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- Chen, Li, et al.
(författare)
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Internet-Enabled Real-Time Collaborative Assembly Modeling via An e-Assembly System: Status and Promise
- 2004
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Ingår i: Computer-Aided Design. - : Elsevier BV. - 0010-4485 .- 1879-2685. ; 36:9, s. 835-847
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Tidskriftsartikel (refereegranskat)abstract
- Collaborative CAD systems enabling collaboration in computer-aided design processes among distributed designers are gaining more and more attention. Yet, such systems, especially in support of collaborative assembly modeling, are hardly achievable. Targeting this gap, this paper addresses an Internet-enabled real-time collaborative assembly modeling system, named e-Assembly. This emerging system allows a group of geographically dispersed designers to jointly build an assembly model in real time over the Internet. In particular, this paper proposes a new assembly representation, called Collaborative Assembly Representation, for Internet-based collaborative assembly modeling. Also, collaborative assembly constraint satisfaction is addressed based on three coordination rules embedded in e-Assembly. Furthermore, the system architecture and realization of e-Assembly are provided. Finally, a prototypic implementation of e-Assembly is presented for demonstration and discussion.
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| 3. |
- Chen, Yun, 1966, et al.
(författare)
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Neonatal losartan treatment suppresses renal expression of molecules involved in cell-cell and cell-matrix interactions
- 2004
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Ingår i: Journal of the American Society of Nephrology. - : Lippincott Williams & Wilkins. - 1046-6673 .- 1533-3450. ; 15:5, s. 1232-43
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Tidskriftsartikel (refereegranskat)abstract
- Lack of neonatal angiotensin II type 1 receptor (AT(1)) stimulation produces renal abnormalities characterized by papillary atrophy and impaired urinary concentrating ability, but the mechanisms involved are still unclear. DNA microarray was used to identify genes that are differentially expressed in renal medulla in response to neonatal treatment with AT(1) receptor antagonist losartan (30 mg/kg per d), which commenced within 24 h after birth. The data showed that losartan treatment for 48 h downregulated 68 genes, approximately 30% of which encode various components of cytoskeleton and cytoskeleton-associated proteins, extracellular matrix, and enzymes involved in extracellular matrix maturation or turnover. With the use of immunohistochemistry and Western immunoblot, the microarray data were confirmed and it was demonstrated that losartan suppressed renal expression of syndecan 2, alpha-smooth muscle actin, MHC class II, and leukocyte type 12-lipoxygenase by day 4. In addition, losartan inhibited medullary expression of integrin alpha6 and caused relocalization of integrins alpha6 and alpha3. Moreover, losartan inhibited cell proliferation in medullary tubules by day 9, as detected by Ki-67 immunostaining. This study provides new data supporting the contention that a lack of AT(1) receptor stimulation results in abnormal matrix assembly, disturbed cell-cell and cell-matrix interactions, and subsequent abnormal tubular maturation. Moreover, regulation of the expression of leukocyte type 12-lipoxygenase and alpha-smooth muscle actin by the renin-angiotensin system in the immature kidney adds new knowledge toward the understanding of renal vascular development.
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| 4. |
- Liu, C, et al.
(författare)
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The study of dissolution kinetics of K2SO4 crystal in aqueous ethanol solutions with a statistical rate theory
- 2004
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Ingår i: Chinese Journal of Chemical Engineering. - 1004-9541 .- 2210-321X. ; 12, s. 128-130
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Tidskriftsartikel (refereegranskat)abstract
- Dissolution kinetics of K2SO4 crystal in aqueous ethanol solutions was studied on-line with ion selective electrode. The concentration of K2SO4 was calculated from the determined electromotive force in which the activity coefficient of components in the liquid phase was calculated with the Pitzer equation. Dissolution kinetic parameters in the modified statistical rate theory were regressed. The correlation results show that dissolution rate of K2SO4 is slower in aqueous ethanol solutions than that in aqueous solutions. The two most important reasons are as follows: (1) The solubility Of K2SO4 in aqueous ethanol solutions is lower than that in aqueous solutions, which causes a decrease of the driving force of mass transfer. (2) The process of surface reaction Of K2SO4 became slower due to the addition of ethanol, so that the whole process is mainly dominated by the surface reaction instead of mass transfer.
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| 7. |
- Zhang, X. F., et al.
(författare)
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catena-Poly (1,10-phenanthroline-kappa N-2,N ')manganese(II) -mu-L-tartrato-kappa O-4(1),O-2 : O-3,O-4 hexahydrate
- 2003
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Ingår i: Acta Crystallographica Section C. - 0108-2701 .- 1600-5759. ; 59, s. M402-M404
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Tidskriftsartikel (refereegranskat)abstract
- The title compound, {[Mn(C4H4O6)(C12H8N2)] . 6H(2)O}(n), has a linear chain structure containing monomeric [ Mn( C4H4O6)( C12H8N2)] repeat units. Each manganese ion is six-coordinate, with the two phenanthroline N atoms [Mn - N = 2.229 (2) and 2.235 (2) Angstrom] and four O atoms from two tartrate anions [Mn - O-COO = 2.1252 (19) and 2.1310 (19) Angstrom, and Mn - O-OH = 2.2404 (19) and 2.2424 (19) Angstrom] forming a seriously distorted octahedral coordination environment. Six water molecules exist outside every repeat unit as solvate molecules. Extensive hydrogen-bonding interactions and pi-pi stacking of the phenanthroline moieties exist between the chains.
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