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Träfflista för sökning "WFRF:(Engstrand J) srt2:(2005-2009)"

Sökning: WFRF:(Engstrand J) > (2005-2009)

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  • Gorski, Dmitri, et al. (författare)
  • Review: Reduction of energy consumption in refining through mechanical pretreatment of wood chips
  • 2009
  • Ingår i: Proceedings - 2009 International Mechanical Pulping Conference, IMPC 2009. ; , s. 17-21
  • Konferensbidrag (refereegranskat)abstract
    • Refining of thermomechanical pulp is a very energy-intensive process. Mechanical pretreatment of wood chips prior to refining has been shown to be able to reduce total energy consumption, facilitate removal of extractives, have positive synergies with chemical and enzymatic pretreatments and improve impregnation of chips. There are a number of publications dealing with different aspects of mechanical pretreatment but few of them give a comprehensive overview of what has been done in this field. No review articles dealing with the subject have been written. Today, mechanical pretreatment is becoming more and more common and therefore a review is important.    
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  • Löfdahl, Magnus J, et al. (författare)
  • Increased intraepithelial T-cells in stable COPD
  • 2008
  • Ingår i: Respiratory Medicine. - : Elsevier BV. - 0954-6111 .- 1532-3064. ; 102:12, s. 1812-1818
  • Tidskriftsartikel (refereegranskat)abstract
    • BACKGROUND: The airway epithelium is the first line of defence in the response to inhaled particles and irritants. Chronic obstructive pulmonary disease (COPD) is an inflammatory disease characterised by an irreversible loss of lung function, with cigarette smoking as a major risk factor. Here, we address intraepithelial T-cells in COPD, as these cells are a distinct T-cell subtype thought to have important regulatory functions. We hypothesised that intraepithelial T-cells play a role in the response to lung irritants and that the T-cell populations would be altered and associated with signs of inflammation in COPD. METHODS: Bronchoscopy with endobronchial mucosal biopsy sampling was performed in 22 patients (mean age; 57) with stable COPD (median FEV(1)% predicted: 51). Age- and smoking- matched smokers (S) with normal lung function (n=14) and age-matched non-smokers (NS) (n=15) served as controls. Airway inflammation was recorded visually using bronchitis index (BI). Biopsy specimens were processed into glycol methacrylate resin and inflammatory cells were stained immunohistochemically. RESULTS: The number of intraepithelial CD4+ T-cells were significantly higher in COPD patients compared to smokers as well as trend towards significance in non-smokers (p=0.005 and p=0.036, respectively), whereas intraepithelial CD8+ T-cells number were increased in patients with COPD compared to non-smokers (p=0.017). Both patients with COPD and smokers had a higher BI than non-smokers (p<0.001 for both). CONCLUSIONS: The present data suggest a role for intraepithelial CD4+ and CD8+ T-cells in stable COPD and indicate that T-cells are of importance in the long-term inflammatory response in COPD or, alternatively, play a regulatory role.
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