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- Uhlemann, Elisabeth, et al.
(författare)
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Deadline dependent coding-a framework for wireless real-time communication
- 2000
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Ingår i: Seventh International Conference on Real-Time Computing Systems and Applications. - Piscataway, NJ : IEEE. - 0769509304 ; , s. 135-142, s. 135-142
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Konferensbidrag (refereegranskat)abstract
- A framework for real-time communication over a wireless channel is proposed. The concept of deadline dependent coding (DDC), previously suggested by the authors, is further developed using soft decision decoding of block codes to maximize the probability of delivering information before a given deadline. The strategy of DDC is to combine different coding and decoding methods with automatic repeat request (ARQ) in order to fulfil the application requirements. These requirements are formulated as two Quality of Service (QoS) parameters: deadline (t_DL) and probability of correct delivery before the deadline (P_d), leading to a probabilistic view of real-time communication. An application can negotiate these QoS parameters with the DDC protocol, thus creating a flexible and dependable scheme.
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| 4. |
- Andersson, Claes, 1973, et al.
(författare)
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Urban settlement transitions
- 2002
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Ingår i: Environment and Planning B: Planning and Design. ; 29, s. 841-865
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Tidskriftsartikel (refereegranskat)
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| 5. |
- Bache, Iben, et al.
(författare)
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An excess of chromosome 1 breakpoints in male infertility.
- 2004
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Ingår i: European Journal of Human Genetics. - : Springer Science and Business Media LLC. - 1476-5438 .- 1018-4813. ; 12:12, s. 993-1000
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Tidskriftsartikel (refereegranskat)abstract
- In a search for potential infertility loci, which might be revealed by clustering of chromosomal breakpoints, we compiled 464 infertile males with a balanced rearrangement from Mendelian Cytogenetics Network database (MCNdb) and compared their karyotypes with those of a Danish nation-wide cohort. We excluded Robertsonian translocations, rearrangements involving sex chromosomes and common variants. We identified 10 autosomal bands, five of which were on chromosome 1, with a large excess of breakpoints in the infertility group. Some of these could potentially harbour a male-specific infertility locus. However, a general excess of breakpoints almost everywhere on chromosome 1 was observed among the infertile males: 26.5 versus 14.5% in the cohort. This excess was observed both for translocation and inversion carriers, especially pericentric inversions, both for published and unpublished cases, and was significantly associated with azoospermia. The largest number of breakpoints was reported in 1q21; FISH mapping of four of these breakpoints revealed that they did not involve the same region at the molecular level. We suggest that chromosome 1 harbours a critical domain whose integrity is essential for male fertility.
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| 9. |
- Herlitz, Johan, et al.
(författare)
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Effects of metoprolol CR/XL on mortality and hospitalizations in patients with heart failure and history of hypertension.
- 2002
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Ingår i: Journal of Cardiac Failure. - : Churchill Livingstone. - 1071-9164 .- 1532-8414. ; 8:1, s. 8-14
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Tidskriftsartikel (refereegranskat)abstract
- BACKGROUND: We describe the effect of controlled-release/extended-release (CR/XL) metoprolol succinate once daily on mortality and hospitalizations among patients with a history of hypertension complicated by chronic systolic heart failure. METHODS AND RESULTS: We enrolled 3,991 patients with chronic heart failure of New York Heart Association functional class II-IV with an ejection fraction of < or = 0.40, stabilized with optimum standard therapy, in a double-blind randomized placebo-controlled study. A total of 1,747 patients (44%) had a history of hypertension; 871 were randomized to receive metoprolol CR/XL and 876 to receive placebo. Treatment with metoprolol CR/XL compared with placebo resulted in a significant reduction in total mortality (relative risk [RR], 0.61; 95% confidence interval [CI], 0.44-0.84; P =.0022), mainly because of reductions in sudden death (RR, 0.51; 95% CI, 0.33-0.79; P =.0022) and mortality from worsening heart failure (RR, 0.49; 95% CI, 0.25-0.99; P =.042). Total number of hospitalizations for worsening heart failure was reduced by 30% in the metoprolol CR/XL group compared with placebo (P =.015). Metoprolol CR/XL was well tolerated: 12% fewer patients withdrew from study medication (all-cause) compared with placebo (P =.048). CONCLUSIONS: A subgroup analysis of MERIT-HF shows that patients with heart failure and a history of hypertension received a similar benefit from metoprolol CR/XL treatment as all patients included in the total study.
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