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1.
  • Spiegel, David, et al. (författare)
  • Disintegrated experience : The dissociative disorders revisited
  • 1991
  • Ingår i: Journal of Abnormal Psychology. - 0021-843X. ; 100, s. 366-378
  • Tidskriftsartikel (refereegranskat)abstract
    • We present proposed changes to the dissociative disorders section of the 4th edition of the Diagnostic and Statistical Manual of Mental Disorders and review the concept of pathological and nonpathological dissociation, including empirical findings on the relations between trauma and dissociative phenomenology and between dissociation and hypnosis. The most important proposals include the creation of two new diagnostic entities, brief reactive dissociative disorder and transient dissociative disturbance, and the readoption of the criterion of amnesia for a multiple personality disorder diagnosis. We conclude that further work on dissociative processes will provide an important link between clinical and experimental approaches to human cognition, emotion, and personality.
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3.
  • Forsman, Mats, et al. (författare)
  • Genetic effects explain the stability of psychopathic personality from mid- to late adolescence
  • 2008
  • Ingår i: Journal of Abnormal Psychology. - 0021-843X .- 1939-1846. ; 38:6, s. 627-627
  • Tidskriftsartikel (refereegranskat)abstract
    • This study examined the importance of genetic and environmental influence for the stability of psychopathic personality between mid- and late adolescence. The target sample consisted of all 1,480 male and female twin pairs born in Sweden between 1985 and 1986. Psychopathic personality was measured with the Youth Psychopathic Traits Inventory (YPI; H. Andershed, M. Kerr, M. Stattin, & S. Levander, 2002) when the participants were 16 and 19 years old. Results showed that the 3 psychopathic personality dimensions were stable at different levels of analysis and linked to a stable higher order general factor (i.e., psychopathic personality factor). Genetic factors contributed substantially to the stability of this general higher order factor, whereas environmental factors were of little importance. However, the authors also found specific genetic stability in the Callous/unemotional and Impulsive/irresponsible dimension. Thus, the model provides evidence for etiologic generality and etiologic specificity for the stability of psychopathic personality between mid- and late adolescence.
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4.
  • Hicks, B. M., et al. (författare)
  • Gender differences and developmental change in externalizing disorders from late adolescence to early adulthood : A longitudinal twin study
  • 2007
  • Ingår i: Journal of Abnormal Psychology. - Washington : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 116:3, s. 433-447
  • Tidskriftsartikel (refereegranskat)abstract
    • Using data from over 1,000 male and female twins participating in the Minnesota Twin Family Study, the authors examined developmental change, gender differences, and genetic and environmental contributions to the symptom levels of four externalizing disorders (adult antisocial behavior, alcohol dependence, nicotine dependence, and drug dependence) from ages 17 to 24. Both men and women increased in symptoms for each externalizing disorder, with men increasing at a greater rate than women, such that a modest gender gap at age 17 widened to a large one at age 24. Additionally, a mean-level gender difference on a latent Externalizing factor could account for the mean-level gender differences for the individual disorders. Biometric analyses revealed increasing genetic variation and heritability for men but a trend toward decreasing genetic variation and increasing environmental effects for women. Results illustrate the importance of gender and developmental context for symptom expression and the utility of structural models to integrate general trends and disorder-specific characteristics.
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6.
  • Krueger, R. F., et al. (författare)
  • Linking antisocial behavior, substance use, and personality : An integrative quantitative model of the adult externalizing spectrum
  • 2007
  • Ingår i: Journal of Abnormal Psychology. - Washington : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 116:4, s. 645-666
  • Tidskriftsartikel (refereegranskat)abstract
    • Antisocial behavior, substance use, and impulsive and aggressive personality traits often co-occur, forming a coherent spectrum of personality and psychopathology. In. the current research, the authors developed a novel quantitative model of this spectrum. Over 3 waves of iterative data collection, 1,787 adult participants selected to represent a range across the externalizing spectrum provided extensive data about specific externalizing behaviors. Statistical methods such as item response theory and semiparametric factor analysis were used to model these data. The model and assessment instrument that emerged from the research shows how externalizing phenomena are organized hierarchically and cover a wide range of individual differences. The authors discuss the utility of this model for framing research on the correlates and the etiology of externalizing phenomena.
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7.
