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Sökning: WFRF:(Sneddon Scott)

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  • Sneddon, Scott, et al. (författare)
  • Precision improvements in ECM via tool insert development by 3D printing
  • 2022
  • Ingår i: 21st CIRP CONFERENCE ON ELECTRO PHYSICAL AND CHEMICAL MACHINING, ISEM XXI June, 14 to 17, 2022 in Zurich. - : Elsevier. ; , s. 459-464
  • Konferensbidrag (refereegranskat)abstract
    • This paper reports the tool developments for electrochemical machining (ECM) of precision parts for automotive industry using polymer3D printed inserts. The role of these inserts is to control the fluid flow in the inter-electrode gap as well as to prevent stray machiningby providing insulation. Initial trial and error machining experiments with various tool insert shapes yielded positive outcomes in terms ofcontrolling stray dissolution. It became evident that by 3D printing the entire machining cell structure with integrated, exchangeable toolinserts, all made from polymer, can further enhance the ECM process. This cell-insert assembly enabled better control of the electrochemicaldissolution process and allowed for less restrictive flow of the electrolyte, reducing turbulence in the inter-electrode gap. Thus, by improvingthe electrolyte flow conditions and providing appropriate insulation to prevent stray machining, the 3D printed cell/insert assemblies enabledECM to achieve a shaped profile of an auto engine component to required tolerances.
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  • Zody, Michael, 1968-, et al. (författare)
  • Analysis of the DNA sequence and duplication history of human chromosome 15
  • 2006
  • Ingår i: Nature. - : Springer Science and Business Media LLC. - 0028-0836 .- 1476-4687. ; 440:7084, s. 671-675
  • Tidskriftsartikel (refereegranskat)abstract
    • Here we present a finished sequence of human chromosome 15, together with a high-quality gene catalogue. As chromosome 15 is one of seven human chromosomes with a high rate of segmental duplication, we have carried out a detailed analysis of the duplication structure of the chromosome. Segmental duplication in chromosome 15 are largely clustered in two regions, on proximal and distal 15q; the proximal region is notable because recombination among the segmental duplications can result in deletions causing Prader-Willi and Angelman syndromes. Sequence analysis shows that the proximal and distal regions of 15q share extensive ancient similarity. Using a simple approach, we have been able to reconstruct many of the events by which the current duplication structure arose. We find that most of the intrachromosomal duplications seem to share a common ancestry. Finally, we demonstrate that some remaining gaps in the genome sequence are probably due to structural polymorphisms between haplotypes; this may explain a significant fraction of the gaps remaining in the human genome.
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