| 1. |
- BENBADIS, L, et al.
(författare)
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WORKING GROUP-VII - FOOD-PRODUCTS
- 1995
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Ingår i: MICROBIAL ECOLOGY IN HEALTH AND DISEASE. - 0891-060X. ; 8, s. S43-S44
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Tidskriftsartikel (övrigt vetenskapligt/konstnärligt)
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| 2. |
- Brentel, Jonas, et al.
(författare)
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Single-wavelength 40 Gb/s soliton field transmission experiments over 400 km fibre without in-line control
- 1999
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Ingår i: OFC/IOOC 1999 - Optical Fiber Communication Conference and the International Conference on Integrated Optics and Optical Fiber Communication. - 155752582X ; 3, s. 121-124
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Konferensbidrag (refereegranskat)abstract
- We report single-wavelength 40-Gbit/s soliton transmission over 400-km installed fiber lines in Sweden. Both polarization-division multiplexed and parallel solitons were transmitted. The system performance is dominated by the polarization-mode dispersion.
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| 3. |
- Grundemar, L, et al.
(författare)
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Characterization of vascular neuropeptide Y receptors
- 1992
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Ingår i: British Journal of Pharmacology. - : Wiley. - 0007-1188. ; 105:1, s. 45-50
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Tidskriftsartikel (refereegranskat)abstract
- 1. In the present study we compared neuropeptide Y (NPY) and NPY-related analogues for their ability to activate or bind to vascular NPY receptors in four experimental set-ups. Previous results have suggested the existence of different receptor subtypes, Y1 receptors requiring full-length NPY (1-36) or [Pro34]-NPY, and Y2 receptors recognizing also N-terminally truncated forms of NPY but not [Pro34]-NPY. 2. NPY 1-36 and [Pro34]-NPY dose-dependently increased arterial pressure in the anaesthetized rat with a similar magnitude and potency. NPY 2-36 was much less potent than NPY 1-36. NPY 4-36 and NPY 11-36 were inactive even at a dose as high as 10 nmol kg-1. 3. NPY 1-36, [Pro34]-NPY, NPY 2-36 and NPY 5-36 concentration-dependently increased the coronary resistance in the Langendorff preparation of the rat. NPY 1-36 and [Pro34]-NPY were equipotent, while NPY 2-36 and NPY 5-36 were about 7 and 20 times less potent. At 0.3 microM, NPY 11-36, NPY 20-36 and NPY 22-36 induced a slight contraction while NPY 23-36 was inactive. 4. NPY 1-36, [Pro34]-NPY, NPY 2-36, NPY 4-36, NPY 5-36 and NPY 11-36 evoked concentration-dependent contractions in the isolated inferior caval vein of the rat and guinea-pig. [Pro34]-NPY was more potent than NPY 1-36. NPY 2-36 was equipotent with NPY 1-36, while NPY 4-36, NPY 5-36 and NPY 11-36 were approximately 30 times less potent.5. [Pro34]-NPY was equipotent with NPY 1-36 in displacing the '25I-labelled gut hormone peptide([1251]-PYY) from rat aortic smooth muscle cells, while NPY 2-36 and shorter forms of NPY were much less potent or inactive.6. In caval vein smooth muscle cells of the rat, the displacement pattern was more complex than in aortic smooth muscle cells, in that both [Pro34]-NPY and NPY 13-36 effectively displaced the radioligand,albeit none of them completely.7. In conclusion, the NPY-evoked pressor response in the whole rat and coronary vessels seems to be mediated by vascular Y1 receptors and the binding characteristics of the NPY-related peptides in the aortic smooth muscle cells correspond to a population of such receptors. In the caval vein, the profile of the bioactivity and the binding affinity of the NPY-related peptides suggest a mixed population of Y1/Y2 receptors.
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| 4. |
- Kolltveit, Erling, et al.
(författare)
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Single-wavelength 40 Gbit/s soliton field transmission experiment over 400 km of installed fibre
- 1999
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Ingår i: Electronics Letters. - : Institution of Engineering and Technology (IET). - 1350-911X .- 0013-5194. ; 35:1, s. 75-76
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Tidskriftsartikel (refereegranskat)abstract
- 40Gbit/s soliton transmission over 400km (57km amplifier spacing) of installed lines without in-line control has been achieved. The maximum transmission penalties for parallel polarised and polarisation division multiplexed soliton transmission were ∼4 and 1dB, respectively. The transmission distance is the longest ever reported for a field trial with a single wavelength bit rate > 20Gbit/s for any optical transmission format. The polarisation mode dispersion was numerically found to completely dominate the system performance.
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| 5. |
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| 6. |
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| 7. |
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| 8. |
- Akselsson, Roland, et al.
(författare)
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Computer Aided Planning of Production and Working Environment
- 1994
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Ingår i: Human Factors in Organizational Design and Management -IV. - Amsterdam : Elsevier. ; , s. 499-504, s. 499-504
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Konferensbidrag (refereegranskat)abstract
- Success of design projects depends on their quality from a technical and economic point of view and on the quality of their implementation and use. This paper discusses a computer aided planning (CAP) technique and its applicability and quality in both these respects. One version of the CAP-technique is developed for planning of industrial working environments and another version for planning of residential and working environments for physically handicapped.
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| 9. |
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| 10. |
- Bergquist, Jonas, et al.
(författare)
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Measurements of catecholamine-mediated apoptosis of immunocompetent cells by capillary electrophoresis.
- 1997
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Ingår i: Electrophoresis. - : Wiley. - 0173-0835 .- 1522-2683. ; 18:10, s. 1760-6
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Tidskriftsartikel (refereegranskat)abstract
- Single cell analysis with capillary electrophoresis, a technique capable of detecting zeptomole quantities (10(-21) mole) of neurochemical species, has been used to demonstrate that lymphocytes are capable of active synthesis of dopamine and norepinephrine. Exposure of lymphocytes to catecholamines at concentrations as low as 10 nM leads to decreased proliferation and differentiation, e.g. interferon-gamma (IFN-gamma), interleukin-4 (IL-4) and immunoglobulin (Ig). In addition, both inhibition of dopamine uptake with nomifensine and inhibition of packing of catecholamines into vesicles with tetrabenazine, results in significantly lower levels of dopamine and norepinephrine (p < 0.01 and p < 0.05, respectively). The catecholamine-dependent inhibition of T- and B-lymphocyte activity is mediated via an induction of a Bcl-2/Bax and Fas/FasL involved apoptosis. These findings indicate a novel mechanism for regulation of lymphocyte activity in the central nervous system, whereby elevated regional levels of catecholamines might lead to the immunoprivilege of the brain.
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