  • Larsson, Henrik, 1975-, et al. (författare)
  • A genetic factor explains most of the variation in the psychopathic personality
  • 2006
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 115:2, s. 221-230
  • Tidskriftsartikel (refereegranskat)abstract
    • The psychopathic personality can be conceptualized as three interrelated dimensions, (a) an interpersonal style of glibness, grandiosity, and manipulation; (b) an affective disposition of callousness, lack of empathy, and unemotionality; and (c) a behavioral/lifestyle dimension of impulsivity, need for stimulation, and irresponsibility, underpinning a higher order construct, psychopathic personality. The authors used a self-report questionnaire (The Youth Psychopathic Traits Inventory) to study the importance of genetic and environmental influences on psychopathic personality traits in a sample of 1,090 monozygotic and dizygotic twin pairs, aged 16-17 years. Results showed a strong genetic influence behind the higher order "psychopathic personality" factor, underpinned by the three psychopathic personality dimensions. Over and above the effects to the higher order factor, significant unique genetic influences were also found in the callous/unemotional and in the impulsive/irresponsible dimension, but not in the grandiose/manipulative dimension. The authors propose that this latent psychopathic personality factor is a meaningful target for future etiological research.
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9.
  • Ruchkin, Vladislav V, et al. (författare)
  • Platelet MAO-B, personality, and psychopathology
  • 2005
  • Ingår i: Journal of Abnormal Psychology. - 0021-843X .- 1939-1846. ; 114:3, s. 477-482
  • Tidskriftsartikel (refereegranskat)abstract
    • The article investigates the relationships between platelet monoamine oxidase-B (MAO-B) activity, personality, and psychopathology (Diagnostic and Statistical Manual of Mental Disorders [4th ed.; American Psychiatric Association, 1994] diagnoses. These relationships were assessed in 178 incarcerated male juvenile delinquents. Even after controlling for smoking, the authors found that both Internalizing and Externalizing Psychopathology were negatively related to MAO-B activity. In the final reduced model, novelty seeking fully mediated the relationships between MAO-B and Externalizing Psychopathology but not between MAO-B and Internalizing Psychopathology. It was hypothesized that low platelet MAO-B activity does not directly predispose individuals to psychopathology but is related to specific personality traits, which in turn represent a vulnerability factor for psychopathology. Future studies should help clarify the nature of the relationships between personality, biological markers, and psychopathology.
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10.
  • Skeem, Jennifer, et al. (författare)
  • Two subtypes of psychopathic violent offenders that parallel primary and secondary variants
  • 2007
  • Ingår i: Journal of Abnormal Psychology. - 0021-843X .- 1939-1846. ; 116:2, s. 395-409
  • Tidskriftsartikel (refereegranskat)abstract
    • Although psychopathy usually is treated as a unitary construct, a seminal theory posits that there are 2 variants: Primary psychopathy is underpinned by an inherited affective deficit, whereas secondary psychopathy reflects an acquired affective disturbance. The authors investigated whether psychopathy phenotypically may be disaggregated into such types in a sample of 367 prison inmates convicted of violent crimes. Model-based cluster analysis of the Revised Psychopathy Checklist (PCL–R; R. D. Hare, 2003) and trait anxiety scores in the psychopathic subgroup (n = 123; PCL–R ≥ 29) revealed 2 clusters. Relative to primary psychopaths, secondary psychopaths had greater trait anxiety, fewer psychopathic traits, and comparable levels of antisocial behavior. Across validation variables, secondary psychopaths manifested more borderline personality features, poorer interpersonal functioning (e.g., irritability, withdrawal, poor assertiveness), and more symptoms of major mental disorder than primary psychopaths. When compared with the nonpsychopathic subgroup (n = 243), the 2 psychopathic variants manifested a theoretically coherent pattern of differences. Implications for etiological research and violence prevention are discussed.
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11.
