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Sökning: WFRF:(Jacobsson Frida)

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1.
  • Aabel, Lise, et al. (författare)
  • A TDD Distributed MIMO Testbed Using a 1-bit Radio-Over-Fiber Fronthaul Architecture
  • 2024
  • Ingår i: IEEE Transactions on Microwave Theory and Techniques. - 0018-9480 .- 1557-9670. ; In Press
  • Tidskriftsartikel (refereegranskat)abstract
    • We present the uplink and downlink of a time-division duplex distributed multiple-input multiple-output (D-MIMO) testbed, based on a 1-bit radio-over-fiber architecture, which is low cost and scalable. The proposed architecture involves a central unit (CU) that is equipped with 1-bit digital-to-analog and analog-to-digital converters, operating at 10 GS/s. The CU is connected to multiple single-antenna remote radio heads (RRHs) via optical fibers, over which a binary radio frequency (RF) waveform is transmitted. In the uplink, a binary RF waveform is generated at the RRHs by a comparator, whose inputs are the received RF signal and a suitably designed dither signal. In the downlink, a binary RF waveform is generated at the CU via bandpass sigma-delta modulation. Our measurement results show that low error-vector magnitude (EVM) can be achieved in both the uplink and the downlink, despite 1-bit sampling at the CU. Specifically, for point-to-point over-cable transmission between a single user equipment (UE) and a CU equipped with a single RRH, we report, for a 10-MBd signal using single-carrier (SC) 16 quadratic-amplitude modulation (QAM) modulation, an EVM of 3.3% in the downlink, and of 4.5% in the uplink. We then consider a CU connected to three RRHs serving over the air two UEs, and show that, after over-the-air reciprocity calibration, a downlink zero-forcing precoder designed on the basis of uplink channel estimates at the CU achieves an EVM of 6.4% and 10.9% at UE 1 and UE 2, respectively. Finally, we investigate the ability of the proposed architecture to support orthogonal frequency-division multiplexing (OFDM) waveforms, and its robustness against both in-band and out-of-band interference.
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2.
  • Meeuwisse, Anna, et al. (författare)
  • "Statlig kunskapsstyrning" - en exkluderingsprocess
  • 2017
  • Ingår i: Den motspänstiga akademikern : Festskrift till Ingrid Sahlin - Festskrift till Ingrid Sahlin. - 9789198131789 ; , s. 107-134
  • Bokkapitel (övrigt vetenskapligt/konstnärligt)
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3.
  • Torinsson Naluai, Åsa, 1968, et al. (författare)
  • Transcriptomics unravels molecular changes associated with cilia and COVID-19 in chronic rhinosinusitis with nasal polyps
  • 2023
  • Ingår i: Scientific Reports. - : Springer Nature. - 2045-2322. ; 13:1
  • Tidskriftsartikel (refereegranskat)abstract
    • Chronic rhinosinusitis with nasal polyps (CRSwNP) is a common upper respiratory tract complication where the pathogenesis is largely unknown. Herein, we investigated the transcriptome profile in nasal mucosa biopsies of CRSwNP patients and healthy individuals. We further integrated the transcriptomics data with genes located in chromosomal regions containing genome-wide significant gene variants for COVID-19. Among the most significantly upregulated genes in polyp mucosa were CCL18, CLEC4G, CCL13 and SLC9A3. Pathways involving "Ciliated epithelial cells" were the most differentially expressed molecular pathways when polyp mucosa and non-polyp mucosa from the same patient was compared. Natural killer T-cell (NKT) and viral pathways were the most statistically significant pathways in the mucosa of CRSwNP patients compared with those of healthy control individuals. Upregulated genes in polyp mucosa, located within the genome-wide associated regions of COVID-19, included LZTFL1, CCR9, SLC6A20, IFNAR1, IFNAR2 and IL10RB. Interestingly, the second most over-expressed gene in our study, CLEC4G, has been shown to bind directly to SARS-CoV-2 spike's N-terminal domain and mediate its entry and infection. Our results on altered expression of genes related to cilia and viruses point to the de-regulation of viral defenses in CRSwNP patients, and may give clues to future intervention strategies.
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  • Resultat 1-3 av 3

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