| 1. |
- Aronsson, Hanna, 1977-
(författare)
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On Sexual Imprinting in Humans
- 2011
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Doktorsavhandling (övrigt vetenskapligt/konstnärligt)abstract
- In this thesis I investigate whether human sexual preferences develop through sexual imprinting. Sexual imprinting is the acquisition of sexual preferences through non-rewarded experiences with parents and siblings during an early sensitive period and it is known to exist in many other animals. Learning is often sex specific so that males, for instance, learn to prefer as sexual partners individuals that look like their mother, and avoid individuals that look like their father. First, sexual imprinting in animals and humans is reviewed and compared to prevailing evolutionary views presupposing genetically determined sexual preferences. Further, by means of web surveys, I have explored the relationship between childhood exposure to parents with certain natural and cultural traits and sexual attraction to these traits in a partner. Cultural traits were included because it is unlikely that preferences for them are genetically determined adaptations. Parental effects varied between traits. For instance, in heterosexual males, a positive effect of mother was found on attraction to smoking, but not glasses, while a negative paternal effect was found on attraction to glasses, but not smoking. However, when maternal and paternal effects were investigated for a large number of artificial and natural traits, including smoking and glasses, an overall positive effect of opposite sex parent emerged in both heterosexual males and females. Additionally, in the last study we explored a sexual preference for pregnant and lactating women. Results suggest that exposure to a pregnant and lactating mother had an effect if it occurred when the respondent was between 1,5 and 5 years old. In conclusion, these results suggest that human sexual preferences are the result of sex specific learning during a sensitive period. Sexual imprinting should therefore be recognised as a plausible explanation to human sexual preferences that deserves further scientific investigation.
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| 2. |
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| 3. |
- Aronsson, Hanna, 1977-, et al.
(författare)
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Parental influences on sexual preferences : The case of attraction to smoking
- 2011
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Ingår i: Journal of Evolutionary Psychology. - Budapest, Ungern : Akadémiai Kiadó. - 0737-4828. ; 9:1, s. 21-41
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Tidskriftsartikel (refereegranskat)abstract
- We investigated whether a sexual preference for smoking can be related to past experiences of parental smoking during childhood, as predicted by the theory of sexual imprinting, but also by sexual conditioning theory. In a sample of over 4000 respondents to five Internet surveys on sexual preferences, we found that parental smoking correlates with increased attraction to smoking in self-reported hetero- and homosexual males. Maternal smoking was associated with an increase in attraction to smoking both in hetero- and homosexual males, while paternal smoking was associated with an increase in attraction to smoking only in males who prefer male partners. We could not explain these findings by considering other factors than parental smoking habits, such as possibly biased reporting, indicators of a sexually liberal lifestyle or phenotype matching. Our data are consistent with the hypothesis that sexual preferences are acquired early in life by exposure to stimuli provided by individuals in the child’s environment, such as caregivers. The sex specificity of the parental effect is consistent with sexual imprinting theory but not with conditioning theory.
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| 4. |
- Arslan, Alan A, et al.
(författare)
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Circulating vitamin d and risk of epithelial ovarian cancer
- 2009
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Ingår i: Journal of oncology. - : Hindawi Limited. - 1687-8450 .- 1687-8469. ; 2009, s. 672492-672500
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Tidskriftsartikel (refereegranskat)abstract
- We conducted a nested case-control study within two prospective cohorts, the New York University Women's Health Study and the Northern Sweden Health and Disease Study, to examine the association between prediagnostic circulating levels of 25-hydroxy vitamin D (25(OH)D) and the risk of subsequent invasive epithelial ovarian cancer (EOC). The 25(OH)D levels were measured in serum or plasma from 170 incident cases of EOC and 373 matched controls. Overall, circulating 25(OH)D levels were not associated with the risk of EOC in combined cohort analysis: adjusted OR for the top tertile versus the reference tertile, 1.09 (95% CI, 0.59-2.01). In addition, there was no evidence of an interaction effect between VDR SNP genotype or haplotype and circulating 25(OH)D levels in relation to ovarian cancer risk, although more complex gene-environment interactions may exist.
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| 5. |
- Ask, Urban, 1956, et al.