  • Walters, Glenn D., et al. (författare)
  • Incremental validity of the Psychopathy Checklist facet scores : Predicting release outcome in six samples
  • 2008
  • Ingår i: Journal of Abnormal Psychology. - 0021-843X .- 1939-1846. ; 117:2, s. 396-405
  • Tidskriftsartikel (refereegranskat)abstract
    • The incremental validity of the 4 facet scores (Interpersonal, Affective, Lifestyle, Antisocial) of the Psychopathy Checklist-Revised (PCL-R; R. D. Hare, 1991, 2003) and the Psychopathy Checklist: Screening Version (PCL:SV; S. D. Hart, D. N. Cox, & R. D. Hare, 1995) was evaluated in 6 forensic/correctional samples with average follow-ups ranging from 20 weeks to 10 years. Results indicated that whereas Facet 4 (Antisocial) achieved incremental validity relative to the first 3 facets (Interpersonal, Affective, and Lifestyle) in predicting recidivism in all 6 samples, a block of the first 3 facets achieved incremental validity relative to the 4th facet in only 1 sample. Thus, although there was consistent support for the incremental validity of Facet 4 above and beyond the first 3 facets, there was minimal support for the incremental validity of Facets 1, 2, and 3 above and beyond Facet 4. The implications of these findings for the psychopathy construct in general and the PCL-R/SV in particular are discussed.
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12.
  • Baker, JH, et al. (författare)
  • Genetic risk factors for disordered eating in adolescent males and females
  • 2009
  • Ingår i: J Abnorm Psychol. - 0021-843X. ; 118:3, s. 576-86
  • Tidskriftsartikel (refereegranskat)abstract
    • The etiologic role of genetic and environmental factors on disordered eating was examined in a sample of 15- to 17-year-old female-female, male-male, and opposite-sex twin pairs. Also assessed was whether a single factor is underlying 3 facets (body dissatisfaction, drive for thinness, bulimia) of disordered eating, including the possible importance of sex differences. Univariate model-fitting analyses indicated that genetic factors are more important for girls and environment more important for boys for body dissatisfaction and drive for thinness. A multivariate common factor analysis indicated that a single factor accounted for the association among these 3 facets of disordered eating in both sexes. However, only 50% of the genetic risk for this factor is shared between the sexes.
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14.
  • Baker, Jessica H., et al. (författare)
  • Shared Familial Risk Between Bulimic Symptoms and Alcohol Involvement During Adolescence
  • 2017
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association. - 0021-843X .- 1939-1846. ; 126:5, s. 506-518
  • Tidskriftsartikel (refereegranskat)abstract
    • Twin studies show the established relation between bulimic symptoms and problematic alcohol involvement in adult females is partly due to shared familial factors, specifically shared genetic effects. However, it is unclear if similar shared etiological factors exist during adolescence or in males. We examined the familial overlap (i.e., genetic and common environmental correlations) between bulimic symptoms and various levels of alcohol involvement in 16- to 17-year-old female and male same-sex twin pairs using sex-specific biometrical twin modeling. Bulimic symptoms were assessed with the Eating Disorder Inventory-2. Alcohol involvement included alcohol use in the last month, having ever been intoxicated, and alcohol intoxication frequency. Results revealed 3 distinct patterns. First, in general, phenotypic correlations indicated statistically similar associations between bulimic symptoms and alcohol involvement in girls and boys. Second, common environmental overlap was significant for the bivariate associations including having ever been intoxicated. Third, moderate genetic correlations were observed between all bulimic symptoms and alcohol involvement in girls and moderate common environmental correlations were observed in boys for the more risky/deviant levels of involvement. Similar to adults, there is familial overlap between bulimic symptoms and alcohol involvement in adolescent girls and boys. These results could inform symptom-and sex-specific, developmentally targeted prevention and intervention programs for the comorbidity between bulimic symptoms and alcohol involvement.
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17.
  • Hedman, Erik, et al. (författare)
  • Health anxiety in a disease-avoidance framework : Investigation of anxiety, disgust and disease perception in response to sickness cues
  • 2016
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 125:7, s. 868-878
  • Tidskriftsartikel (refereegranskat)abstract
    • Severe health anxiety is characterized by a debilitating fear of somatic illness, and avoidance of disease-related stimuli plays a key role in the maintenance of the disorder. The aim of this study was to investigate severe health anxiety within an evolutionary disease-avoidance framework. We hypothesized that, compared to healthy controls, participants with severe health anxiety would perceive others as sicker, more contagious, and less attractive. We also expected individuals with severe health anxiety to be more prone to avoid interaction with persons who appeared sick, as well as to respond with more health-related worry, more disgust, and more anxiety when confronting such individuals. In addition, this sensitivity was expected to be larger if people showed manifest sickness symptoms. Participants with and without severe health anxiety (N = 224) were exposed to facial photos with a varying degree of apparent sickness. Patients with severe health anxiety, compared to controls, rated apparently healthy people as being less healthy and less attractive. There were significant interaction effects showing that that the increase in disgust, anxiety, perceived contagiousness, and worry over one's own health as a function of how sick the person in the photo appeared, was significantly larger in the clinical sample compared to the healthy control sample (ps < .047). Results from regression analyses using health anxiety as a dimensional predictor also supported our hypotheses. We suggest that disgust and cognitive biases relating to the disease-avoidance model are significant features of severe health anxiety. (PsycINFO Database Record
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18.
