| 1. |
- Blom, Peter, et al.
(författare)
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Nordiska museets konservering
- 1987
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Ingår i: Kulturminnesvård. - Stockholm : Riksantikvarieämbetet. - 0346-9077. ; :4-5, s. 24-28
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Tidskriftsartikel (populärvet., debatt m.m.)abstract
- Nordiska museet med sina stora och mångskiftande samlingar har en Teknisk avdelning som ansvarar för vård och konservering. Avdelningens chef Bengt Nyström berättar tillsammans med sina medarbetare Peter Blom och Marie-Louise Wulfcronaom verksamheten.
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| 2. |
- Edgren, Lars, et al.
(författare)
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Från skråhantverk till näringsfrihet
- 1989
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Ingår i: Hantverk i Sverige : Om bagare, kopparslagare,vagnmakare och 286 andra hantverksyrken - Om bagare, kopparslagare,vagnmakare och 286 andra hantverksyrken. - 9136026379 ; , s. 13-21
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Bokkapitel (populärvet., debatt m.m.)
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| 3. |
- Edgren, Lars, et al.
(författare)
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Lärling, gesäll, mästare
- 1989
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Ingår i: Hantverk i Sverige : Om bagare, kopparslagare, vagnmakare och 286 andra hantverksyrken - Om bagare, kopparslagare, vagnmakare och 286 andra hantverksyrken. - 9136026379 ; , s. 30-36
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Bokkapitel (populärvet., debatt m.m.)
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| 4. |
- Helmius, G, et al.
(författare)
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Myocardial infarction without coronary occlusion. A morphologic study in sheep.
- 1982
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Ingår i: Upsala Journal of Medical Sciences. - 0300-9734 .- 2000-1967. ; 87:1, s. 67-80
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Tidskriftsartikel (refereegranskat)abstract
- The macroscopic, histologic and enzyme-histochemical characteristics of the myocardial lesion obtained after heating of a thermoprobe in a branch of the left coronary artery in sheep is reported. In 13 sheep such myocardial lesions were produced distal to the location of the thermoprobe. Alterations were observed in accordance with generally accepted morphologic criteria for myocardial infarction. The coronary artery branch in which the thermoprobe was located showed erythrocyte and platelet aggregates immediately after the heating episode, which disappeared within a few min, as demonstrated by coronary arteriography. Injection of radiolabelled microspheres into the coronary circulation after induction of the myocardial lesion, cryosectioning of the heart and autoradiography revealed a lack of blood flow in the damaged myocardial region. We consider this new method a suitable tool for further studies on the complex pathology involved in the development of myocardial infarction.
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| 5. |
- Holtz, A, et al.
(författare)
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Blocking weight-induced spinal cord injury in rats : effects of TRH or naloxone on motor function recovery and spinal cord blood flow.
- 1989
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Ingår i: Acta Neurologica Scandinavica. - 0001-6314 .- 1600-0404. ; 80:3, s. 215-20
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Tidskriftsartikel (refereegranskat)abstract
- The ability of thyrotropin releasing hormone (TRH) or naloxone to reduce the motor function deficit and to improve the spinal cord blood flow (SCBF) was investigated in a rat spinal cord compression injury model. Spinal cord injury was induced by compression for 5 min with a load of 35 g on a 2.2 x 5.0 mm sized compression plate causing a transient paraparesis. One group of animals was given TRH, one group naloxone and one group saline alone. Each drug was administered intravenously as a bolus dose of 2 mg/kg 60 min after injury followed by a continuous infusion of 2 mg/kg/h for 4 h. The motor performance was assessed daily on the inclined plane until Day 4, when SCBF was measured with the 14C-iodoantipyrine autoradiographic method. It was found that neither TRH nor naloxone had promoted motor function recovery or affected SCBF 4 days after spinal cord injury.
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| 6. |
- Holtz, A, et al.
(författare)
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Regulation of spinal cord blood flow in the rat as measured by quantitative autoradiography.
