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Träfflista för sökning "WFRF:(Johansson I) srt2:(1980-1984)"

Sökning: WFRF:(Johansson I) > (1980-1984)

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1.
  • Asp, N G, et al. (författare)
  • Dietary fibre in type II diabetes
  • 1982
  • Ingår i: Acta medica Scandinavica. Supplementum. - : Wiley. - 0365-463X .- 0001-6101. ; 210:S656, s. 47-50
  • Tidskriftsartikel (refereegranskat)abstract
    • Recent studies have indicated that diets rich in digestible carbohydrates and dietary fibre might be beneficial in the regulation of type II non insulin dependent diabetes (NIDD). Addition of the gel forming type of dietary fibre such as pectin and guar gum to meals or glucose solutions reduces post-prandial glucose and insulin response. Addition of cereal fibres in the form of bran seems to have long term beneficial effect improving glucose tolerance. Little is known, however, concerning effects of dietary fibre naturally occurring in food on postprandial glucose and hormone response. In the present study we prepared two breakfast meals which were similar regarding digestible carbohydrates but differed in their dietary fibre content. One of the meals, including whole grain bread and whole apples, contained 8.4 g of dietary fibre, and the other one, containing white bread and apple juice, 3.1 g. When given to eight NIDD, the fibre rich breakfast gave significantly lower blood glucose increment during the three hours following ingestion. The results indicate that foods rich in dietary fibre might be useful in the regulation of type II diabetes.
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2.
  • Ekberg, JO, et al. (författare)
  • Accurate experimental lifetimes of excited levels in sodiumlike sulfur, S VI
  • 1983
  • Ingår i: Physica Scripta. - : IOP Publishing. - 0031-8949 .- 1402-4896. ; 27:6, s. 425-425
  • Tidskriftsartikel (refereegranskat)abstract
    • The lifetimes of the terms 3p2P, 3d2D, 4s2S, 4p2P, 4d2D, 4f2F, 5g2G, 6g2G, 6h2H, 7h2H, 7i2I and 8k2K in sodiumlike sulfur, S VI, have been determined using the beam-foil excitation technique. Most of the decay curves were analyzed by means of the ANDC method which corrects for cascading in a very detailed way. Accurate results were thereby obtained which show that an excellent agreement exists between experimental and theoretical values. Some previously noted disagreements between the results of beam-foil experiments and theoretical investigations are explained as being due to less accurate analyses of the experimental data.
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5.
  • Thornell, L E, et al. (författare)
  • Intermediate filament and associated proteins in the human heart : an immunofluorescence study of normal and pathological hearts.
  • 1984
  • Ingår i: European Heart Journal. - 0195-668X .- 1522-9645. ; 5 Suppl F, s. 231-41
  • Tidskriftsartikel (refereegranskat)abstract
    • The cytoskeleton of human ventricular myocardium in normal and pathological hearts has been analysed with immunocytochemical techniques. Specific antibodies against the intermediate filament proteins desmin (Mr 55 000) and vimentin (Mr 58 000) and antibodies against two cytoskeleton-associated proteins, a spectrin-like protein (Mr 230 000) and vinculin (Mr 130 000), have been used. We show that desmin is localized in the myocytes as an intermyofibrillar lattice at the Z disk level of the myofibrils, and at the intercalated disks. The spectrin-like protein is localized as a transverse striated pattern interlinked with fine longitudinal strands in the subplasmalemmal region of the myocytes. Vinculin is abundant in the intercalated disks and in myotendinous junctions but occurs also at the peripheral sarcolemma in the form of a regular repeat of dots and of fine bar-like extensions into the cytoplasm from the dots. These patterns were observed both in normal and in abnormal hearts, but a number of altered patterns in pathological myocytes were also seen. It is concluded that the intermediate filament system has important implications in the structural function of normal and abnormal hearts but that further studies are needed to elucidate how the different components are related to each other and how they are influenced by different disease processes.
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