  • Holmes, Emily A., et al. (författare)
  • Developing Interpretation Bias Modification as a "Cognitive Vaccine" for Depressed Mood : Imagining Positive Events Makes You Feel Better Than Thinking About Them Verbally
  • 2009
  • Ingår i: Journal of Abnormal Psychology. - : AMER PSYCHOLOGICAL ASSOC. - 0021-843X .- 1939-1846. ; 118:1, s. 76-88
  • Tidskriftsartikel (refereegranskat)abstract
    • Two interpretation bias modification experiments found that mental imagery vs. verbal processing of positive material have differential emotional effects. In Experiment 1, participants were instructed to imagine positively resolved auditory descriptions or to listen to the same events while thinking about their verbal meaning. Increases in positive mood and bias were greater in the imagery than in the verbal condition, replicating E. A. Holmes, A. Mathews, T. Dalgleish, and B. Mackintosh (2006). An emotional vulnerability test showed that imagery (relative to the verbal condition) protected against a later negative mood induction. Experiment 2 created 2 new verbal conditions aimed to increase or reduce verbal comparisons. Results suggest making unfavorable comparisons with the highly positive material might be partially responsible for the inferiority of the verbal condition in Experiment 1. The findings demonstrate that imagery can play a key role in cognitive bias modification procedures and thus that task instructions are crucial. Imagining a positive event can make you feel better than thinking about the same event verbally. The authors propose that recruiting imagery will be useful in therapeutic innovations to develop a "cognitive vaccine" for depressed mood.
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19.
  • Hunsche, Michelle, et al. (författare)
  • Social functioning and the presentation of anxiety in children on the autism spectrum: A multimethod, multiinformant analysis
  • 2022
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X. ; 131:2, s. 198-208
  • Tidskriftsartikel (refereegranskat)abstract
    • Co-occurring anxiety in children on the autism spectrum is associated with greater social challenges, including poorer social skills and relationships, which may influence the severity and presentation of anxiety symptoms, particularly social anxiety. The current study used Bayesian network analytics (Williams & Mulder, 2020) and a multimethod approach to examine (a) how different facets of social functioning relate to one another and to anxiety severity and comorbidity, (b) which facet(s) are most influential and thus may represent optimal targets for intervention, and (c) how social functioning relates to the presentation of social fears in a large treatment-seeking sample of autistic children with anxiety disorders (n = 191, 7–13 years). Results indicated strong associations among measures of social ability (i.e., theory of mind [ToM], social motivation, friendship attainment) and among measures of social integration (i.e., bullying, interpersonal and peer difficulties), with only bullying demonstrating a significant association with anxiety. ToM was the most interconnected variable in the network, and social motivation demonstrated the strongest individual connections with other variables, particularly with other facets of social ability. Socially anxious children with impaired ToM were less likely to express fears of negative evaluation, resulting in a distinct diagnostic presentation of social fears. Findings suggest that social motivation and ToM may represent important targets for intervention for autistic children with co-occurring anxiety. Further, social–cognitive difficulties associated with autism, like ToM, may play a role in distinct manifestations of anxiety in these youth.
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20.
