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Träfflista för sökning "WFRF:(Wahlund LO) srt2:(1995-1999)"

Search: WFRF:(Wahlund LO) > (1995-1999)

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1.	Almkvist, O, et al. (author) Cerebrospinal fluid levels of alpha-secretase-cleaved soluble amyloid precursor protein mirror cognition in a Swedish family with Alzheimer disease and a gene mutation 1997 In: Archives of neurology. - : American Medical Association (AMA). - 0003-9942. ; 54:5, s. 641-644 Journal article (peer-reviewed)
2.	Almkvist, O, et al. (author) Mild cognitive impairment--an early stage of Alzheimer's disease? 1998 In: Journal of neural transmission. Supplementum. - Vienna : Springer Vienna. - 0303-6995. ; 54:54, s. 21-29 Journal article (peer-reviewed)
3.	Andersen, C, et al. (author) Bilateral temporal lobe volume reduction parallels cognitive impairment in progressive aphasia 1997 In: Archives of neurology. - : American Medical Association (AMA). - 0003-9942. ; 54:10, s. 1294-1299 Journal article (peer-reviewed)
4.	Basun, H, et al. (author) Clinical characteristics of a chromosome 17-linked rapidly progressive familial frontotemporal dementia 1997 In: Archives of neurology. - : American Medical Association (AMA). - 0003-9942. ; 54:5, s. 539-544 Journal article (peer-reviewed)
5.	Blomberg, M, et al. (author) Increasing cerebrospinal fluid tau levels in a subgroup of Alzheimer patients with apolipoprotein E allele epsilon 4 during 14 months follow-up 1996 In: Neuroscience letters. - : Elsevier BV. - 0304-3940. ; 214:2-3, s. 163-166 Journal article (peer-reviewed)
6.	Bronge, L, et al. (author) White matter lesions in Alzheimer patients are influenced by apolipoprotein E genotype 1999 In: Dementia and geriatric cognitive disorders. - : S. Karger AG. - 1420-8008 .- 1421-9824. ; 10:2, s. 89-96 Journal article (peer-reviewed)abstract To analyse the influence of apolipoprotein E (APOE) genotype on the extent of white matter lesions (WMLs) in Alzheimer’s disease (AD), we examined 60 AD patients with magnetic resonance imaging. The WMLs were rated visually in different brain regions. The patients with the APOE genotype σ4/4 had more extensive WMLs in the deep white matter than patients with genotypes σ3/3 and σ3/4. There was a correlation with age for WMLs in the deep white matter in patients with the APOE σ3/3 genotype. In patients carrying at least one σ4 allele, the WMLs showed no age correlation. The results could imply that in APOE allele σ4 carriers, the WMLs represent a pathological process related to the aetiology of the disease.
7.	Dierks, T, et al. (author) EEG-microstates in mild memory impairment and Alzheimer's disease: possible association with disturbed information processing 1997 In: Journal of neural transmission (Vienna, Austria : 1996). - 0300-9564. ; 104:4-5, s. 483-495 Journal article (peer-reviewed)
8.	Erkinjuntti, T, et al. (author) Imaging of static brain lesions in vascular dementia: implications for clinical trials 1999 In: Alzheimer disease and associated disorders. - : Ovid Technologies (Wolters Kluwer Health). - 0893-0341. ; 1313 Suppl 3, s. S81-S90 Journal article (peer-reviewed)
9.	Garbarg, M, et al. (author) Biochemical markers of histaminergic neurons in cerebrospinal fluid of Alzheimer patients and in an animal model of Alzheimer's disease 1998 In: NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY. - 0028-1298. ; 358:1, s. R116-R116 Conference paper (other academic/artistic)
10.	Ingelson, M, et al. (author) Tau immunoreactivity detected in human plasma, but no obvious increase in dementia 1999 In: Dementia and geriatric cognitive disorders. - : S. Karger AG. - 1420-8008 .- 1421-9824. ; 10:6, s. 442-445 Journal article (peer-reviewed)abstract <i>Tau</i> proteins are central to the neuropathology of Alzheimer’s disease and <i>tau</i> levels in cerebrospinal fluid are elevated in affected individuals. In this study, we investigated the presence of <i>tau</i> in plasma from subjects with Alzheimer’s disease (n = 16), frontotemporal dementia (n = 10), vascular dementia (n = 16) and from healthy controls (n = 15). By using an ELISA with monoclonal <i>tau</i> antibodies, <i>tau</i> immunoreactivity was detected in approximately 20% of the subjects. However, no difference between the disease and control groups was seen. After gel filtration of <i>tau</i> immunopositive plasma, the peak reactivity was found in the 160-kD fraction, indicating the source to be <i>tau</i>-like molecules of high-molecular-weight or polymers of low-molecular-weight <i>tau</i> isoforms. We conclude that measurements of <i>tau</i> in plasma cannot be utilized diagnostically for Alzheimer’s disease or for the other dementias investigated.

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Result 1-10 of 38

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Type of publication: journal article (37); conference paper (1)

Type of content: peer-reviewed (36); other academic/artistic (2)

Author/Editor: Wahlund, LO (38); Lannfelt, L (16); Julin, P (15); Almkvist, O (14); Basun, H (14); Winblad, B (11); show more...; Jelic, V (7); Svensson, L (4); Nordberg, A (4); Jensen, M (4); Lindqvist, J (3); Backman, L (3); Scheltens, P (3); Viitanen, M (2); Fazekas, F. (2); Rudberg, U (2); Isberg, B (2); Andersen, C (2); Johansson, SE (2); Wagner, SL (2); Rowe, BA (2); Axelman, K (2); Amberla, K (2); Dierks, T (2); O'Brien, JT (1); Olson, L (1); Hartmann, T. (1); Karlsson, E (1); Larsson, M (1); Wallin, A (1); Mare, K (1); Wetterberg, L (1); Berglund, B (1); Marz, W. (1); Theodorsson, E (1); Seiger, A (1); Jonhagen, ME (1); Cowburn, RF (1); Persson, A. (1); Ostberg, P (1); Herlitz, A (1); Small, B (1); Fernaeus, SE (1); Barkhof, F (1); Leys, D (1); Lindau, M (1); Dahl, C (1); FORSELL, C (1); Brun, A (1); Vanmechelen, E (1); show less...

University: Karolinska Institutet (38)

Language: English (38)

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