| 1. |
- Heijl, A
(författare)
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Computerized perimetry in glaucoma management
- 1989
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Ingår i: Acta Ophthalmologica. - : Wiley. - 0001-639X .- 1755-375X. ; 67:1, s. 1-12
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Forskningsöversikt (refereegranskat)abstract
- The present article discusses the role of computerized perimetry in the management of patients with suspect and manifest glaucoma. The value of visual field examination is compared to that of inspection and photography of the optic disc and to some extent to retinal nerve fibre layer photography. Computerized perimetry is related to standard manual visual field examination. Guidelines are offered for the choice of test programs and for the interpretation of results.
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| 2. |
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| 3. |
- Larsson, E. M., et al.
(författare)
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Regional differences in the proton magnetic resonance relaxation times T1 and T2 within the normal human brain
- 1986
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Ingår i: Acta Radiologica. - : SAGE Publications. - 0284-1851 .- 0567-8056. ; 27:2, s. 231-234
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Forskningsöversikt (refereegranskat)abstract
- The proton magnetic resonance (MR) relaxation times T1 and T2 were determined in autopsy specimens from 13 different regions of normal human brains. One hundred and seventy-four tissue samples from 25 brains were examined in a pulsed MR analyzer of 0.25 T and were then also studied histologically. There were regional differences in T1 and T2 within the cerebral gray matter but not within the white matter. These regional differences might reflect the different composition and cytoarchitectonic structure of the cortical regions and should be taken into consideration in the interpretation of cortical lesions on MR images.
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| 4. |
- Lyttkens, K., et al.
(författare)
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Incidental findings of gastrointestinal tumours at abdominal ultrasound examinations
- 1985
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Ingår i: Acta Radiologica. - : SAGE Publications. - 0284-1851 .- 0567-8056. ; 26:6, s. 671-675
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Forskningsöversikt (refereegranskat)abstract
- A retrospective review was performed of 11 patients referred to ultrasound examination because of abdominal pain and/or a palpable abdominal tumour, which eventually was proven to be gastrointestinal malignancy. Primary gastric carcinoma was present in 4 cases, carcinoma of the small bowel in one case, and of the large bowel in 6 cases. All the patients were examined with conventional ultrasound technique using a 3.5 MHz and a 5.0 MHz transducer. In all the cases bowel wall thickness exceeded 10 mm. A correct organ localization and primary diagnosis of tumour was made in 6 cases, of which only 2 had a palpable abdominal mass. In the remaining cases a bowel tumour was revealed in 3 but the site was incorrectly defined. Reviewing the documentations made at ultrasonography in these cases the tumour origin corresponded well with radiologic and surgical findings. In 2 patients an abscess was diagnosed which later proved to be due to a large bowel carcinoma. Ultrasound examination of patients with uncharacteristic abdominal complaints can spare the patient unnecessary examinations when the findings are pointing at a tumour in the gastrointestinal tract, save time and therefore is of economical importance.
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| 5. |
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| 6. |
- Smith, Maj‐Lis ‐L, et al.
(författare)
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Models for studying long‐term recovery following forebrain ischemia in the rat. 2. A 2‐vessel occlusion model
- 1984
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Ingår i: Acta Neurologica Scandinavica. - : Hindawi Limited. - 0001-6314 .- 1600-0404. ; 69:6, s. 385-401
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Forskningsöversikt (refereegranskat)abstract
- ABSTRACT— A model is described in which transient ischemia is induced in rats anaesthetized with N2O:O2 (70:30) by bilateral carotid artery clamping combined with a lowering of mean arterial blood pressure to 50 mm Hg, the latter being achieved by bleeding, or by bleeding supplemented with administration of trimetaphan or phentolamine. By the use of intubation, muscle paralysis with suxamethonium chloride, and insertion of tail arterial and venous catheters, it was possible to induce reversible ischemia for long‐term recovery studies. Autoradiographic measurements of local CBF showed that the procedure reduced CBF in neocortical areas, hippocampus, and caudoputamen to near‐zero values, flow rates in a number of subcortical areas being variable. Administration of trimethaphane or phentolamine did not affect ischemic and postischemic flow rates, nor did they alter recovery of EEG and sensory‐evoked responses, but trimetaphan blunted the changes in plasma concentrations of adrenaline and noradrenaline. Recovery experiments showed that 10 min of ischemia gave rise to clear signs of permanent brain damage, with a small number of animals developing postischemic seizures that led to the death of the animals in status epilepticus. After 15 min of ischemia, such alterations were more pronounced, and the majority of animals died. It is concluded that the short revival times noted are explained by the fact that the model induces near‐complete ischemia, and that recovery following forebrain ischemia is critically dependent on residual flow rates during the period of ischemia.
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