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- Andersson, Berit, et al.
(författare)
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Fire Behaviour of Upholstered Furniture - An Experimental Study
- 1982
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Rapport (övrigt vetenskapligt/konstnärligt)abstract
- The first and largely experimental part of a research program on flammability characteristics of upholstered furniture has been completed with full-scale tests in an extensively instrumented burn-out room as main element. The results indicate that fire response parameters vary within a large range with choice of filling, interlining and fabric material. Selection of performance criteria may cause difficulties, as parameters like heat release and smoke potential fluctuate strongly during the fire process. A second part of the program will be directed towards measurement of heat release by oxygen consumption methods, more detailed smoke and gas analysis measurements and use of mathematical simulation procedures.
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- Andersson, Berit, et al.
(författare)
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Fire Behaviour of Upholstered Furniture – An Experimental Study
- 1985
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Ingår i: Fire and Materials. - : Wiley. - 1099-1018 .- 0308-0501. ; 9:1, s. 41-45
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Tidskriftsartikel (refereegranskat)abstract
- A series of experiments for studies of flammability characteristics of upholstered furniture is presented. The series comprises 53 experiments on a reduced scale and 11 full-scale experiments in an extensively instrumented room. Results are presented from measurements of rate of heat release and smoke production. The experiments indicate that ignitability can be considerably decreased by careful choice of the upholstery materials.
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- Andersson, Sven B, 1952, et al.
(författare)
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N2O-Emissions from Fluidized Bed Combustion
- 1988
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Ingår i: Proceedings of IEA AFBC Technical Meting held in November 1988 in Amsterdam, The Netherlands.
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Konferensbidrag (övrigt vetenskapligt/konstnärligt)abstract
- Nitrous oxide, N20, is formed during combustion. In flames the N20 is removed because of the fast destruction at high temperatures. At the temperatures of fluidized bed combustion the destruction is much slower and N20 will leave the system, normally at a concentration of 50-200 ppm when burning coal. Using the results of the preliminary model in a CFB, it can be assumed that:- N20 is formed from char nitrogen in the bottom bed because of reactions in the pores of the particle. - Only some N20 is formed when NO is reduced during its journey up through the combustion chamber. - N20 is formed from HCN. This is a slow process (taking seconds) and it therefore takes place principally above the bottom bed.
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| 10. |
- Andersson, Sven
(författare)
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Cardiovascular responses to intravenous calcitonin gene-related peptide (CGRP) in the albino rabbit
- 1989
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Ingår i: Acta Physiologica Scandinavica. - 0001-6772. ; 137:2, s. 279-290
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Tidskriftsartikel (refereegranskat)abstract
- The regional sensitivity of different vascular beds to i.v. CGRP was investigated in the albino rabbit by using the microsphere method. Experiments were performed without pre-treatment on both conscious and pentobarbital-anaesthetized animals. In addition, in one series on conscious animals, rabbits were pre-treated with indomethacin in order to reduce the formation of prostaglandins. In another series, anaesthetized rabbits were subjected to ganglionic blockade with hexamethonium bromide in order to abolish reflexes involving the autonomic nervous system. 120 pmol kg-1 of CGRP was given to all the animals, the conscious animals receiving the peptide in one infusion lasting 4 min. In the anaesthetized animals, the dose was divided into first a 5-min infusion of 30 pmol kg-1 followed some minutes later by a 3-min infusion of 90 pmol kg-1. The most pronounced vasodilatory effects were seen in the pancreas, gallbladder, stomach, duodenum, tongue, teeth and the conjunctiva/nictitating membrane. In some series marked effects were also seen in the dura mater, choroid plexus and some parts of the brain. In the anaesthetized animals almost no statistically significant effects on local blood flows were seen following the first, smaller, dose, but following the larger dose more pronounced effects were observed. Pre-treatment with indomethacin did not to any great extent affect the responses, which contradicts the involvement of prostaglandins. The pattern of the responses was unaffected by the ganglionic blockade, but the variability of response was reduced. In conclusion there are great regional variations in the sensitivity to circulating CGRP. The patterns shows a resemblance to that obtained in other species, but there are some marked differences, The tissues most susceptible to the peptide are those easily exposed to noxious stimuli and containing CGRP in the sensory nerve endings, observations in agreement with the proposed role for the peptide in neurogenic defence mechanisms.
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