| 1. |
- Adcox, K, et al.
(författare)
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PHENIX detector overview
- 2003
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Ingår i: Nuclear Instruments & Methods in Physics Research. Section A: Accelerators, Spectrometers, Detectors, and Associated Equipment. - 0167-5087. ; 499:2-3, s. 469-479
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Tidskriftsartikel (refereegranskat)abstract
- The PHENIX detector is designed to perform a broad study of A-A, p-A, and p-p collisions to investigate nuclear matter under extreme conditions. A wide variety of probes, sensitive to all timescales, are used to study systematic variations with species and energy as well as to measure the spin structure of the nucleon. Designing for the needs of the heavy-ion and polarized-proton programs has produced a detector with unparalleled capabilities. PHENIX measures electron and muon pairs, photons, and hadrons with excellent energy and momentum resolution. The detector consists of a large number of subsystems that are discussed in other papers in this volume. The overall design parameters of the detector are presented. (C) 2002 Elsevier Science B.V. All rights reserved.
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| 2. |
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| 3. |
- Yu, H.Y., et al.
(författare)
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Current enhancement with alternating gate voltage in the Coulomb blockade regime of a single wall carbon nanotube
- 2004
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Ingår i: Applied Physics A. - : Springer Science and Business Media LLC. - 0947-8396 .- 1432-0630. ; 79, s. 1613-1615
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Tidskriftsartikel (refereegranskat)abstract
- We investigated the current–voltage characteristics of a carbon nanotube in a single electron transistor structure with alternating gate voltage. A continuous current enhancement effect with increasing frequency of the applied gate voltage up to 13 MHz is reported. Assuming that I=nef, more than 1000 electrons are driven to flow across the source–drain channel at VDS=100 mV, 13 MHz of gate voltage (Vp-p=2 V) and T=1.8 K. The continuous current enhancement is explained by the broadening effect of the discrete energy levels of the finite-length carbon nanotube.
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| 4. |
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| 5. |
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| 6. |
- Sohler, D, et al.
(författare)
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Band-terminating states in Ag-101
- 2004
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Ingår i: Nuclear Physics, Section A. - : Elsevier BV. - 0375-9474 .- 1873-1554. ; 733:1-2, s. 37-52
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Tidskriftsartikel (refereegranskat)abstract
- Excited states of the neutron deficient Ag-101 nucleus have been investigated via the Cr-50(Ni-58, 3rho1alpha) heavy-ion induced reaction at 261 meV by use of in-beam spectroscopic methods. On the basis of the measured gammagamma-cincidence relations and angular distribution ratios high-spin bands have been extended up to I-pi = 35/2(+), 45/2((-)) and (49/2(-)). The negative parity states at the highest energy have been interpreted as terminating non-collective oblate states in the framework of the Nilsson-Strutinsky cranking formalism.
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| 7. |
- Sohler, D, et al.
(författare)
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Maximally aligned states in Ag-99
- 2003
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Ingår i: European Physical Journal A. Hadrons and Nuclei. - : Springer Science and Business Media LLC. - 1434-6001 .- 1434-601X. ; 16:2, s. 171-175
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Tidskriftsartikel (refereegranskat)abstract
- Excited states of Ag-99 were populated via the Cr-50 + Ni-58 (261 MeV) reaction using the NORDBALL detector array equipped with charged-particle and neutron. detector systems for reaction channel separation. On the basis of the measured gammagamma-coincidence relations and angular distribution ratios a significantly extended level scheme has been constructed up to E-x similar to 7.8 MeV and I = 35/2. The experimental results were described within the framework of the shell model. Candidates for states fully aligned in the pig(9/2)(-3)nu(d(5/2),g(7/2))(2) valence configuration space were found at 4109 and 6265 keV.
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| 8. |
- Spentchian, M, et al.
(författare)
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Severe hypophosphatasia: Characterization of fifteen novel mutations in the ALPL gene
- 2003
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Ingår i: Human Mutation. - : Hindawi Limited. - 1059-7794. ; 22:1, s. 105-106
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Tidskriftsartikel (refereegranskat)abstract
- Hypophosphatasia is an inherited disorder characterized by defective bone mineralization and deficiency of serum and tissue liver/bone/kidney alkaline phosphatase (L/B/K ALP) activity. We report the characterization of ALPL gene mutations in a series of 11 families from various origins affected by perinatal and infantile hypophosphatasia. Sixteen distinct mutations were found, fifteen of them not previously reported: M45V, G46R, 388-391delGTAA, 389delT, T131I, G145S, D172E, 662delG, G203A, R255L, 876-881delAGGGGA, 962delG, E294K, E435K, and A451T. This confirms that severe hypophosphatasia is due to a large spectrum of mutations in Caucasian populations.
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| 10. |
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