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Träfflista för sökning "WFRF:(Monath Thomas) "

Sökning: WFRF:(Monath Thomas)

  • Resultat 1-3 av 3
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1.
  • Jentes, Emily S, et al. (författare)
  • The revised global yellow fever risk map and recommendations for vaccination, 2010 : consensus of the Informal WHO Working Group on geographic risk for Yellow Fever.
  • 2011
  • Ingår i: The Lancet - Infectious diseases. - : Elsevier. - 1473-3099 .- 1474-4457. ; 11:8, s. 622-632
  • Tidskriftsartikel (refereegranskat)abstract
    • The changing epidemiology of yellow fever and continued reports of rare but serious adverse events associated with yellow fever vaccine have drawn attention to the need to revisit criteria for the designation of areas with risk for yellow fever virus activity, and to revise the vaccine recommendations for international travel. WHO convened a working group of international experts to review factors important for the transmission of yellow fever virus and country-specific yellow fever information, to establish criteria for additions to or removal from the list of countries with risk for yellow fever virus transmission, to update yellow fever risk maps, and to revise the recommendations for vaccination for international travel. This report details the recommendations made by the working group about criteria for the designation of risk and specific changes to the classification of areas with risk for transmission of yellow fever virus.
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2.
  • Khadraoui, Younes, et al. (författare)
  • On connection control and traffic optimisation in FMC networks
  • 2016
  • Ingår i: 2015 IEEE 16th International Conference on High Performance Switching and Routing, HPSR 2015. - 9781479998715 ; 2016-June
  • Konferensbidrag (refereegranskat)abstract
    • With the introduction of smartphones and tablets and the dramatical increase of the data traffic on the cellular network, operators are seeking for new solutions to provide the users with continuous connectivity while preserving a good quality of service. Since most of current user terminals are equipped with multiple interfaces, one solution is to take advantage of the different access networks. However, with the current network architectures and user application, it is not possible to use both accesses at the same time or even to switch from one to another without loosing current sessions. In this paper, we present and analyse the main mechanisms of the literature that allow a user to use multiple access networks at the same time or to move between the different networks. We show how a converged network can permit the deployment of new solutions and present some of them accompanied by use cases.
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3.
  • Vianello, Eleonora, et al. (författare)
  • Global blood miRNA profiling unravels early signatures of immunogenicity of Ebola vaccine rVSVΔG-ZEBOV-GP
  • 2023
  • Ingår i: iScience. - 2589-0042. ; 26:12
  • Tidskriftsartikel (refereegranskat)abstract
    • The vectored Ebola vaccine rVSVΔG-ZEBOV-GP elicits protection against Ebola Virus Disease (EVD). In a study of forty-eight healthy adult volunteers who received either the rVSVΔG-ZEBOV-GP vaccine or placebo, we profiled intracellular microRNAs (miRNAs) from whole blood cells (WB) and circulating miRNAs from serum-derived extracellular vesicles (EV) at baseline and longitudinally following vaccination. Further, we identified early miRNA signatures associated with ZEBOV-specific IgG antibody responses at baseline and up to one year post-vaccination, and pinpointed target mRNA transcripts and pathways correlated to miRNAs whose expression was altered after vaccination by using systems biology approaches. Several miRNAs were differentially expressed (DE) and miRNA signatures predicted high or low IgG ZEBOV-specific antibody levels with high classification performance. The top miRNA discriminators were WB-miR-6810, EV-miR-7151-3p, and EV-miR-4426. An eight-miRNA antibody predictive signature was associated with immune-related target mRNAs and pathways. These findings provide valuable insights into early blood biomarkers associated with rVSVΔG-ZEBOV-GP vaccine-induced IgG antibody responses.
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