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Träfflista för sökning "bo håkansson srt2:(1995-1999)"

Sökning: bo håkansson > (1995-1999)

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  • Håkansson, Bo, et al. (författare)
  • Linearity of sound transmission through the human skull in vivo
  • 1996
  • Ingår i: Journal of the Acoustical Society of America. - : Acoustical Society of America (ASA). - 0001-4966 .- 1520-8524. ; 99:4, s. 2239-2243
  • Tidskriftsartikel (refereegranskat)abstract
    • The linearity of sound propagation through the human skull was investigated. One male subject, equipped with bilateral skin-penetrating titanium fixtures for attachment of bone-anchored hearing aids, was studied thoroughly. Three different methods were used: comparison of the frequency response functions estimated at different signal levels (using stepped sine as well as random noise), comparison of the coherence function at different signal levels (using random noise), and the Hilbert transform of the estimated frequency response function. Frequencies from 0.1 to 10 kHz and signal levels up to 77 dB HL at discrete frequencies were used. No indication of any significant nonlinear behavior was found with the three methods used.
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  • Håkansson, Bo, 1953, et al. (författare)
  • The Bone-Anchored Hearing Aid
  • 1998
  • Ingår i: Osseointegration in Craniofacial Reconstruction, editor P.-I. Brånemark and D.E. Tolman. ; , s. 311-326
  • Bokkapitel (övrigt vetenskapligt/konstnärligt)
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  • Altraja, Alan, et al. (författare)
  • Expression of laminins in the airways in various types of asthmatic patients: A morphometric study
  • 1996
  • Ingår i: American Journal of Respiratory Cell and Molecular Biology. - 1044-1549 .- 1535-4989. ; 15:4, s. 482-488
  • Tidskriftsartikel (refereegranskat)abstract
    • Laminins (Ln) are crucial in airway morphogenesis. Because they are able to interact with inflammatory cells, they are likely to participate in inflammation accompanied by airway structural remodeling in asthma. Taking biopsies and using immunohistochemistry and quantitative image analysis, we characterized the distribution of Ln chains alpha 1, alpha 2, and beta 2 in the bronchial mucosa of patients with seasonal (n = 17), early occupational (n = 8), and chronic asthma (n = 16) for comparison with that of normal controls (n = 8). In all asthmatic patients, both Ln chains alpha 1 and beta 2 were confined to the superficial margin of the basement membrane (BM), blood vessels, and smooth muscle. The thickness of Ln beta 2 expression in BM was significantly greater in patients with chronic (1.9 +/- 0.1 microns; P < 0.001) and occupational asthma (1.7 +/- 0.1 microns; P < 0.05) than in controls (0.4 +/- 0.3 microns). Only in patients with occupational asthma was the thickness of the Ln alpha 1 layer (2.3 +/- 0.2 microns; mean +/- SEM) significantly different from that in controls (1.4 +/- 0.5 microns; P < 0.05). There was no immunoreactivity for the Ln alpha 2 chain in controls or patients with mild asthma, but in clinically severe chronic asthma we found a discontinuous staining along the epithelial margin of the BM. Since Ln chains alpha 2 and beta 2 appear to function only during morphogenesis, increased expression of these Ln chains in adult asthma patients suggests accelerated tissue turnover in the airways, possibly as a result of airway inflammation in asthma.
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