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Träfflista för sökning "WFRF:(Graham Colin) srt2:(2005-2009)"

Sökning: WFRF:(Graham Colin) > (2005-2009)

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1.
  • Claesson, Lillemor, et al. (författare)
  • The timescale and mechanisms of fault sealing and water-rock interaction after an earthquake
  • 2007
  • Ingår i: Geofluids. ; 7, s. 427-440
  • Tidskriftsartikel (populärvet., debatt m.m.)abstract
    • Hydrogeochemical monitoring of a basalt-hosted aquifer, which contains Ice Age meteoric water and is situatedat 1220 m below sea level in the Tjornes Fracture Zone, northern Iceland, has been ongoing since July 2002.Based on hydrogeochemical changes following an earthquake of magnitude (Mw) 5.8 on 16 September 2002, weconstrained the timescales of post-seismic fault sealing and water–rock interaction. We interpret that the earthquakeruptured a hydrological barrier, permitting a rapid influx of chemically and isotopically distinct Ice Agemeteoric water from a second aquifer. During the two subsequent years, we monitored a chemical and isotopicrecovery towards pre-earthquake aquifer compositions, which we interpret to have been mainly facilitated byfault-sealing processes. This recovery was interrupted in November 2004 by a second rupturing event, which wasprobably induced by two minor earthquakes and which reopened the pathway to the second aquifer. We concludethat the timescale of fault sealing was approximately 2 years and that the approach to isotopic equilibrium(from global meteoric water line) was approximately 18% after >10^4 years. Key words: earthquake, fault sealing, hydrogeochemistry, Iceland, Tjornes Fracture Zone, water–rock interaction
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2.
  • Zeggini, Eleftheria, et al. (författare)
  • Meta-analysis of genome-wide association data and large-scale replication identifies additional susceptibility loci for type 2 diabetes
  • 2008
  • Ingår i: Nature Genetics. - : Springer Science and Business Media LLC. - 1546-1718 .- 1061-4036. ; 40:5, s. 638-645
  • Tidskriftsartikel (refereegranskat)abstract
    • Genome-wide association (GWA) studies have identified multiple loci at which common variants modestly but reproducibly influence risk of type 2 diabetes (T2D)(1-11). Established associations to common and rare variants explain only a small proportion of the heritability of T2D. As previously published analyses had limited power to identify variants with modest effects, we carried out meta-analysis of three T2D GWA scans comprising 10,128 individuals of European descent and similar to 2.2 million SNPs (directly genotyped and imputed), followed by replication testing in an independent sample with an effective sample size of up to 53,975. We detected at least six previously unknown loci with robust evidence for association, including the JAZF1 (P=5.0 x 10(-14)), CDC123-CAMK1D (P=1.2 x 10(-10)), TSPAN8-LGR5 (P=1.1 x 10(-9)), THADA (P=1.1 x 10(-9)), ADAMTS9 (P=1.2 x 10(-8)) and NOTCH2 (P=4.1 x 10(-8)) gene regions. Our results illustrate the value of large discovery and follow-up samples for gaining further insights into the inherited basis of T2D.
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