(författare)
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Applied business intelligence in the making : An inter-university case from swedish higher education
- 2009
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Ingår i: Business Information Systems Workshops. - Berlin, Heidelberg : Springer Berlin/Heidelberg. - 9783642034237 ; , s. 226-230
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Konferensbidrag (refereegranskat)abstract
- There has long been a debate regarding the inclusion of IT into the curriculum for business students. With IT being a natural part of their coming working environment, the under-developed use of for instance Enterprise Resource Planning (ERP) and Business Intelligence (BI) solutions has suffered much critique. As a response to this, the Centre for Business Solutions and the Scandinavian Academic Network for Teaching Enterprise Systems (SANTE) have created a joint initiative together with the industry. Through making the full accounts from a medium-sized manufacturing company available to the students through a specially designed BI solution, the students are given the task to identify potential problems with the accounts. The assignment is intended to be run in the form of a competition, where the students from different Swedish universities compete in analyzing the company in a given time-frame. The purpose of this case is to present the outline and outset for the competition, together with some initial reflections on the setup-phase.
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| 6. |
- Bader, Thomas K., 1980-, et al.
(författare)
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Path dependence in OSB sheathing-to-framing nailed connection revealed by biaxial testing
- 2018
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Ingår i: Journal of Structural Engineering. - Reston, VA : American Society of Civil Engineers (ASCE). - 0733-9445 .- 1943-541X. ; 144:10
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Tidskriftsartikel (refereegranskat)abstract
- OSB sheathing-to-wood framing connection, as typically used in light-frame shear walls, was experimentally examined in a novel biaxial test setup with respect to possible path dependence of the load-displacement relation. The connection with an annular-ringed shank nail was loaded under displacement control following nine different displacement paths within the sheathing plane, which coincided at a number of points. In intersection points, resultant connection force, its orientation and work performed on the connection system to reach the specific point were calculated and compared. Evaluation of experiments revealed significant path dependence with respect to orientation of force resultants at path intersection points. However, magnitude of the forces and the work carried out showed relatively small dependence of the displacement path undertaken. Comparison of uniaxial connection tests with the European yield model demonstrated strong contribution of withdrawal resistance of the ringed shank nail to its lateral strength. Results of this type are a valuable basis to build better models when simulating such connections in wood structures.
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| 7. |
- Bartlett, S E, et al.
(författare)
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Dopamine responsiveness is regulated by targeted sorting of D2 receptors
- 2005
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Ingår i: Proceedings of the National Academy of Sciences. - : Proceedings of the National Academy of Sciences. - 1091-6490 .- 0027-8424. ; 102:32, s. 11521-11526
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Tidskriftsartikel (refereegranskat)abstract
- Aberrant dopaminergic signaling is a critical determinant in multiple psychiatric disorders, and in many disease states, dopamine receptor number is altered. Here we identify a molecular mechanism that selectively targets D2 receptors for degradation after their activation by dopamine. The degradative fate of D2 receptors is determined by an interaction with G protein coupled receptor-associated sorting protein (GASP). As a consequence of this GASP interaction, D2 responses in rat brain fail to resensitize after agonist treatment. Disruption of the D2-GASP interaction facilitates recovery of D2 responses, suggesting that modulation of the D2-GASP interaction is important for the functional down-regulation of D2 receptors.
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| 8. |
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| 9. |
- Berglin-Enquist, Ida, et al.
(författare)
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Murine models of acute neuronopathic Gaucher disease
- 2007
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Ingår i: Proceedings of the National Academy of Sciences of the United States of America. - : Proceedings of the National Academy of Sciences. - 0027-8424 .- 1091-6490. ; 104:44, s. 17483-17488
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Tidskriftsartikel (refereegranskat)abstract
- Gaucher disease (GD) is an autosomal recessive lysosomal storage disorder caused by mutations in the glucosidase, beta, acid (GBA) gene that encodes the lysosomal enzyme glucosylceramidase (GCase). GCase deficiency leads to characteristic visceral pathology and, in some patients, lethal neurological manifestations. Here, we report the generation of mouse models with the severe neuronopathic form of GD. To circumvent the lethal skin phenotype observed in several of the previous GCase-deficient animals, we genetically engineered a mouse model with strong reduction in GCase activity in all tissues except the skin. These mice exhibit rapid motor dysfunction associated with severe neurodegeneration and apoptotic cell death within the brain, reminiscent of neuronopathic GD. In addition, we have created a second mouse model, in which GCase deficiency is restricted to neural and glial cell progenitors and progeny. These mice develop similar pathology as the first mouse model, but with a delayed onset and slower disease progression, which indicates that GCase deficiency within microglial cells that are of hematopoietic origin is not the primary determinant of the CNS pathology. These findings also demonstrate that normal microglial cells cannot rescue this neurodegenerative disease. These mouse models have significant implications for the development of therapy for patients with neuronopathic GD.
