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Sökning: id:"swepub:oai:DiVA.org:oru-101767" > Cognitive behaviour...

Cognitive behavioural therapy for comorbid insomnia and depression : A randomised, controlled study

Norell-Clarke, A., 1979- (författare)
Örebro universitet,Institutionen för juridik, psykologi och socialt arbete,Center for Health and Medical Psychology
Jansson-Fröjmark, Markus (författare)
Stockholm University, Center for Health and Medical Psychology, Stockholm, Sweden
Tillfors, Maria, 1963- (författare)
Örebro universitet,Institutionen för juridik, psykologi och socialt arbete,Center for Health and Medical Psychology
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Holländare, Fredrik, 1972- (författare)
Örebro universitet,Institutionen för medicinska vetenskaper,Region Örebro län,Örebro County Council, Psychiatric Research Center, Örebro, Sweden
Engström, Ingemar, 1952- (författare)
Örebro universitet,Institutionen för medicinska vetenskaper,Region Örebro län,Örebro County Council, Psychiatric Research Center, Örebro, Sweden
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 (creator_code:org_t)
Amsterdam : Elsevier, 2013
2013
Engelska.
Ingår i: Sleep Medicine. - Amsterdam : Elsevier. - 1389-9457 .- 1878-5506. ; 14:Suppl. 1, s. e99-e99
  • Tidskriftsartikel (övrigt vetenskapligt/konstnärligt)
Abstract Ämnesord
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  • Introduction: Insomnia and depression is a common comorbidity and several pilot studies have demonstrated promising results on both conditions by targeting insomnia only. The aim was to investigate the effects of CBT for insomnia (CBT-I) on both sleep and depressive symptoms in a sample with insomnia comorbid with major depression, minor depression or depressive symptoms, using a randomized controlled study.Materials and methods: 64 participants were recruited through advertisements and randomised to receive either CBT-I or an active control (relaxation training: RT) in groups during four sessions over seven weeks. Insomnia and depressive severity was measured before, during and after treatment, using Insomnia Severity Index (ISI) and Beck Depression Inventory (BDI- II).Results: We used independent t-tests to investigate if groups were different on symptom severity prior to treatment. There was no difference between CBT-I and RT regarding insomnia severity (t (55) = 1.30, p = 0.20) or depression severity (t (53) = −0.77, p = 0.44). Looking at development over time, mixed between-within subjects ANOVAs demonstrated a significant interaction between treatment type and time for both insomnia and depression (ISI: F (2, 54) = 4.96, p = 0.01; BDI: F (2, 58) = 2.80, p = 0.07) meaning that CBT-I meant a larger decrease of both insomnia and depressive severity compared to control treatment. There was also a significant main effect for time with decreasing scores for both groups over time on ISI (F (2, 52) = 28.86, p = 0.0005) and BDI-II (F (2, 58) = 7.11, p = 0.002) and a main effect for group on ISI (F (1, 53) = 9.25, p = 0.01) but not on BDI-II (F (1, 59) = 0.27, p = 0.60). A six months follow-up assessment is currently conducted and those results will also be presented during the conference.Conclusion: CBT-I was associated with a greater reduction in insomnia and depression severity compared to control treatment. These results show that it is possible to have an effect on both insomnia and depression during a relatively short and cost effective group treatment, targeting insomnia only.

Ämnesord

MEDICIN OCH HÄLSOVETENSKAP  -- Hälsovetenskap -- Folkhälsovetenskap, global hälsa, socialmedicin och epidemiologi (hsv//swe)
MEDICAL AND HEALTH SCIENCES  -- Health Sciences -- Public Health, Global Health, Social Medicine and Epidemiology (hsv//eng)

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