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Sökning: WFRF:(Wierzbicka Aneta)

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  • Bekö, Gabriel, et al. (författare)
  • Ultrafine particles: Exposure and source apportionment in 56 Danish homes.
  • 2013
  • Ingår i: Abstract of the 2013 Conference of the International Society of Environmental Epidemiology (ISEE), the International Society of Exposure Science (ISES), and the International Society of Indoor Air Quality and Climate (ISIAQ). - Environ Health Perspect.. ; No. 4059, ID: P-2-08-23
  • Annan publikation (refereegranskat)
  • Bekö, Gabriel, et al. (författare)
  • Ultrafine Particles: Exposure and Source Apportionment in 56 Danish Homes
  • 2013
  • Ingår i: Environmental Science & Technology. - AMER CHEMICAL SOC. - 0013-936X. ; 47:18, s. 10240-10248
  • Tidskriftsartikel (refereegranskat)abstract
    • ABSTRACT: Particle number (PN) concentrations (10−300 nm in size) were continuously measured over a period of ∼45 h in 56 residences of nonsmokers in Copenhagen, Denmark. The highest concentrations were measured when occupants were present and awake (geometric mean, GM: 22.3 × 103 cm−3), the lowest when the homes were vacant (GM: 6.1 × 103 cm−3) or the occupants were asleep (GM: 5.1 × 103 cm−3). Diary entries regarding occupancy and particle related activities were used to identify source events and apportion the daily integrated exposure among sources. Source events clearly resulted in increased PN concentrations and decreased average particle diameter. For a given event, elevated particle concentrations persisted for several hours after the emission of fresh particles ceased. The residential daily integrated PN exposure in the 56 homes ranged between 37 × 103 and 6.0 × 106 particles per cm3·h/day (GM: 3.3 × 105 cm−3·h/day). On average, ∼90% of this exposure occurred outside of the period from midnight to 6 a.m. Source events, especially candle burning, cooking, toasting, and unknown activities, were responsible on average for ∼65% of the residential integrated exposure (51% without the unknown activities). Candle burning occurred in half of the homes where, on average, it was responsible for almost 60% of the integrated exposure.
  • Hemmingsen, Jette Gjerke, et al. (författare)
  • Controlled exposure to diesel exhaust and traffic noise - Effects on oxidative stress and activation in mononuclear blood cells.
  • 2015
  • Ingår i: Mutation research. - 1873-135X. ; 775, s. 66-71
  • Tidskriftsartikel (refereegranskat)abstract
    • Particulate air pollution increases risk of cancer and cardiopulmonary disease, partly through oxidative stress. Traffic-related noise increases risk of cardiovascular disease and may cause oxidative stress. In this controlled random sequence study, 18 healthy subjects were exposed for 3h to diesel exhaust (DE) at 276μg/m(3) from a passenger car or filtered air, with co-exposure to traffic noise at 48 or 75dB(A). Gene expression markers of inflammation, (interleukin-8 and tumor necrosis factor), oxidative stress (heme oxygenase (decycling-1)) and DNA repair (8-oxoguanine DNA glycosylase (OGG1)) were unaltered in peripheral blood mononuclear cells (PBMCs). No significant differences in DNA damage levels, measured by the comet assay, were observed after DE exposure, whereas exposure to high noise levels was associated with significantly increased levels of hOGG1-sensitive sites in PBMCs. Urinary levels of 8-oxo-7,8-dihydro-2'-deoxyguanosine were unaltered. In auxiliary ex vivo experiments whole blood was incubated with particles from the exposure chamber for 3h without effects on DNA damage in PBMCs or intracellular reactive oxygen species production and expression of CD11b and CD62L adhesion molecules in leukocyte subtypes.
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