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| 10. |
- Berglin-Enquist, Ida, et al.
(författare)
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Successful Low-Risk Hematopoietic Cell Therapy in a Mouse Model of Type 1 Gaucher Disease
- 2009
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Ingår i: Stem Cells. - : Oxford University Press (OUP). - 1549-4918 .- 1066-5099. ; 27:3, s. 744-752
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Tidskriftsartikel (refereegranskat)abstract
- Hematopoietic stem cell-based gene therapy offers the possibility of permanent correction for genetic disorders of the hematopoietic system. However, optimization of present protocols is required before gene therapy can be safely applied as general treatment of genetic diseases. In this study we have used a mouse model of type 1 Gaucher disease (GD) to demonstrate the feasibility of a low-risk conditioning regimen instead of standard radiation, which is associated with severe adverse effects. We first wanted to establish what level of engraftment and glucosylceramidase (GCase) activity is required to correct the pathology of the type 1 GD mouse. Our results demonstrate that a median wild-type (WT) cell engraftment of 7%, corresponding to GCase activity levels above 10 nmoles/hour and mg protein, was sufficient to reverse pathology in bone marrow and spleen in the GD mouse. Moreover, we applied nonmyeloablative doses of busulfan as a pretransplant conditioning regimen and show that even WT cell engraftment in the range of 1%-10% can confer a beneficial therapeutical outcome in this disease model. Taken together, our data provide encouraging evidence for the possibility of developing safe and efficient conditioning protocols for diseases that require only a low level of normal or gene-corrected cells for a permanent and beneficial therapeutic outcome. STEM CELLS 2009; 27: 744-752
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| 11. |
- Berglin-Enquist, Ida, et al.
(författare)
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Successful low-risk hematopoietic cell therapy in a mouse model of type 1 Gaucher disease.
- 2008
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Ingår i: - : Mary Ann Liebert Inc. ; , s. 1189-1190
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Konferensbidrag (refereegranskat)abstract
- Hematopoietic stem cell (HSC) based gene therapy offers the possibility of permanent correction for genetic disorders of the hematopoietic system. However, optimization of present protocols is required before gene therapy can be safely applied as general treatment of genetic diseases. In this study we have used a mouse model of type 1 Gaucher disease (GD) to demonstrate the feasibility of a low-risk conditioning regimen instead of standard radiation, which is associated with severe adverse effects. We first wanted to establish what level of engraftment and glucosylceramidase (GCase) activity is required to correct the pathology of the type 1 GD mouse. Our results demonstrate that a median WT cell engraftment of 7 % corresponding to GCase activity levels above 10 nmol/hr and mg protein was sufficient to reverse pathology within bone marrow (BM) and spleen in the GD mouse. Moreover, we applied non-myeloablative doses of busulphan as a pretransplant conditioning regimen and show that even WT cell engraftment in the range of 1-10% can confer a beneficial therapeutical outcome in this disease model. Taken together, our data provide encouraging evidence for the possibility to develop safe and efficient conditioning protocols for diseases that only require a low level of normal or gene corrected cells for a permanent and beneficial therapeutic outcome. ______________________________________________________________________________ Author contributions: I.B.E.: Conception and design, collection and assembly of data, data analysis and interpretation, manuscript writing; E.N.: Collection of data, data analysis and interpretation; J.-E.M.: Collection and assembly of data, data analysis and interpretation; M.E.: Collection and assembly of data, data analysis and interpretation; J.R.: Data analysis and interpretation, manuscript writing; S.K.: Conception and design, financial support, data analysis and interpretation, manuscript writing, final approval of manuscript.
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| 12. |
- Björkman, Anne, 1981, et al.
