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Numrering	Referens	Omslagsbild	Hitta
1.	Jakobsson, J., et al. (författare) Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1 2021 Ingår i: Autophagy. - : Informa UK Limited. - 1554-8635 .- 1554-8627. ; 17:1, s. 1-382 Tidskriftsartikel (refereegranskat)
2.	Vitale, I, et al. (författare) Apoptotic cell death in disease-Current understanding of the NCCD 2023 2023 Ingår i: Cell death and differentiation. - 1476-5403. ; 30:5, s. 1097-1154 Tidskriftsartikel (refereegranskat)
3.	Vitale, I, et al. (författare) Apoptotic cell death in disease-Current understanding of the NCCD 2023 2023 Ingår i: Cell death and differentiation. - 1476-5403. ; 30:5, s. 1097-1154 Tidskriftsartikel (refereegranskat)
4.	Galluzzi, L, et al. (författare) Guidelines for the use and interpretation of assays for monitoring cell death in higher eukaryotes. 2009 Ingår i: Cell death and differentiation. - : Springer Science and Business Media LLC. - 1476-5403 .- 1350-9047. ; 16:8, s. 1093-107 Forskningsöversikt (refereegranskat)abstract Cell death is essential for a plethora of physiological processes, and its deregulation characterizes numerous human diseases. Thus, the in-depth investigation of cell death and its mechanisms constitutes a formidable challenge for fundamental and applied biomedical research, and has tremendous implications for the development of novel therapeutic strategies. It is, therefore, of utmost importance to standardize the experimental procedures that identify dying and dead cells in cell cultures and/or in tissues, from model organisms and/or humans, in healthy and/or pathological scenarios. Thus far, dozens of methods have been proposed to quantify cell death-related parameters. However, no guidelines exist regarding their use and interpretation, and nobody has thoroughly annotated the experimental settings for which each of these techniques is most appropriate. Here, we provide a nonexhaustive comparison of methods to detect cell death with apoptotic or nonapoptotic morphologies, their advantages and pitfalls. These guidelines are intended for investigators who study cell death, as well as for reviewers who need to constructively critique scientific reports that deal with cellular demise. Given the difficulties in determining the exact number of cells that have passed the point-of-no-return of the signaling cascades leading to cell death, we emphasize the importance of performing multiple, methodologically unrelated assays to quantify dying and dead cells.
5.	Galluzzi, L, et al. (författare) Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 2018 Ingår i: Cell death and differentiation. - : Springer Science and Business Media LLC. - 1476-5403 .- 1350-9047. ; 25:3, s. 486-541 Tidskriftsartikel (refereegranskat)
6.	Chen, J, et al. (författare) Long-Term Exposure to Fine Particle Elemental Components and Natural and Cause-Specific Mortality-a Pooled Analysis of Eight European Cohorts within the ELAPSE Project 2021 Ingår i: Environmental health perspectives. - 1552-9924. ; 129:4, s. 47009- Tidskriftsartikel (refereegranskat)
7.	Cole-Hunter, T, et al. (författare) Long-term air pollution exposure and Parkinson's disease mortality in a large pooled European cohort: An ELAPSE study 2023 Ingår i: Environment international. - 1873-6750. ; 171, s. 107667- Tidskriftsartikel (refereegranskat)
8.	Kriit, Hedi Katre, et al. (författare) Using Distributed Lag Non-Linear Models to Estimate Exposure Lag-Response Associations between Long-Term Air Pollution Exposure and Incidence of Cardiovascular Disease 2022 Ingår i: International Journal of Environmental Research and Public Health. - : MDPI AG. - 1661-7827 .- 1660-4601. ; 19:5 Tidskriftsartikel (refereegranskat)abstract Long-term air pollution exposure increases the risk for cardiovascular disease, but little is known about the temporal relationships between exposure and health outcomes. This study aims to estimate the exposure-lag response between air pollution exposure and risk for ischemic heart disease (IHD) and stroke incidence by applying distributed lag non-linear models (DLNMs). Annual mean concentrations of particles with aerodynamic diameter less than 2.5 µm (PM2.5 ) and black carbon (BC) were estimated for participants in five Swedish cohorts using dispersion models. Simultaneous estimates of exposure lags 1–10 years using DLNMs were compared with separate year specific (single lag) estimates and estimates for lag 1–5-and 6–10-years using moving average exposure. The DLNM estimated no exposure lag-response between PM2.5 total, BC, and IHD. However, for PM2.5 from local sources, a 20% risk increase per 1 µg/m3 for 1-year lag was estimated. A risk increase for stroke was suggested in relation to lags 2–4-year PM2.5 and BC, and also lags 8–9-years BC. No associations were shown in single lag models. Increased risk estimates for stroke in relation to lag 1–5-and 6–10-years BC moving averages were observed. Estimates generally supported a greater contribution to increased risk from exposure windows closer in time to incident IHD and incident stroke. © 2022 by the authors. Licensee MDPI, Basel, Switzerland.
9.	Stafoggia, M, et al. (författare) Long-term exposure to low ambient air pollution concentrations and mortality among 28 million people: results from seven large European cohorts within the ELAPSE project 2022 Ingår i: The Lancet. Planetary health. - 2542-5196. ; 6:1, s. e9-e18 Tidskriftsartikel (refereegranskat)
10.	Wolf, K, et al. (författare) Long-term exposure to low-level ambient air pollution and incidence of stroke and coronary heart disease: a pooled analysis of six European cohorts within the ELAPSE project 2021 Ingår i: The Lancet. Planetary health. - 2542-5196. ; 5:9, s. E620-E632 Tidskriftsartikel (refereegranskat)

