennbert, Kristina, et al. (författare)
  • Introduction to Life Through Death
  • 2003
  • Ingår i: Fishbones and Glittering Emblems. Southeast Asian Archaelogy 2002. - Museum of Far Eastern Antiquities. Östasiatiska museet. Stockholm.. - 91-970616-0-3 ; s. 71-73
  • Bokkapitel (övrigt vetenskapligt)
  • Arkeologisk Framtid : Arkeologmötet 2008
  • 2009
  • Proceedings (redaktörskap) (övrigt vetenskapligt)abstract
    • Sedan 1994 arrangerar Svenska Arkeologiska Samfundet konferenser som kretsar kring arkeologins roll för människan och samhället. Det åttonde Arkeologmötet hölls i april 2008 på Kulturen i Lund, med temat Framtidens arkeologier - tendenser och visioner. Femton aktiva arkeologer, forskare, kulturskribenter, politiker och myndighetspersoner inviterades för att ge sin syn på den arkeologiska framtiden. I den här boken finner du deras samtidsanalyser och framtidsvisioner presenterade o tretton essäer.
  • Bengt Kallen, A. J., et al. (författare)
  • Cerebral palsy in children born after in vitro fertilization. Is the risk decreasing?
  • 2010
  • Ingår i: European journal of paediatric neurology. - Elsevier Science B.V., Amsterdam. - 1090-3798. ; 14:6, s. 526-530
  • Tidskriftsartikel (refereegranskat)abstract
    • Background: Infants born after in vitro fertilization (IVF) differ from spontaneously conceived infants in a number of aspects which could increase the risk for future cerebral palsy (CP), e.g., multiple births, preterm births, neonatal complications. Aims: To follow up children conceived by IVF with respect to risk for CP. Methods: Infants born after IVF were identified from all IVF clinics in Sweden 1982-2007. Perinatal characteristics were obtained by linkage with the Medical Birth Register. The presence of CP in children born after IVF and in other children was identified from the Patient Register which contains diagnoses given at hospitalizations or specialist outpatient clinics. The risk for CP after IVF was studied after adjustment for year of birth, maternal age, parity, and smoking, all factors which co-vary both with IVF and with CP. Stratification was made for singletons and multiple births and for various neonatal outcomes. Results: The adjusted odds ratio for CP after IVF was 1.81 (95% confidence interval, 95% CI 1.52-2.13), lower and not statistically significant when singletons or when unlike-sexed twins were analyzed. Stratification for various neonatal characteristics also reduced odds ratios to non-significant levels. For the last few years of the study (2004-2007) when the twinning rate after IVF was less than10%, the odds ratio for CP was 0.97 (95% CI 0.57-1.66). Conclusions: The moderately increased risk for CP was most likely a consequence of an increased risk of neonatal morbidity, notably associated with multiple births.
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