(författare)
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Plant functional trait change across a warming tundra biome
- 2018
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Ingår i: Nature. - : Springer Science and Business Media LLC. - 0028-0836 .- 1476-4687. ; 562:7725, s. 57-62
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Tidskriftsartikel (refereegranskat)abstract
- The tundra is warming more rapidly than any other biome on Earth, and the potential ramifications are far-reaching because of global feedback effects between vegetation and climate. A better understanding of how environmental factors shape plant structure and function is crucial for predicting the consequences of environmental change for ecosystem functioning. Here we explore the biome-wide relationships between temperature, moisture and seven key plant functional traits both across space and over three decades of warming at 117 tundra locations. Spatial temperature–trait relationships were generally strong but soil moisture had a marked influence on the strength and direction of these relationships, highlighting the potentially important influence of changes in water availability on future trait shifts in tundra plant communities. Community height increased with warming across all sites over the past three decades, but other traits lagged far behind predicted rates of change. Our findings highlight the challenge of using space-for-time substitution to predict the functional consequences of future warming and suggest that functions that are tied closely to plant height will experience the most rapid change. They also reveal the strength with which environmental factors shape biotic communities at the coldest extremes of the planet and will help to improve projections of functional changes in tundra ecosystems with climate warming.
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| 13. |
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| 14. |
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| 15. |
- Enquist, Håkan, 1960-, et al.
(författare)
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Change management implications for network organizations
- 2004
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Ingår i: Proceedings of the 37th Hawaii International Conference on System Sciences Volume 37, 2004, Article numberCLDKM04, Pages 397-406. - : IEEE Computer Society. - 0769520561 ; , s. 397-406
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Konferensbidrag (refereegranskat)abstract
- The purpose of this paper is to investigatepossible change management implicationsfor networks organizations. This is achievedthrough applying six Critical ManagementIssues (CMI’s) from the changemanagement framework DELTA on ataxonomy consisting of three generic typesof network organizations. The paper is builton an empirical base comprised of over 150expert interviews and 121 questionnairerespondents. Questionnaire respondents andinterviewed experts represent middle to topmanagement of companies of all sizesinvolved in networks. The results show thatthe framework applied to the changemanagement in the different networkshighlights and addresses differentmanagement activities per CMI. Given this,we discuss the differences in managerialimplications that the framework identifiesfor the different types of networks.
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| 16. |
- Enquist, Håkan, 1960-, et al.
(författare)
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DELTA, an architecture for management of enterprise development
- 2006
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Ingår i: Proceedings of the 14th European Conference on Information Systems, ECIS 2006 2006, 12p.
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Konferensbidrag (refereegranskat)abstract
- Management of enterprise development refers to the complex task of transforming an enterprise fromone state to another (desirable) state in a controlled manner. This paper presents a framework thatenables the systematization of empirical as well as theoretical knowledge contributions relevant formanagement of enterprise development. The framework is validated theoretically as well asempirically. The framework has proved effective to systemize and relate this variety of issues andprovide a tool for comprehensibility for practitioners trying to grasp the development situation inwhich they engage. The framework is a valid foundation for elaborating a knowledgebase supportingthe management of enterprise and IS development in complex organisations.
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| 17. |
- Enquist, Johan, et al.
(författare)
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Kinin-Stimulated B1 Receptor Signaling Depends on Receptor Endocytosis Whereas B2 Receptor Signaling Does Not.
