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Sökning: onr:"swepub:oai:DiVA.org:uu-422102" > Risk of Colon Cance...

  • Zhang, XuehongHarvard Univ, Dept Nutr, Dept Epidemiol, Sch Publ Hlth, Boston, MA 02115 USA. (författare)

Risk of Colon Cancer and Coffee, Tea, and Sugar-Sweetened Soft Drink Intake : Pooled Analysis of Prospective Cohort Studies

  • Artikel/kapitelEngelska2010

Förlag, utgivningsår, omfång ...

  • 2010-06-02
  • OXFORD UNIV PRESS INC,2010
  • printrdacarrier

Nummerbeteckningar

  • LIBRIS-ID:oai:DiVA.org:uu-422102
  • https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-422102URI
  • https://doi.org/10.1093/jnci/djq107DOI
  • http://kipublications.ki.se/Default.aspx?queryparsed=id:120557577URI

Kompletterande språkuppgifter

  • Språk:engelska
  • Sammanfattning på:engelska

Ingår i deldatabas

Klassifikation

  • Ämneskategori:ref swepub-contenttype
  • Ämneskategori:art swepub-publicationtype

Anmärkningar

  • The relationships between coffee, tea, and sugar-sweetened carbonated soft drink consumption and colon cancer risk remain unresolved. We investigated prospectively the association between coffee, tea, and sugar-sweetened carbonated soft drink consumption and colon cancer risk in a pooled analysis of primary data from 13 cohort studies. Among 731 441 participants followed for up to 6-20 years, 5604 incident colon cancer case patients were identified. Study-specific relative risks (RRs) and 95% confidence intervals (CIs) were estimated using Cox proportional hazards models and then pooled using a random-effects model. All statistical tests were two-sided. Compared with nonconsumers, the pooled multivariable relative risks were 1.07 (95% CI = 0.89 to 1.30, P-trend = .68) for coffee consumption greater than 1400 g/d (about six 8-oz cups) and 1.28 (95% CI = 1.02 to 1.61, P-trend = .01) for tea consumption greater than 900 g/d (about four 8-oz cups). For sugar-sweetened carbonated soft drink consumption, the pooled multivariable relative risk comparing consumption greater than 550 g/d (about 18 oz) to nonconsumers was 0.94 (95% CI = 0.66 to 1.32, P-trend = .91). No statistically significant between-studies heterogeneity was observed for the highest category of each beverage consumed (P > .20). The observed associations did not differ by sex, smoking status, alcohol consumption, body mass index, physical activity, or tumor site (P > .05). Drinking coffee or sugar-sweetened carbonated soft drinks was not associated with colon cancer risk. However, a modest positive association with higher tea consumption is possible and requires further study.

Ämnesord och genrebeteckningar

Biuppslag (personer, institutioner, konferenser, titlar ...)

  • Albanes, DemetriusNCI, Nutr Epidemiol Branch, NIH, Bethesda, MD 20892 USA. (författare)
  • Beeson, W. LawrenceLoma Linda Univ, Sch Publ Hlth, Ctr Hlth Res, Loma Linda, CA 92350 USA. (författare)
  • van den Brandt, Piet A.Maastricht Univ, Dept Epidemiol, GROW Sch Oncol & Dev Biol, Maastricht, Netherlands. (författare)
  • Buring, Julie E.Harvard Univ, Brigham & Womens Hosp, Div Prevent Med, Sch Med,Dept Med, Boston, MA 02115 USA. (författare)
  • Flood, AndrewUniv Minnesota, Sch Publ Hlth, Div Epidemiol & Community Hlth, Minneapolis, MN USA.;Univ Minnesota, Mason Canc Ctr, Minneapolis, MN USA. (författare)
  • Freudenheim, Jo L.SUNY Buffalo, Dept Social & Prevent Med, Buffalo, NY 14260 USA. (författare)
  • Giovannucci, Edward L.Harvard Univ, Brigham & Womens Hosp, Channing Lab, Sch Med,Dept Med, Boston, MA 02115 USA. (författare)
  • Goldbohm, R. AlexandraTNO Qual Life, Dept Food & Chem Risk Anal, Zeist, Netherlands. (författare)
  • Jaceldo-Siegl, KarenLoma Linda Univ, Sch Publ Hlth, Ctr Hlth Res, Loma Linda, CA 92350 USA. (författare)
  • Jacobs, Eric J.Amer Canc Soc, Dept Epidemiol, Atlanta, GA 30329 USA. (författare)
  • Krogh, VittorioNatl Canc Inst, Nutr Epidemiol Unit, I-20133 Milan, Italy. (författare)
  • Larsson, Susanna C.Karolinska Institutet (författare)
  • Marshall, James R.Roswell Pk Canc Inst, Div Canc Prevent & Populat Sci, Buffalo, NY 14263 USA. (författare)
  • McCullough, Marjorie L.Amer Canc Soc, Dept Epidemiol, Atlanta, GA 30329 USA. (författare)
  • Miller, Anthony B.Univ Toronto, Dalla Lana Sch Publ Hlth, Toronto, ON, Canada. (författare)
  • Robien, KimUniv Minnesota, Sch Publ Hlth, Div Epidemiol & Community Hlth, Minneapolis, MN USA.;Univ Minnesota, Mason Canc Ctr, Minneapolis, MN USA. (författare)
  • Rohan, Thomas E.Albert Einstein Coll Med, Dept Epidemiol & Populat Hlth, Bronx, NY 10467 USA. (författare)
  • Schatzkin, ArthurNCI, Div Canc Epidemiol & Genet, Dept Hlth & Human Serv, NIH, Bethesda, MD 20892 USA. (författare)
  • Sieri, SabinaNatl Canc Inst, Nutr Epidemiol Unit, I-20133 Milan, Italy. (författare)
  • Spiegelman, DonnaHarvard Univ, Sch Publ Hlth, Dept Biostat, Boston, MA 02115 USA. (författare)
  • Virtamo, JarmoNatl Inst Hlth & Welf, Dept Chron Dis Prevent, Helsinki, Finland. (författare)
  • Wolk, AlicjaKarolinska Institutet (författare)
  • Willett, Walter C.Harvard Univ, Brigham & Womens Hosp, Channing Lab, Sch Med,Dept Med, Boston, MA 02115 USA. (författare)
  • Zhang, Shumin M.Harvard Univ, Brigham & Womens Hosp, Div Prevent Med, Sch Med,Dept Med, Boston, MA 02115 USA. (författare)
  • Smith-Warner, Stephanie A. (författare)
  • Harvard Univ, Dept Nutr, Dept Epidemiol, Sch Publ Hlth, Boston, MA 02115 USA.NCI, Nutr Epidemiol Branch, NIH, Bethesda, MD 20892 USA. (creator_code:org_t)

Sammanhörande titlar

  • Ingår i:Journal of the National Cancer Institute: OXFORD UNIV PRESS INC102:11, s. 771-7830027-88741460-2105

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