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Typ av publikation: tidskriftsartikel (53); konferensbidrag (8); forskningsöversikt (1)

Typ av innehåll: refereegranskat (50); övrigt vetenskapligt/konstnärligt (12)

Författare/redaktör: Rizzuto, D (48); Pershagen, G (24); Bellander, T (23); Ketzel, M (21); Nagel, G (19); Chen, J. (19); visa fler...; Wolf, K (19); Brunekreef, B (19); Leander, K (19); Andersen, ZJ (19); Samoli, E (19); Strak, M (19); Weinmayr, G (19); Hoek, G (19); Forastiere, F (17); Hoffmann, B. (17); Fratiglioni, L (17); Katsouyanni, K (17); de Hoogh, K (17); Brandt, J (17); Zitt, E (17); Fecht, D (17); Gulliver, J (17); Hvidtfeldt, UA (17); Stafoggia, M (17); Magnusson, PKE (16); Raaschou-Nielsen, O (16); Ljungman, P (15); Peters, A (11); Schramm, S. (10); Liu, S. (10); Lager, A (10); Severi, G (10); Tjonneland, A (9); Vetrano, DL (9); van der Schouw, YT (8); Grande, G. (7); Kumar, S (6); Madeo, F (6); Kroemer, G (6); Zhivotovsky, B (6); Boutron-Ruault, MC (6); Jockel, KH (6); Kepp, O (6); Galluzzi, L (6); Calderon-Larranaga, ... (6); So, R (6); Cole-Hunter, T (6); Mortensen, LH (6); Lim, YH (6); visa färre...

Lärosäte: Karolinska Institutet (58); Göteborgs universitet (8); Stockholms universitet (8); Umeå universitet (6); Lunds universitet (4); Uppsala universitet (2); visa fler...; Linköpings universitet (1); Chalmers tekniska högskola (1); Sveriges Lantbruksuniversitet (1); Röda Korsets Högskola (1); visa färre...

Språk: Engelska (62)

Forskningsämne (UKÄ/SCB): Medicin och hälsovetenskap (13); Naturvetenskap (6)

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