  • Kruijt, Anne-Wil, 1982-, et al. (författare)
  • A Meta-Analysis of Bias at Baseline in RCTs of Attention Bias Modification : No Evidence for Dot-Probe Bias Towards Threat in Clinical Anxiety and PTSD
  • 2019
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 128:6, s. 563-573
  • Tidskriftsartikel (refereegranskat)abstract
    • Background: Considerable effort and funding have been spent on developing Attention Bias Modification (ABM) as a treatment for anxiety disorders, theorized to exert therapeutic effects through reduction of a tendency to orient attention towards threat. However, meta-analytical evidence that clinical anxiety is characterized by threat-related attention bias is thin. The largest meta-analysis to date included dot-probe data for n=337 clinically anxious individuals. Baseline measures of biased attention obtained in ABM RCTs form an additional body of data that has not previously been meta-analyzed.Method: This paper presents a meta-analysis of threat-related dot-probe bias measured at baseline for 1005 clinically anxious individuals enrolled in 13 ABM RCTs.Results: Random-effects meta-analysis indicated no evidence that the mean bias index (BI) differed from zero (k= 13, n= 1005, mean BI = 1.8 ms, SE = 1.26 ms, p = .144, 95% CI [-0.6 - 4.3]. Additional Bayes factor analyses also supported the point-zero hypothesis (BF10 = .23), whereas interval-based analysis indicated that mean bias in clinical anxiety is unlikely to extend beyond the 0 to 5 ms interval. Discussion: Findings are discussed with respect to strengths (relatively large samples, possible bypassing of publication bias), limitations (lack of control comparison, repurposing data, specificity to dot-probe data), and theoretical and practical context. We suggest that it should no longer be assumed that clinically anxious individuals are characterized by selective attention towards threat.Conclusion: Clinically anxious individuals enrolled in RCTs for Attention Bias Modification are not characterized by threat-related attention bias at baseline.
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21.
  • Laursen, Brett, et al. (författare)
  • Friend influence over adolescent problem behaviors as a function of relative peer acceptance : to be liked is to be emulated
  • 2012
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 121:1, s. 88-94
  • Tidskriftsartikel (refereegranskat)abstract
    • Friend influence over alcohol intoxication and delinquent behavior was examined as a function of relative peer acceptance in a 3-year study of Swedish youth (N = 184 girls, 145 boys). Participants were in the first year of secondary school (7th grade, M = 11.7 years old) or the first year of high school (10th grade, M = 15.3 years old) at the outset. Friends resembled one another before the friendship; resemblances were even greater after the friendship began. Resemblances continued to grow among those who remained friends one year later, but declined among those whose friendships dissolved. Partners were not equally responsible for increases in similarity. In stable friendships, the more accepted partner exerted greater influence over the less accepted partner, such that the greatest increases in problem behaviors were found among less accepted youth whose friends had higher initial levels of delinquency and alcohol intoxication. Unstable friends resembled random pairs of youth in that more- and less-accepted partners were comparably uninfluential.
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23.
  • Narusyte, Jurgita, et al. (författare)
  • Parental criticism and externalizing behavior problems in adolescents : the role of environment and genotype-environment correlation
  • 2011
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 120:2, s. 365-376
  • Tidskriftsartikel (refereegranskat)abstract
    • Genetic factors are important for the association between parental negativity and child problem behavior, but it is not clear whether this is due to passive or evocative genotype environment correlation (rGE). In this study, we applied the extended children-of-twins model to directly examine the presence of passive and evocative rGE as well as direct environmental effects in the association between parental criticism and adolescent externalizing problem behavior. The cross-sectional data come from the Twin and Offspring Study in Sweden (N = 909 pairs of adult twins) and from the Twin Study of Child and Adolescent Development (N = 915 pairs of twin children). The results revealed that maternal criticism was primarily due to evocative rGE emanating from their adolescent’s externalizing behavior. On the other hand, fathers’ critical remarks tended to affect adolescent problem behavior in a direct environmental way. This suggests that previously reported differences in caretaking between mothers and fathers also are reflected in differences in why parenting is associated with externalizing behavior in offspring.
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24.
