| 1. |
- Aardal, Karen, et al.
(författare)
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Decomposition principles applied to the dynamic production and work-force scheduling problem
- 1987
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Ingår i: Engineering Costs and Production Economics. - 0167-188X .- 1878-4011. ; 12:1-4, s. 39-49
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Tidskriftsartikel (refereegranskat)abstract
- One of the most important problems in the production and inventory planning field, is the scheduling of production and work force in a dynamic environment. Although this problem can be formulated as a linear program, it is often quite difficult to solve directly, due to its large scale. Instead, it might be fruitful to use a decomposition approach. Decomposition, in general, means decomposing a difficult problem into several easier, or a sequence of easier problems which are later coordinated to reconstruct the original problem.In this report we discuss several possibilities of applying the most common decomposition principles, namely Benders (primal) and Datnzig-Wolfe (dual), as well as a relatively new decomposition method, called cross decomposition, to the dynamic, multiproduct production and employment planning problem. A number of special cases are also presented.
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| 2. |
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| 3. |
- Gottfries, Nils, et al.
(författare)
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Are Mit Students Rational : Report on A Survey
- 1987
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Ingår i: Journal of Economic Behavior and Organization. - : Elsevier BV. - 0167-2681 .- 1879-1751. ; 8:1, s. 113-120
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Tidskriftsartikel (refereegranskat)
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| 4. |
- Hartman, Thomas, 1957-, et al.
(författare)
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Pricing - A strategic decision
- 1987
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Ingår i: Engineering Costs and Production Economics. - : Elsevier BV. - 0167-188X .- 1878-4011. ; 12, s. 375-381
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Tidskriftsartikel (refereegranskat)abstract
- This paper reviews the factors of pricing and costs in determining electricity tariffs and contracts, move specifically discussing changes in consumers' demands and strategies for large users.
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| 5. |
- Hedenstierna, Nils, 1959, et al.
(författare)
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New algorithms for increased efficiency in hierarchical design rule checking
- 1987
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Ingår i: Integration, the VLSI Journal. - : Elsevier BV. - 0167-9260. ; 5:3-4, s. 319-336
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Tidskriftsartikel (refereegranskat)abstract
- This paper presents two new algorithms that make hierarchical geometric design rule checkers more efficient. The first is a method to reduce the number of design rules to be checked when a subcell interact with layout outside the subcell. The second method checks large array of cells (PLA, RAM, ROM, datapaths) in a very efficient way. It could also be used to validate certain types of module generators. These algorithms have been implemented in a program using corner-based rules and an adaptive quad tree as data structure.
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| 6. |
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| 7. |
- Johansson, Christer, et al.
(författare)
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Robots in Flexible Assembly
- 1987
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Ingår i: Robotics. - 0167-8493. ; 3:1, s. 41-46
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Tidskriftsartikel (refereegranskat)
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| 8. |
- Mattiasson, Bo, et al.
(författare)
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Perfluorochemicals in biotechnology
- 1987
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Ingår i: Trends in Biotechnology. - : Elsevier BV. - 0167-7799. ; 5:9, s. 250-254
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Forskningsöversikt (refereegranskat)abstract
- Gas transport often is a limiting factor in biotechnology. Perfluorochemicals provide a new vehicle for the transport of gases.
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| 9. |
- Nilsson Ekdahl, Kristina, et al.
(författare)
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Effects of epinephrine, glucagon and insulin on the activity and degree of phosphorylation of fructose-1,6-bisphosphatase in cultured hepatocytes.
- 1987
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Ingår i: Biochimica et Biophysica Acta. Molecular Cell Research. - 0167-4889 .- 1879-2596. ; 929:3, s. 318-326
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Tidskriftsartikel (refereegranskat)abstract
- The effects of epinephrine, glucagon and insulin on the activity and degree of phosphorylation of fructose-1,6-bisphosphatase in isolated hepatocytes maintained in cell culture for 24 h were investigated. Epinephrine caused a rapid decrease in the apparent Km monitored as the activity ratio between the activity at 12.5 and 83 microM fructose-1,6-bisphosphate, reaching a maximum after 5 min. Glucagon caused a slower and less pronounced activation, and insulin caused an equally slow increase in Km. The effect of epinephrine and glucagon was completely reciprocated by insulin and the action of insulin was totally erased by the other two. Glucagon stimulated the incorporation of [32P]phosphate into fructose-1,6-bisphosphatase from about 2.5 to 4.2 mol/mol enzyme and epinephrine to 3.5 mol/mol. The effect of the two hormones acting together was cumulative. Insulin brought about a decrease in the degree of phosphorylation to 2.0 mol/mol. The effect of epinephrine was shown to be caused by the beta-receptors, since it was completely blocked by propanolol (a beta-antagonist) and remained unaffected by the presence of phentolamine (an alpha-antagonist).
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| 10. |
- Nånberg, Eewa, 1957-, et al.
(författare)
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Alpha-1-adrenergic inositol trisphosphate production in brown adipocytes is Na+ dependent
- 1987
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Ingår i: Biochimica et Biophysica Acta. - : Elsevier. - 0006-3002 .- 1878-2434. ; 930:3, s. 438-445
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Tidskriftsartikel (refereegranskat)abstract
- In order to investigate the ionic requirements for inositol trisphosphate production, brown adipocytes were prelabelled with myo-[3H]inositol and the formation of inositol trisphosphates and inositol bisphosphates as a consequence of alpha 1-adrenergic stimulation was monitored. Omission of Ca2+ from the incubation medium diminished the norepinephrine-induced increase in inositol trisphosphate levels, but it would seem that this reduction can be fully accounted for by a decreased level of the 'inactive' isomer inositol 1,3,4-trisphosphate. Omission of Na+ fully abolished the norepinephrine-induced inositol trisphosphate response. However, it was observed that the presence of Li+ in the incubation medium could fully reconstitute the ability of the cells to yield the early response of inositol trisphosphate production; Li+ could, however, not substitute for Na+ in the entire alpha 1-adrenergic cellular pathway. It was concluded that the Na+-dependent step is found in the coupling mechanism between the alpha 1-receptor and the activation of the phosphodiesterase responsible for inositol trisphosphate production. Thus, all events in the alpha 1-adrenergic pathway which are consequences of IP3 production should appear to be Na+-dependent in these cells.
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