SwePub
Sök i SwePub databas

  Utökad sökning

Träfflista för sökning "WFRF:(Hirota A.) srt2:(2000-2004)"

Sökning: WFRF:(Hirota A.) > (2000-2004)

  • Resultat 1-4 av 4
Sortera/gruppera träfflistan
   
NumreringReferensOmslagsbildHitta
1.
  • Muto, S., et al. (författare)
  • TEM and EELS characterization of carbon dust and co-deposited layers from the TEXTOR tokamak
  • 2002
  • Ingår i: Journal of Nuclear Materials. - 0022-3115 .- 1873-4820. ; 307, s. 1289-1293
  • Tidskriftsartikel (refereegranskat)abstract
    • Co-deposited layers and dust agglomerates formed at TEXTOR tokamak were collected and examined in detail by means of transmission electron microscopy and electron energy-loss spectroscopy. The analysis has shown that thick deposits and dust contain mainly carbon with an admixture of boron precipitates and small amounts of other elements. The carbon deposit was mostly amorphous, while strips were graphite crystallites embedded in dust agglomerates. In thin co-deposits on the collector probe very small graphite crystallites were piled up with a preferential orientation. The results are discussed in terms of processes decisive for the erosion of plasma facing components and for the fuel retention.
  •  
2.
  •  
3.
  •  
4.
  • Weber, M., et al. (författare)
  • The physiological basis of conduction slowing in ALS patients homozygous for the autosomal recessive D90A CuZn-SOD mutation
  • 2001
  • Ingår i: Muscle and Nerve. - 0148-639X .- 1097-4598. ; 24:1, s. 89-97
  • Tidskriftsartikel (refereegranskat)abstract
    • Familial amyotrophic lateral sclerosis (ALS) with the autosomal-recessively inherited D90A CuZn-superoxide dismutase (CuZn-SOD) mutation is characterized by a stereotypic slowly progressive, distinctive phenotype and very slow central motor conduction. To determine the basis of this slowing, we assessed corticomotoneuronal function using peristimulus time histograms (PSTHs) in 8 ALS patients homozygous for the D90A CuZn-SOD mutation. The results were compared with findings in 10 patients with multiple sclerosis (MS), in which slowing of central motor conduction is common, and 11 healthy subjects. PSTHs were constructed from 3-7 different, voluntarily recruited motor units recorded in each patient from the extensor digitorum communis muscle (EDC). In D90A and MS patients, the stimulus threshold, onset latency, number of excess bins, duration, amplitude, and synchrony of the primary peak differed significantly from controls (P < 0.0004). The mean onset latency of the primary peak in D90A patients was 35.3 ms, compared to 23.6 ms for MS patients and 19.3 ms for normal subjects (P < 0.0001). In the D90A patients, the onset latencies of the primary peak had a bimodal distribution, whereas in MS the distribution showed a continuum. Loss of synchrony was similar in D90A and MS patients, but the threshold, number of excess bins, and duration differed significantly (P < 0.0057), which suggests that either axonal loss or demyelination can result in delayed and desynchronized primary peaks. We propose that conduction slowing in the D90A homozygotes results from selective loss of fast-conducting large pyramidal cells with preservation of slow-conducting mono- or polysynaptic corticomotoneuronal connections. Copyright 2001 John Wiley & Sons, Inc.
  •  
Skapa referenser, mejla, bekava och länka
  • Resultat 1-4 av 4

Kungliga biblioteket hanterar dina personuppgifter i enlighet med EU:s dataskyddsförordning (2018), GDPR. Läs mer om hur det funkar här.
Så här hanterar KB dina uppgifter vid användning av denna tjänst.

 
pil uppåt Stäng

Kopiera och spara länken för att återkomma till aktuell vy