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1.	Danielsson, B, et al. (author) Drug induced embryonic cardiac arrhythmia: A teratogenic mechanism of human relevance 2012 In: TOXICOLOGY LETTERS. - : Elsevier BV. - 0378-4274. ; 211, s. S186-S186 Conference paper (other academic/artistic)
2.	Danielsson, C, et al. (author) Is Cardiac Arrhythmia in the Human Embryo a Possible Mechanism for Cardiovascular Defects by I-Kr Blocking Antidepressants? 2011 In: BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY. - 1542-0752. ; 91:5, s. 345-345 Conference paper (other academic/artistic)
3.	Danielsson, C, et al. (author) Is Cardiac Arrhythmia in the Human Embryo a Possible Mechanism for Cardiovascular Defects by I-Kr Blocking Antidepressants? in BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY, vol 91, issue 5, pp 345-345 2011 In: BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY. - : WILEY-BLACKWELL, COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA. ; , s. 345-345 Conference paper (peer-reviewed)abstract n/a
4.	Fletcher, N, et al. (author) 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) alters the mRNA expression of critical genes associated with cholesterol metabolism, bile acid biosynthesis, and bile transport in rat liver: a microarray study 2005 In: Toxicology and applied pharmacology. - : Elsevier BV. - 0041-008X. ; 207:1, s. 1-24 Journal article (peer-reviewed)
5.	Glinghammar, B, et al. (author) Proliferative and molecular effects of the dual PPARalpha/gamma agonist tesaglitazar in rat adipose tissues: relevance for induction of fibrosarcoma 2011 In: Toxicologic pathology. - : SAGE Publications. - 1533-1601 .- 0192-6233. ; 39:2, s. 325-336 Journal article (peer-reviewed)abstract The dual peroxisome-proliferator-activated receptor (PPAR) α/γ agonist tesaglitazar has been shown to produce fibrosarcomas in rats. Here, the authors studied morphology, proliferation, differentiation, and inflammation markers in adipose tissue from rats exposed to 1, 3, or 10 µmol/kg tesaglitazar for 2 or 12 weeks, including recovery groups (12 weeks treatment followed by 12 weeks recovery), and 3 or 10 µmol/kg tesaglitazar for 24 weeks. Subcutaneous white and brown fat revealed reversible dose-related histopathological alterations and after 12 and 24 weeks developed areas of thickened skin (fatty lumps). There was a dose-dependent increase in proliferation of interstitial cells in white and brown fat as shown by increased mitotic index in all dose groups after 2 weeks. This was limited to the high dose after 12 and 24 weeks in white fat. Gene expression analyses showed that while tesaglitazar induced differentiation of adipose tissue characterized with a switch in cyclin D1 and D3 mRNA by 12 weeks, longer exposure at high doses reversed this differentiation concurrent with a reappearance of early adipocyte and inflammatory markers. These data suggest that sustained increased turnover of mesenchymal cells in adipose tissues, concomitant with onset of inflammation and fibrosis, drives development of fibrosarcomas in rats treated with tesaglitazar.
6.	Skold, A C, et al. (author) Translational Research: Electrophysiological Development in the Embryonic Heart of Rats, Rabbits, and Humans in BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY, vol 91, issue 5, pp 357-357 2011 In: BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY. - : WILEY-BLACKWELL, COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA. ; , s. 357-357 Conference paper (peer-reviewed)abstract n/a
7.	Skold, AC, et al. (author) Translational Research: Electrophysiological Development in the Embryonic Heart of Rats, Rabbits, and Humans 2011 In: BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY. - 1542-0752. ; 91:5, s. 357-357 Conference paper (other academic/artistic)
8.	Sun, Y., et al. (author) Short-Term Embryonic Stem Cell Exposure Assay As a Tool to Predict Developmental Toxicity 2014 In: Birth defects research. Clinical and molecular teratology. - 1542-0752 .- 1542-0760. ; 100:5, s. 402-402 Journal article (other academic/artistic)
9.	Thulin, P, et al. (author) PPARalpha regulates the hepatotoxic biomarker alanine aminotransferase (ALT1) gene expression in human hepatocytes 2008 In: Toxicology and applied pharmacology. - : Elsevier BV. - 0041-008X. ; 231:1, s. 1-9 Journal article (peer-reviewed)

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Result 1-9 of 9

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Type of publication: conference paper (5); journal article (4)

Type of content: peer-reviewed (5); other academic/artistic (4)

Author/Editor: Hellmold, H (9); Stockling, K (9); Sylven, C (5); Danielsson, C. (5); Pehrson, R (5); Andersson, A (4); show more...; Elinder, F (3); Brask, J (3); Danielsson, B. (3); Blomgren, B (3); Skold, AC (3); Larsson, M (2); Andersson, U (2); Elinder, Fredrik (2); Brask, Johan (2); Lindstrom, A (2); Glinghammar, B (2); Jalkesten, E (2); Grebius, M (2); Sun, Y (1); Skanberg, I (1); Salter, H (1); Cotgreave, I (1); Ehrenborg, E (1); Hakansson, H (1); Dencker, Lennart (1); Stigson, Michael (1); DANIELSSON, BR (1); Thulin, P (1); Berg, AL (1); Jergil, Måns (1); Rafter, I (1); Salari, Sajjad (1); Sköld, A-C (1); Danielsson, B. R. (1); Norjavaara, E. (1); Wahlstrom, D (1); Fletcher, N (1); Lundberg, R (1); Nilsson, CB (1); Nilsson, KC (1); Bjurstrom, S (1); Westerberg, R (1); Skold, A C (1); Salari, S (1); Gustafson, A-L (1); Tomkiewicz, C (1); Aggerbeck, M (1); show less...

University: Karolinska Institutet (7); Linköping University (2); Uppsala University (1)

Language: English (9)

Research subject (UKÄ/SCB): Natural sciences (1); Medical and Health Sciences (1)

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