| 1. |
- Lembrechts, Jonas J., et al.
(författare)
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Global maps of soil temperature
- 2022
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Ingår i: Global Change Biology. - : Wiley. - 1354-1013 .- 1365-2486. ; 28:9, s. 3110-3144
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Tidskriftsartikel (refereegranskat)abstract
- Research in global change ecology relies heavily on global climatic grids derived from estimates of air temperature in open areas at around 2m above the ground. These climatic grids do not reflect conditions below vegetation canopies and near the ground surface, where critical ecosystem functions occur and most terrestrial species reside. Here, we provide global maps of soil temperature and bioclimatic variables at a 1-km2 resolution for 0–5 and 5–15cm soil depth. These maps were created by calculating the difference (i.e. offset) between in situ soil temperature measurements, based on time series from over 1200 1-km2 pixels (summarized from 8519 unique temperature sensors) across all the world's major terrestrial biomes, and coarse-grained air temperature estimates from ERA5-Land (an atmospheric reanalysis by the European Centre for Medium-Range Weather Forecasts). We show that mean annual soil temperature differs markedly from the corresponding gridded air temperature, by up to 10°C (mean=3.0±2.1°C), with substantial variation across biomes and seasons. Over the year, soils in cold and/or dry biomes are substantially warmer (+3.6±2.3°C) than gridded air temperature, whereas soils in warm and humid environments are on average slightly cooler (−0.7±2.3°C). The observed substantial and biome-specific offsets emphasize that the projected impacts of climate and climate change on near-surface biodiversity and ecosystem functioning are inaccurately assessed when air rather than soil temperature is used, especially in cold environments. The global soil-related bioclimatic variables provided here are an important step forward for any application in ecology and related disciplines. Nevertheless, we highlight the need to fill remaining geographic gaps by collecting more in situ measurements of microclimate conditions to further enhance the spatiotemporal resolution of global soil temperature products for ecological applications.
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| 2. |
- Fontal-Cazalla, F.M., Buffington, M., Nordlander, G., Liljeblad, J., Ros-Farré, P., Nieves-Aldrey, J.L., Pujade-Villar, J. & Ronquist, F.
(författare)
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Phylogeny of the Eucoilinae (Hymenoptera: Cynipoidea: Figitidae).
- 2002
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Ingår i: Cladistics. ; :18, s. 154-199
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Tidskriftsartikel (refereegranskat)
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| 3. |
- Ericson, E., et al.
(författare)
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Hepatic patatin-like phospholipase domain-containing 3 levels are increased in I148M risk allele carriers and correlate with NAFLD in humans
- 2022
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Ingår i: Hepatology communications.. - : Ovid Technologies (Wolters Kluwer Health). - 2471-254X. ; 6:10, s. 2689-2701
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Tidskriftsartikel (refereegranskat)abstract
- In nonalcoholic fatty liver disease (NAFLD) the patatin-like phospholipase domain-containing 3 (PNPLA3) rs738409 variant is a contributor. In mice, the Pnpla3 148M variant accumulates on lipid droplets and probably leads to sequestration of a lipase cofactor leading to impaired mobilization of triglycerides. To advance our understanding of the localization and abundance of PNPLA3 protein in humans, we used liver biopsies from patients with NAFLD to investigate the link to NAFLD and the PNPLA3 148M genotype. We experimentally qualified an antibody against human PNPLA3. Hepatic PNPLA3 protein fractional area and localization were determined by immunohistochemistry in biopsies from a well-characterized NAFLD cohort of 67 patients. Potential differences in hepatic PNPLA3 protein levels among patients related to degree of steatosis, lobular inflammation, ballooning, and fibrosis, and PNPLA3 I148M gene variants were assessed. Immunohistochemistry staining in biopsies from patients with NAFLD showed that hepatic PNPLA3 protein was predominantly localized to the membranes of small and large lipid droplets in hepatocytes. PNPLA3 protein levels correlated strongly with steatosis grade (p = 0.000027) and were also significantly higher in patients with lobular inflammation (p = 0.009), ballooning (p = 0.022), and significant fibrosis (stage 2-4, p = 0.014). In addition, PNPLA3 levels were higher in PNPLA3 rs738409 148M (CG, GG) risk allele carriers compared to 148I (CC) nonrisk allele carriers (p = 0.0029). Conclusion: PNPLA3 protein levels were associated with increased hepatic lipid content and disease severity in patients with NAFLD and were higher in PNPLA3 rs738409 (148M) risk allele carriers. Our hypothesis that increased hepatic levels of PNPLA3 may be part of the pathophysiological mechanism of NAFLD is supported.
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| 4. |
- Liljeblad, Elisabet, 1986-, et al.
(författare)
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MESSENGER observations of the dayside low-latitude boundary layer in Mercury's magnetosphere
- 2015
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Ingår i: Journal of Geophysical Research - Space Physics. - : Blackwell Publishing. - 2169-9380 .- 2169-9402. ; 120:10
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Tidskriftsartikel (refereegranskat)abstract
- Observations from MESSENGER's MAG and FIPS instruments during the first orbital year have resulted in the identification of 25 magnetopause crossings in Mercury's magnetosphere with significant low-latitude boundary layers (LLBLs). Of these crossings 72% are observed dawnside, and 65% for northward interplanetary magnetic field.The estimated LLBL thickness is 450 ± 56 km, and increases with distance to noon. The Na+-group ion is sporadically present in 14 of the boundary layers, with an observed average number density of 22 ± 11% of the proton density. Furthermore, the average Na+-group gyroradii in the layers is 220 ± 34 km, the same order of magnitude as the LLBL thickness.Magnetic shear, plasma β and reconnection rates have been estimated for the LLBL crossings, and compared to those of a control group (non-LLBL) of 61 distinct magnetopause crossings which show signs of nearly no plasma inside the magnetopause. The results indicate that reconnection is significantly slower, or even suppressed, for the LLBL crossings compared to the non-LLBL cases.Possible processes that form or impact the LLBL are discussed. Protons injected through the cusp or flank may be important for the formation of the LLBL. Furthermore, the opposite asymmetry in the Kelvin-Helmholtz instability (KHI) as compared to the LLBL, rules out the KHI as a dominant formation mechanism. However, the KHI and LLBL could be related to each other, either by the impact of sodium ions gyrating across the magnetopause, or by the LLBL preventing the growth of KH waves on the dawnside.
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| 5. |
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| 6. |
- Liljeblad, J, et al.
(författare)
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A phylogenetic analysis of higher-level gall wasp relationships (Hymenoptera : Cynipidae)
- 1998
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Ingår i: SYSTEMATIC ENTOMOLOGY. - : BLACKWELL SCIENCE LTD. - 0307-6970. ; 23:3, s. 229-252
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Tidskriftsartikel (övrigt vetenskapligt/konstnärligt)abstract
- We present the most comprehensive analysis of higher-level relationships in gall wasps conducted thus far. The analysis was based on detailed study of the skeletal morphology of adults, resulting in 164 phylogenetically informative characters, complemente
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| 7. |
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