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- Hedenström, Anders, et al.
(författare)
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Radar Observations of Arctic Bird Migration in the Beringia Region
- 2009
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Ingår i: Arctic. - 0004-0843. ; 62:1, s. 25-37
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Tidskriftsartikel (refereegranskat)abstract
- Bird migration was recorded by tracking radar and visual observations in the Beringia region. The data were subdivided into seven areas extending from north of Wrangel Island southeastward toward the Bering Strait and then northwestward off the coast of Alaska to Point Barrow. The studies, which took place during a ship-based expedition between 30 July and 19 August 2005, recorded a total of 557 tracks (average duration 120 seconds) of bird flocks or individuals on post-breeding migration. The dominant eastward-flying flocks were likely composed of shorebirds on their way from breeding areas in central or eastern Siberia to intermediate stopovers and final destinations in North and South America. The courses were more southerly into the Bering Strait, possibly because of topographical influence. At two areas, the Chukchi Sea and Koluchinskaya Bay, there was also a westward component of migrants. At the Chukchi Sea these were almost certainly passerine birds migrating from Alaska to wintering areas in Asia and Africa, while at Koluchinskaya Bay, king ciders on molt migration could represent an important part of the westward component. The overall mean altitude of flights was 1157 m, and flight altitude was positively correlated with latitude. The mean ground speed was 15.9 m/s and the mean airspeed was 14.1 m/s, indicating that on average the birds were experiencing a small tail wind component. The airspeed was a function of the tail wind component and the vertical speed; altitude and the side wind component did not contribute significantly to variation in airspeed in this data set. Comparing these results with similar data obtained from Siberia and Canada, we concluded that Beringia is a migration hotspot where intense bird migration crosses between Asia and Alaska in both directions.
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| 2. |
- Henningsson, G, et al.
(författare)
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Universal parameters for reporting speech outcomes in individuals with cleft palate
- 2008
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Ingår i: The Cleft palate-craniofacial journal : official publication of the American Cleft Palate-Craniofacial Association. - : SAGE Publications. - 1055-6656. ; 45:1, s. 1-17
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Tidskriftsartikel (refereegranskat)abstract
- To achieve consistency and uniformity in reporting speech outcomes in individuals born with cleft palate with or without cleft lip using perceptual parameters that characterize their speech production behavior regardless of the language or languages spoken. Design: A working group of six individuals experienced in speech and cleft palate was formed to develop a system of universal parameters for reporting speech outcomes in individuals born with cleft palate. The system was adopted in conjunction with a workshop held in Washington, D.C., that was devoted to developing the universal system. The system, which was refined further following the workshop, involves a three-stage plan consisting of (1) evaluation, (2) mapping, and (3) reporting. The current report focuses primarily on the third stage, reporting speech outcomes. Results: A set of five universal speech parameters has been devised for the reporting stage. These consist of (1) hypernasality, (2) hyponasality, (3) audible nasal air emission and/or nasal turbulence, (4) consonant production errors, and (5) voice disorder. Also included are speech understandability and speech acceptability, global parameters that can be reported for any type of speech disorder. The parameters are described in detail, and guidelines for speech-sampling content and scoring procedures in relation to the parameters are presented. Conclusion: A plan has been developed to document speech outcomes in individuals with cleft palate, regardless of the spoken language, using a set of five universal reporting parameters and two global speech parameters.
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| 3. |
- Henningsson, G, et al.
(författare)
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Untitled
- 2008
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Ingår i: CLEFT PALATE CRANIOFACIAL JOURNAL. - : SAGE Publications. - 1055-6656 .- 1545-1569. ; 45:4, s. 454-455
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Tidskriftsartikel (övrigt vetenskapligt/konstnärligt)
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| 6. |
- Lohmander, Anette, 1956, et al.
(författare)
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Methodology for speech assessment in the scandcleft project-an international randomized clinical trial on palatal surgery: experiences from a pilot study.
- 2009
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Ingår i: The Cleft palate-craniofacial journal : official publication of the American Cleft Palate-Craniofacial Association. - : SAGE Publications. - 1055-6656. ; 46:4, s. 347-62
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Tidskriftsartikel (refereegranskat)abstract
- Objective: To present the methodology for speech assessment in the Scandcleft project and discuss issues from a pilot study. Design: Description of methodology and blinded test for speech assessment. Speech samples and instructions for data collection and analysis for comparisons of speech outcomes across five included languages were developed and tested. Participants and Materials: Randomly selected video recordings of 10 5-year-old children from each language (n = 50) were included in the project. Speech material consisted of test consonants in single words, connected speech, and syllable chains with nasal consonants. Five experienced speech and language pathologists participated as observers. Main Outcome Measures: Narrow phonetic transcription of test consonants translated into cleft speech characteristics, ordinal scale rating of resonance, and perceived velopharyngeal closure (VPC). A velopharyngeal composite score (VPC-sum) was extrapolated from raw data. Intra-agreement comparisons were performed. Results: Range for intra-agreement for consonant analysis was 53% to 89%, for hypernasality on high vowels in single words the range was 20% to 80%, and the agreement between the VPC-sum and the overall rating of VPC was 78%. Conclusions: Pooling data of speakers of different languages in the same trial and comparing speech outcome across trials seems possible if the assessment of speech concerns consonants and is confined to speech units that are phonetically similar across languages. Agreed conventions and rules are important. A composite variable for perceptual assessment of velopharyngeal function during speech seems usable; whereas, the method for hypernasality evaluation requires further testing.
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| 9. |
- Mosén, Henrik, et al.
(författare)
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Defective glucose-stimulated insulin release in the diabetic Goto-Kakizaki (GK) rat coincides with reduced activity of the islet carbon monoxide signaling pathway
- 2005
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Ingår i: Endocrinology. - : The Endocrine Society. - 0013-7227 .- 1945-7170. ; 146:3, s. 1553-1558
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Tidskriftsartikel (refereegranskat)abstract
- The Goto-Kakizaki (GK) rat displays a markedly reduced insulin response to glucose, a defect that is thought to be coupled to an impaired glucose signaling in the beta-cell. We have examined whether carbon monoxide (CO), derived from beta-cell heme oxygenase (HO), might be involved in the secretory dysfunction. Immunocytochemical labeling of constitutive HO (HO-2) showed no overt difference in fluorescence pattern in islets from GK vs. Wistar controls. However, isolated islets from GK rats displayed a markedly impaired HO activity measured as CO production (-50%), and immunoblotting revealed an approximately 50% reduction of HO-2 protein expression compared with Wistar controls. Furthermore, there was a prominent expression of inducible HO (HO-1) in GK islets. Incubation of isolated islets showed that the glucose-stimulated CO production and the glucose-stimulated insulin response were considerably reduced in GK islets compared with Wistar islets. Addition of the HO activator hemin or gaseous CO to the incubation media brought about a similar amplification of glucose-stimulated insulin release in GK and Wistar islets, suggesting that distal steps in the HO-CO signaling pathway were not appreciably affected. We conclude that the defective insulin response to glucose in the GK rat can be explained, at least in part, by a marked impairment of the glucose-HO-CO signaling pathway as manifested by a prominent decrease in glucose stimulation of islet CO production and a reduced expression of HO-2. A possible role of HO-1 expression as a compensatory mechanism in the GK islets is presently unclear.
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