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| 2. |
- Fujita, Y, et al.
(författare)
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Evidence for the existence of the [202]3/2 deformed band in mirror nuclei Mg-25 and Al-25
- 2004
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Ingår i: Physical Review Letters. - 1079-7114. ; 92:6
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Tidskriftsartikel (refereegranskat)abstract
- After 50 years of its prediction, the highest-lying [2 0 2]3/2 orbit among the six Nilsson single-particle orbits originating from the sd shells in prolately deformed nuclei and the rotational band on this orbit were identified. The band members were observed in Al-25 at excitation energies of 6-7.5 MeV in a high-resolution Mg-25(He-3,t) charge-exchange reaction at 0degrees having a strong selectivity for Gamow-Teller transitions. In the comparison with the analogous M1 transitions in Mg-25, the J(pi)=3/2(+) bandhead state and the excited 5/2(+) and 7/2(+) members were clearly assigned.
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| 3. |
- Margolis, Russell L, et al.
(författare)
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Huntington's Disease-like 2 (HDL2) in North America and Japan.
- 2004
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Ingår i: Annals of Neurology. - : Wiley. - 0364-5134 .- 1531-8249. ; 56:5, s. 670-4
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Tidskriftsartikel (refereegranskat)abstract
- Huntington's Disease-like 2 (HDL2) is a progressive, autosomal dominant, neurodegenerative disorder with marked clinical and pathological similarities to Huntington's disease (HD). The causal mutation is a CTG/CAG expansion mutation on chromosome 16q24.3, in a variably spliced exon of junctophilin-3. The frequency of HDL2 was determined in nine independent series of patients referred for HD testing or selected for the presence of an HD-like phenotype in North America or Japan. The repeat length, ancestry, and age of onset of all North American HDL2 cases were determined. The results show that HDL2 is very rare, with a frequency of 0 to 15% among patients in the nine case series with an HD-like presentation who do not have the HD mutation. HDL2 is predominantly, and perhaps exclusively, found in individuals of African ancestry. Repeat expansions ranged from 44 to 57 triplets, with length instability in maternal transmission detected in a repeat of r2=0.29, p=0.0098). The results further support the evidence that the repeat expansion at the chromosome 16q24.3 locus is the direct cause of HDL2 and provide preliminary guidelines for the genetic testing of patients with an HD-like phenotype.
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| 4. |
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| 5. |
- Shimbara, Y, et al.
(författare)
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Suppression of Gamow-Teller and M1 transitions in deformed mirror nuclei Mg-25 and Al-25 - Direct observation of K selection rules
- 2004
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Ingår i: European Physical Journal A. Hadrons and Nuclei. - : Springer Science and Business Media LLC. - 1434-6001. ; 19:1, s. 25-31
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Tidskriftsartikel (refereegranskat)abstract
- The mirror nuclei Mg-25 and Al-25 are expected to have very similar structures. The Gamow-Teller (GT) transitions from the J(pi) = 5/2(+) ground state of Mg-25 to the excited states in Al-25 were studied by high-resolution measurements of the Mg-25(He-3,t) charge-exchange reaction at 0degrees and at 140 MeV/nucleon. Assuming the usual DeltaJ(pi) = 1(+) selection rule for the spin-isospin-type GT transitions, the states with J(pi) = 3/2(+), 5/2(+), and 7/2 (+) should be excited. However, of the more than ten states with these J(pi) values below 6 MeV excitation energy, only the 5/2 (+) ground state and the 7/2 (+) , 1.613 MeV state in Al-25 were strongly populated, while all other states were strongly suppressed. The analysis of M1 transitions in Mg-25 also suggested a very similar feature for the analogous M1 transitions. Both Mg-25 and Al-25 are known to be largely deformed, and most low-lying states can be interpreted in terms of one-particle quantum numbers in the deformed potential and the associated rotational spectra. The observed suppression can be explained in terms of the K quantum number selection rules that are inherent to axially deformed nuclei.
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