  • O'Reilly, L. M., et al. (författare)
  • The Association Between General Childhood Psychopathology and Adolescent Suicide Attempt and Self-Harm: A Prospective, Population-Based Twin Study
  • 2020
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 129:4, s. 364-375
  • Tidskriftsartikel (refereegranskat)abstract
    • Few quantitative behavior genetic studies have examined why psychopathology is associated with suicide attempt (SA) and self-harm (SH) in adolescence. The present study analyzed data from the Child and Adolescent Twin Study in Sweden to examine the extent to which genetic and environmental factors explain SA/SH and its association with psychopathology in childhood, an often-cited risk factor of subsequent SA/SH. When children were 9 or 12 years old (n = 30,444), parents completed the Autism-Tics, AD/HD and other Comorbidities Inventory (Larson et al., 2010) regarding their children's psychiatric problems as part of an ongoing, longitudinal study. At age 18 years (n = 10,269), adolescents completed self-report questionnaires, including SA/SH assessments. In a bifactor model of childhood psychopathology, a general factor of psychopathology was a statistically significant predictor of adolescvnt SA/SH at a higher magnitude (beta, 0.25, 95% confidence interval [CI; 0.15, 0.34] for suicide attempt), as compared with specific factors of inattention, impulsivity, oppositional behavior, and anxiety/emotion symptoms. Quantitative genetic modeling indicated that the additive genetic influences on the general factor accounted for the association with each outcome (beta, 0.24, 95% CI [0.13, 0.34] for suicide attempt). The results remained virtually identical when we fit a higher order factors model. Two additional outcomes demonstrated comparable results. The results extend current literature by revealing the shared genetic overlap between general psychopathology during childhood and adolescent SA/SH.
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26.
  • Patrick, C. J., et al. (författare)
  • A construct-network approach to bridging diagnostic and physiological domains : Application to assessment of externalizing psychopathology
  • 2013
  • Ingår i: Journal of Abnormal Psychology. - Washington : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 122:3, s. 902-916
  • Tidskriftsartikel (refereegranskat)abstract
    • A crucial challenge in efforts to link psychological disorders to neural systems, with the aim of developing biologically informed conceptions of such disorders, is the problem of method variance (Campbell & Fiske, 1959). Since even measures of the same construct in differing domains correlate only moderately, it is unsurprising that large sample studies of diagnostic biomarkers yield only modest associations. To address this challenge, a construct-network approach is proposed in which psychometric operationalizations of key neurobehavioral constructs serve as anchors for identifying neural indicators of psychopathology-relevant dispositions, and as vehicles for bridging between domains of clinical problems and neurophysiology. An empirical illustration is provided for the construct of inhibition disinhibition, which is of central relevance to problems entailing deficient impulse control. Findings demonstrate that: (1) a well-designed psychometric index of trait disinhibition effectively predicts externalizing problems of multiple types, (2) this psychometric measure of disinhibition shows reliable brain response correlates, and (3) psychometric and brain-response indicators can be combined to form a joint psychoneurometric factor that predicts effectively across clinical and physiological domains. As a methodology for bridging between clinical problems and neural systems, the construct-network approach provides a concrete means by which existing conceptions of psychological disorders can accommodate and be reshaped by neurobiological insights.
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27.
  • Pennington, Bruce, et al. (författare)
  • Individual prediction of dyslexia by single vs. multiple deficit models.
  • 2012
  • Ingår i: Journal of Abnormal Psychology. - : American Psychological Association. - 0021-843X .- 1939-1846. ; 121:1, s. 212-224
  • Tidskriftsartikel (refereegranskat)abstract
    • The overall goals of this study were to test single versus multiple cognitive deficit models of dyslexia (reading disability) at the level of individual cases and to determine the clinical utility of these models for prediction and diagnosis of dyslexia. To accomplish these goals, we tested five cognitive models of dyslexia-two single-deficit models, two multiple-deficit models, and one hybrid model-in two large population-based samples, one cross-sectional (Colorado Learning Disability Research Center) and one longitudinal (International longitudinal Twin Study). The cognitive deficits included in these cognitive models were in phonological awareness, language skill, and processing speed and/or naming speed. To determine whether an individual case fit one of these models, we used two methods: 1) the presence or absence of the predicted cognitive deficits, and 2) whether the individuals level of reading skill best fit the regression equation with the relevant cognitive predictors (i.e., whether their reading skill was proportional to those cognitive predictors.) We found that roughly equal proportions of cases met both tests of model fit for the multiple deficit models (30-36%) and single deficit models (24-28%); hence, the hybrid model provided the best overall fit to the data. The remaining roughly 40% of cases in each sample lacked the deficit or deficits that corresponded with their best-fitting regression model. We discuss the clinical implications of these results for both diagnosis of school-age children and preschool prediction of children at risk for dyslexia.
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28.
