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- Petrov, P. P., et al.
(författare)
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Another deep dimming of the classical T Tauri star RW Aurigae A
- 2015
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Ingår i: Astronomy and Astrophysics. - : EDP Sciences. - 0004-6361 .- 1432-0746. ; 577
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Tidskriftsartikel (refereegranskat)abstract
- Context. RWAur A is a classical T Tauri star (CTTS) with an unusually rich emission line spectrum. In 2014 the star faded by similar to 3 mag in the V band and went into a long-lasting minimum. In 2010 the star underwent a similar fading, although less pronounced. These events in RW Aur A are very unusual among the CTTS, and have been attributed to occultations by passing dust clouds. Aims. We want to find out if any spectral changes took place after the last fading of RW Aur A with the intention of gathering more information on the occulting body and the cause of the phenomenon. Methods. We collected spectra of the two components of RW Aur. The photometry was performed before and during the minimum. Results. The overall spectral signatures reflecting emission from accretion flows from disk to star did not change after the fading. However, blue-shifted absorption components related to the stellar wind increased in strength in certain resonance lines, and the profiles and strengths but not the fluxes of forbidden lines became drastically different. Conclusions. The extinction through the obscuring cloud is grey indicating the presence of large dust grains. At the same time, there are no traces of related absorbing gas. The cloud occults the star and the interior part of the stellar wind, but not the wind or jet further out. The dimming in 2014 was not accompanied by changes in the accretion flows at the stellar surface. There is evidence that the structure and velocity pattern of the stellar wind did change significantly. The dimmings could be related to passing condensations in a tidally disrupted disk, as proposed earlier, but we also speculate that large dust grains have been stirred up from the inclined disk into the line of sight through the interaction with an enhanced wind.
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- Petrov, P. P., et al.
(författare)
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Dynamics of wind and the dusty environments in the accreting T Tauri stars RY Tauri and SU Aurigae
- 2019
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Ingår i: Monthly notices of the Royal Astronomical Society. - : Oxford University Press (OUP). - 0035-8711 .- 1365-2966. ; 483:1, s. 132-146
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Tidskriftsartikel (refereegranskat)abstract
- Classical T Tauri stars with ages of less than 10 Myr possess accretion discs. Magnetohydrodynamic processes at the boundary between the disc and the stellar magnetosphere control the accretion and ejections gas flows. We carried out a long series of simultaneous spectroscopic and photometric observations of the classical T Tauri stars, RY Tauri and SU Aurigae, with the aim to quantify the accretion and outflow dynamics at time-scales from days to years. It is shown that dust in the disc wind is the main source of photometric variability of these stars. In RY Tau, we observed a new effect: during events of enhanced outflow, the circumstellar extinction becomes lower. The characteristic time of changes in outflow velocity and stellar brightness indicates that the obscuring dust is near the star. The outflow activity in both stars is changing on a time-scale of years. Periods of quiescence in the variability of the H alpha profile were observed during the 2015-2016 period in RY Tau and during the 2016-2017 period in SU Aur. We interpret these findings in the framework of the magnetospheric accretion model, and we discuss how the global stellar magnetic field can influence the long-term variations of the outflow activity.
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- Sinha, Pratima, et al.
(författare)
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Myeloid-derived suppressor cells express the death receptor Fas and apoptose in response to T cell-expressed FasL
- 2011
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Ingår i: Blood. - : American Society of Hematology. - 0006-4971 .- 1528-0020. ; 117:20, s. 5381-5390
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Tidskriftsartikel (refereegranskat)abstract
- Myeloid-derived suppressor cells (MDSCs) inhibit adaptive and innate immunity and accumulate in the blood of persons with cancer, chronic inflammation, trauma, infection, and stress. Some of the factors inducing their accumulation are known; however, mechanisms regulating their turnover have not been identified. Mass spectrometry showed prominent expression of apoptosis pathway proteins, suggesting that MDSC turnover may be regulated by Fas-FasL-mediated apoptosis. This hypothesis was confirmed by showing that blood MDSCs induced by 3 mouse tumors were Fas(+) and apoptosed in response to Fas agonist in vitro and to activated FasL(+) T cells in vivo. FasL-deficient mice contained significantly more blood MDSCs than FasL(+/+) mice, and after removal of primary tumors MDSCs regressed in STAT6(-/-) and CD1(-/-) mice but not in STAT6(-/-)FasL(-/-) or CD1(-/-) FasL(-/-) mice. Fas(+) macrophages and dendritic cells did not apoptose in response to activated T cells, indicating that Fas-FasL regulation of myeloid cells was restricted to MDSCs. These results identify a new mechanism regulating MDSC levels in vivo and show a retaliatory relationship between T cells and MDSCs in that MDSCs suppress T-cell activation; however, once activated, T cells mediate MDSC apoptosis.
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