- 2014
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Ingår i: Neurochemical Research. - : Springer Science and Business Media LLC. - 1573-6903 .- 0364-3190. ; 39:6, s. 1037-1047
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Tidskriftsartikel (refereegranskat)abstract
- Kinins are potent pro-inflammatory peptides that act through two G protein-coupled receptor subtypes, B1 (B1R) and B2 (B2R). Kinin-stimulated B2R signaling is often transient, whereas B1R signaling is sustained. This was confirmed by monitoring agonist-stimulated intracellular Ca(2+) mobilization in A10 smooth muscle cells expressing human wild-type B2R and B1R. We further studied the role of receptor membrane trafficking in receptor-mediated phosphoinositide (PI) hydrolysis in model HEK293 cell lines stably expressing the receptors. Treatment of cells with brefeldin A, to inhibit maturation of de novo synthesized receptors, or hypertonic sucrose, to inhibit receptor endocytosis, showed that the basal cell surface receptor turnover was considerably faster for B1R than for B2R. Inhibition of endocytosis, which stabilized B1R on the cell surface, inhibited B1R signaling, whereas B2R signaling was not perturbed. Signaling by a B1R construct in which the entire C-terminal domain was deleted remained sensitive to inhibition of receptor endocytosis, whereas signaling by a B1R construct in which this domain was substituted with the corresponding domain in B2R was not sensitive. B2R and B1R co-expression, which also appeared to stabilize B1R on the cell surface, presumably by receptor hetero-dimerization, also inhibited B1R signaling, whereas B2R signaling was slightly enhanced. Furthermore, the B2R-specific agonist bradykinin (BK) directed both receptors through a common endocytic pathway, whereas the B1R-specific agonist Lys-desArg(9)-BK was unable to do so. These results suggest that B1R-mediated PI hydrolysis depends on a step in receptor endocytosis, whereas B2R-mediated PI hydrolysis does not. We propose that B1R uses at least part of the endocytic machinery to sustain agonist-promoted signaling.
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| 18. |
- Enquist, Johan, et al.
(författare)
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Kinins promote B2 receptor endocytosis and delay constitutive B1 receptor endocytosis.
- 2007
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Ingår i: Molecular Pharmacology. - : American Society for Pharmacology & Experimental Therapeutics (ASPET). - 1521-0111 .- 0026-895X. ; 71:2, s. 494-507
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Tidskriftsartikel (refereegranskat)abstract
- Upon sustained insult, kinins are released and many kinin responses, such as inflammatory pain, adapt from a B2 receptor (B2R) type in the acute phase to a B1 receptor (B1R) type in the chronic phase. In this study, we show that kinins modulate receptor endocytosis to rapidly decrease B2R and increase B1R on the cell surface. B2Rs, which require agonist for activity, are stable plasma membrane components without agonist but recruit beta-arrestin 2, internalize in a clathrin-dependent manner, and recycle rapidly upon agonist treatment. In contrast, B1Rs, which are inducible and constitutively active, constitutively internalize without agonist via a clathrin-dependent pathway, do not recruit beta-arrestin 2, bind G protein-coupled receptor sorting protein, and target lysosomes for degradation. Agonist delays B1R endocytosis, thus transiently stabilizing the receptor. Most of the receptor trafficking phenotypes are transplantable from one receptor to the other through exchange of the C-terminal receptor tails, indicating that the tails contain epitopes that are important for the binding of protein partners that participate in the endocytic and postendocytic receptor choices. It is noteworthy that the agonist delay of B1R endocytosis is not transplanted to the B2R via the B1R tail, suggesting that this property of the B1R requires another domain. These events provide a rapid kinin-dependent mechanism for 1) regulating the constitutive B1R activity and 2) shifting the balance of accessible receptors in favor of B1R.
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| 19. |
- Enquist, Johan
(författare)
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The Role of Membrane Trafficking in G Protein-Coupled Receptor Regulation
- 2007
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Doktorsavhandling (övrigt vetenskapligt/konstnärligt)abstract
- Movements of a receptor in the plasma membrane and within the cell influence receptor function and physiology. I have investigated the role of such movements, generally known as membrane trafficking, in G protein-coupled receptor regulation. My studies show that agonist-promoted internalization and postendocytic sorting determine the degree of resensitization of opioid m (MOR) and d (DOR) receptors, and dopamine D2 (D2R) receptors. Further, postendocytic sorting of DOR and D2R is determined by the non-covalent interaction of the receptors with the protein G protein-coupled associated sorting protein (GASP). Abrogation of the DOR- and D2R-GASP interaction leads to an altered postendocytic fate of the two receptors and results in their recycling and resensitization. I have also shown that the cognate agonist des-Arg10-kallidin for the G protein-coupled bradykinin B1 receptor (B1R) acts as an inverse agonist on receptor endocytosis and does not cause receptor desensitization. Further, I have mapped the internalization routes utilized by the B1R and bradykinin B2 receptors and isolated the domains necessary for their internalization and postendocytic sorting. In summary, my studies highlight and detail a novel regulatory mode of activity in three receptor groups of great physiological and pharmacological significance in areas such as pain transmission. Further, I show that receptor membrane trafficking can be artificially modulated to impact in vivo receptor function. Thus, my result may be directly utilized for future therapeutic purposes.