  • Pisetsky, EM, et al. (författare)
  • Suicide attempts in women with eating disorders
  • 2013
  • Ingår i: Journal of abnormal psychology. - : American Psychological Association (APA). - 1939-1846 .- 0021-843X. ; 122:4, s. 1042-1056
  • Tidskriftsartikel (refereegranskat)
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29.
  • Ronald, A., et al. (författare)
  • Symptoms of Autism and ADHD: A Swedish Twin Study Examining Their Overlap
  • 2014
  • Ingår i: Journal of Abnormal Psychology. - Washington DC, USA : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 123:2, s. 440-451
  • Tidskriftsartikel (refereegranskat)abstract
    • Autism spectrum disorders (ASD) and attention deficit hyperactivity disorder (ADHD) show high comorbidity. The following questions were addressed regarding their specific symptoms: What is the factor structure of ASD and ADHD symptoms, to what degree do different symptom domains cluster together, to what extent are these domains caused by the same genetic and environmental influences, and what is the best model of their co-occurrence? A population-based twin cohort of over 17,000 9- and 12-year-olds were assessed using the Autism-Tics, AD/HD, and other Comorbidities parental interview inventory. Principal component analyses were conducted, and symptom domain clustering was assessed. Four multivariate twin models were compared. Factors split into three ASD (social impairments, communication impairments, and restricted repetitive behaviors and interests), and three ADHD (inattention, hyperactivity, and impulsivity) symptom domains. Some ASD-ADHD symptom domain combinations clustered together often, although others not at all. A two-factor common pathway model fit the data, suggesting that ASD and ADHD symptom domains tap into separate "ASD" and "ADHD" latent factors that showed high genetic overlap. All subdomains also showed significant specific genetic and environmental influences, reflecting the etiological heterogeneity both within and between ASD and ADHD. These findings support the conceptual distinction of ASD and ADHD, and demonstrate the considerable natural co-occurrence of particular ASD/ADHD symptom domains. The results imply that more children with 1 condition show features of the other condition than show complete comorbidity. Emphasis on symptom co-occurrence, rather than complete comorbidity between disorders, may help focus clinical approaches and advance molecular genetic research.
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30.
  • Soares, Joaquim, 1947-, et al. (författare)
  • On the automatic nature of phobic fear: Conditioned electrodermal responses to masked fear-relevant stimuli
  • 1993
  • Ingår i: Journal of Abnormal Psychology. - 0021-843X .- 1939-1846. ; 102:1, s. 121-132
  • Tidskriftsartikel (refereegranskat)abstract
    • Normal subjects (n = 64) were exposed either to pictures of snakes and spiders or to pictures of flowers and mushrooms in a differential conditioning paradigm in which one of the pictures signaled an electric shock. In a subsequent extinction series, these stimuli were presented backwardly masked by another stimulus for half of the subjects, whereas the other half received non-masked extinction. In support of a hypothesis that suggests that nonconscious information-processing mechanims are sufficient to activate responses to fear-relevant stimuli, differential skin conductance response to masked conditioning and control stimuli was obvious only for subjects conditioned to fear-relevant stimuli. These results were replicated in a second experiment (n = 32), which also demonstrated that the effect was unaffected by which visual half-field was used for stimulus presentation.
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33.
  • Öhman, A, et al. (författare)
  • Unconscious anxiety: Phobic responses to masked stimuli
  • 1994
  • Ingår i: Journal of Abnormal Psychology. - 0021-843X .- 1939-1846. ; 103:2, s. 231-240
  • Tidskriftsartikel (refereegranskat)abstract
    • We tested the hypothesis that an unconscious preattentive perceptual analysis of phobic stimuli is sufficient to elicit human fear responses. Selected snake- and spider-fearful Ss, as well as normal controls, were exposed to pictures of snakes, spiders, flowers, and mushrooms. A separate forced-choice recognition experiment established backward masking conditions that effectively precluded recognition of experimental stimuli both for fearful and nonfearful Ss. In the main experiment, these conditions were used to compare skin conductance responses (SCRs) to masked and nonmasked phobic and control pictures among fearful and nonfearful Ss. In support of the hypotheses, snake- and spider-fearful Ss showed elevated SCRs to snake and spider pictures as compared with neutral pictures and with responses of the nonfearful Ss under both masking conditions. Ratings of valence, arousal, and dominance indicated that the fearful Ss felt more negative, more aroused, and less dominant in relation to both masked and nonmasked phobic stimuli.
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