- 1988
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Ingår i: Acta Physiologica Scandinavica. - : Wiley. - 0001-6772 .- 1365-201X. ; 133:4, s. 485-93
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Tidskriftsartikel (refereegranskat)abstract
- Spinal cord blood flow (SCBF) and its response to alterations in blood gases and to systemic hypotension, haemodilution and barbiturate anaesthesia were measured in 47 rats at the level of Th 7-8 by quantitative autoradiography with [14C]iodoantipyrine ([14C]IAP) as tracer. During normocapnia and normoxia the mean SCBF values in the grey and white matter were 94 +/- 21 and 17 +/- 3 ml min-1 100 g-1, respectively. SCBF was highly dependent on PaCO2. Thus in hypercapnic animals (PaCO2 greater than 9 kPa) SCBF was increased in grey and white matter to 228 +/- 22 and 54 +/- 7 ml min-1 100 g-1, while during hypocapnia (PaCO2 less than 3.9 kPa) it was decreased to 64 +/- 14 and 11 +/- 2 ml min-1 100 g-1, respectively. Mean arterial blood pressure (MABP) was reduced by withdrawal of blood to 80 +/- 8 mmHg in a light hypotension group and to 53 +/- 11 mmHg in a severe hypotension group, compared with 126 +/- 13 mmHg in a control group. There was no significant difference in SCBF between the control group and the hypotension groups, suggesting that autoregulation is maintained down to an MABP of at least 50 mmHg. Normovolaemic haemodilution, with a reduction of the haematocrit from 50 +/- 2 to 33 +/- 3, increased SCBF to 113 +/- 9 ml min-1 100 g-1 in grey matter and to 21 +/- 12 ml min-1 100 g-1 in white matter.(ABSTRACT TRUNCATED AT 250 WORDS)
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| 7. |
- Holtz, A, et al.
(författare)
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Spinal cord blood flow measured by 14C-iodoantipyrine autoradiography during and after graded spinal cord compression in rats.
- 1989
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Ingår i: Surgical Neurology. - 0090-3019 .- 1879-3339. ; 31:5, s. 350-60
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Tidskriftsartikel (refereegranskat)abstract
- The relations between degree of thoracic spinal cord compression causing myelographic block, reversible paraparesis, and extinction of the sensory evoked potential on one hand, and spinal cord blood flow on the other, were investigated. This was done in rats using the blocking weight-technique and 14C-iodoantipyrine autoradiography. A load of 9 g caused myelographic block. Five minutes of compression with that load caused a reduction of spinal cord blood flow to about 25%, but 5 and 60 minutes after the compression spinal cord blood flow was restored to 60% of the pretrauma value. A load of 35 g for 5 minutes caused transient paraparesis. Recovery to about 30% was observed 5 and 60 minutes thereafter. During compression at a load of 55 g, which caused almost total extinction of sensory evoked potential and irreversible paraplegia, spinal cord blood flow under the load ceased. The results indicate that myelographic block occurs at a load which does not cause irreversible paraparesis and that a load which permits sensory evoked potential to be elicited results in potentially salvageable damage.
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| 8. |
- Holtz, A, et al.
(författare)
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Spinal cord injury in rats : inability of nimodipine or anti-neutrophil serum to improve spinal cord blood flow or neurologic status.
- 1989
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Ingår i: Acta Neurologica Scandinavica. - 0001-6314 .- 1600-0404. ; 79:6, s. 460-7
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Tidskriftsartikel (refereegranskat)abstract
- The role of a calcium-mediated increase in vascular resistance and of vascular damage caused by polymorphonuclear leukocytes (PMNLs) in the development of neurologic deficit and disturbance of spinal cord circulation following spinal cord compression was studied in the rat. Spinal cord injury was induced by 5 min of compression with a load of 35 g on a 2.2 X 5.0 mm compression plate. This caused transient paraparesis. The rats received either the calcium receptor antagonist nimodipine or an anti-rat neutrophil serum (ANS). Nimodipine was infused i.v. for 4 h in an amount of 1.5 micrograms/kg/min starting 60 min after trauma. The number of circulating PMNLs was depleted by intraperitoneal injection of an ANS raised in sheep given 12 h before trauma. This caused a reduction to about 2% of the pre-ANS value. Controls received saline or normal sheep serum. The motor performance was assessed daily on the inclined plane. On day one, the day after injury, the capacity angle had decreased from about 63 degrees preoperatively to close to 32 degrees in the experimental groups. There was then a slow improvement in both the control and experimental groups and on day 4 the capacity angle was close to 43 degrees in all 3 groups. Spinal cord blood flow, as measured with the 14C-iodoantipyrine autoradiography method, was similar in all groups on day 4. As neither the neurologic dysfunction nor the spinal cord blood flow was affected by post-trauma treatment with nimodipine or pretreatment with ANS, the possibility that calcium-mediated vasoconstriction or PMNLs play a role in the development of posttraumatic neurologic disability was not supported by this study.
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| 9. |
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| 10. |
- Nyström, Bengt, 1937-
(författare)
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Centralmuseerna och kulturmiljövården
- 1988
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Ingår i: Kulturmiljövård. - Stockholm : Riksantikvarieämbetet. - 1100-4800. ; [13]:2, s. 10-12
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Tidskriftsartikel (populärvet., debatt m.m.)
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