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| 20. |
- Enquist, Magnus, et al.
(författare)
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The power of associative learning and the ontogeny of optimal behaviour
- 2016
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Ingår i: Royal Society Open Science. - : The Royal Society. - 2054-5703. ; 3:11
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Tidskriftsartikel (refereegranskat)abstract
- Behaving efficiently (optimally or near-optimally) is central to animals' adaptation to their environment. Much evolutionary biology assumes, implicitly or explicitly, that optimal behavioural strategies are genetically inherited, yet the behaviour of many animals depends crucially on learning. The question of how learning contributes to optimal behaviour is largely open. Here we propose an associative learning model that can learn optimal behaviour in a wide variety of ecologically relevant circumstances. The model learns through chaining, a term introduced by Skinner to indicate learning of behaviour sequences by linking together shorter sequences or single behaviours. Our model formalizes the concept of conditioned reinforcement (the learning process that underlies chaining) and is closely related to optimization algorithms from machine learning. Our analysis dispels the common belief that associative learning is too limited to produce ‘intelligent’ behaviour such as tool use, social learning, self-control or expectations of the future. Furthermore, the model readily accounts for both instinctual and learned aspects of behaviour, clarifying how genetic evolution and individual learning complement each other, and bridging a long-standing divide between ethology and psychology. We conclude that associative learning, supported by genetic predispositions and including the oft-neglected phenomenon of conditioned reinforcement, may suffice to explain the ontogeny of optimal behaviour in most, if not all, non-human animals. Our results establish associative learning as a more powerful optimizing mechanism than acknowledged by current opinion.
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| 21. |
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| 22. |
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| 23. |
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| 24. |
- Ghirlanda, Stefano, et al.
(författare)
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A-learning : A new formulation of associative learning theory
- 2020
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Ingår i: Psychonomic Bulletin & Review. - : Springer Science and Business Media LLC. - 1069-9384 .- 1531-5320. ; 27, s. 1166-1194
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Tidskriftsartikel (refereegranskat)abstract
- We present a new mathematical formulation of associative learning focused on non-human animals, which we call A-learning. Building on current animal learning theory and machine learning, A-learning is composed of two learning equations, one for stimulus-response values and one for stimulus values (conditioned reinforcement). A third equation implements decision-making by mapping stimulus-response values to response probabilities. We show that A-learning can reproduce the main features of: instrumental acquisition, including the effects of signaled and unsignaled non-contingent reinforcement; Pavlovian acquisition, including higher-order conditioning, omission training, autoshaping, and differences in form between conditioned and unconditioned responses; acquisition of avoidance responses; acquisition and extinction of instrumental chains and Pavlovian higher-order conditioning; Pavlovian-to-instrumental transfer; Pavlovian and instrumental outcome revaluation effects, including insight into why these effects vary greatly with training procedures and with the proximity of a response to the reinforcer. We discuss the differences between current theory and A-learning, such as its lack of stimulus-stimulus and response-stimulus associations, and compare A-learning with other temporal-difference models from machine learning, such as Q-learning, SARSA, and the actor-critic model. We conclude that A-learning may offer a more convenient view of associative learning than current mathematical models, and point out areas that need further development.
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| 25. |
- Ghirlanda, Stefano, et al.
(författare)
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Coevolution of intelligence, behavioral repertoire, and lifespan
- 2014
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Ingår i: Theoretical Population Biology. - : Elsevier BV. - 0040-5809 .- 1096-0325. ; 91, s. 44-49
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Tidskriftsartikel (refereegranskat)abstract
- Across many taxa, intriguing positive correlations exist between intelligence (measured by proxy as encephalization), behavioral repertoire size, and lifespan. Here we argue, through a simple theoretical model, that such correlations arise from selection pressures for efficient learning of behavior sequences. We define intelligence operationally as the ability to disregard unrewarding behavior sequences, without trying them out, in the search for rewarding sequences. We show that increasing a species' behavioral repertoire increases the number of rewarding behavior sequences that can be performed, but also the time required to learn such sequences. This trade-off results in an optimal repertoire size that decreases rapidly with increasing sequence length. Behavioral repertoire size can be increased by increasing intelligence or lengthening the lifespan, giving rise to the observed correlations between these traits.
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