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Sökning: WFRF:(Li NX)

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  • Abazov, V. M., et al. (författare)
  • Search for scalar top quarks in the acoplanar charm jets and missing transverse energy final state in p(p)over-bar collisions at root s=1.96 TeV
  • 2008
  • Ingår i: Physics Letters B. - : Elsevier BV. - 0370-2693 .- 1873-2445. ; 665:1, s. 1-8
  • Tidskriftsartikel (refereegranskat)abstract
    • We present a search for the pair production of scalar top quarks, (t) over tilde, using 995 pb(-1) of data collected in p (p) over bar collisions with the Dempty set detector at the Fermilab Tevatron Collider at root s = 1.96 TeV. Both scalar top quarks are assumed to decay into a charm quark and a neutralino ((chi) over tilde (0)(1)), where (chi) over tilde (0)(1) is the lightest supersymmetric particle. This leads to a final state with two acoplanar charm jets and missing transverse energy. We find the yield of such events to be consistent with the standard model expectation, and exclude sets of (t) over tilde and (chi) over tilde (0)(1) masses at the 95% C.L. that substantially extend the domain excluded by previous searches.
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  • Li, X, et al. (författare)
  • MicroRNA-132 promotes fibroblast migration via regulating RAS p21 protein activator 1 in skin wound healing
  • 2017
  • Ingår i: Scientific reports. - : Springer Science and Business Media LLC. - 2045-2322. ; 7:1, s. 7797-
  • Tidskriftsartikel (refereegranskat)abstract
    • MicroRNA (miR)-132 has been identified as a top up-regulated miRNA during skin wound healing and its inhibition impairs wound repair. In a human in vivo surgical wound model, we showed that miR-132 was induced in epidermal as well as in dermal wound–edge compartments during healing. Moreover, in a panel of cells isolated from human skin wounds, miR-132 was found highly expressed in human dermal fibroblasts (HDFs). In HDFs, miR-132 expression was upregulated by TGF-β1. By overexpression or inhibition of miR-132, we showed that miR-132 promoted HDF migration. Mechanistically, global transcriptome analysis revealed that RAS signaling pathway was regulated by miR-132 in HDFs. We found that RAS p21 protein activator 1 (RASA1), a known target of miR-132, was downregulated in HDFs upon miR-132 overexpression. Silencing of RASA1 phenocopied the pro-migratory effect of miR-132. Collectively, our study reveals an important role for miR-132 in HDFs during wound healing and indicates a therapeutic potential of miR-132 in hard-to-heal skin wounds.
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