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Sökning: WFRF:(Söderberg Olof) > (1975-1999)

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1.
  • Erlandsson, Olof, et al. (författare)
  • Demonstrating the Performance and Emission Characteristics of a Variable Compression Ratio, Alvar-Cycle Engine
  • 1998
  • Ingår i: SAE Technical Paper Series. - 0148-7191.
  • Konferensbidrag (refereegranskat)abstract
    • This paper is a direct continuation of a previous study that addressed the performance and design of a variable compression engine, the Alvar-Cycle Engine [1]. The earlier study was presented at the SAE International Conference and Exposition in Detroit during February 23- 26, 1998 as SAE paper 981027. In the present paper test results from a single cylinder prototype are reviewed and compared with a similar conventional engine. Efficiency and emissions are shown as function of speed, load, and compression ratio. The influence of residual gas on knock characteristics is shown. The potential for high power density through heavy supercharging is analyzed.
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4.
  • Naredi, Peter, 1955, et al. (författare)
  • The influence of hepatic artery ligation and of vasopressin on liver tumour blood flow in rats.
  • 1992
  • Ingår i: Journal of surgical oncology. - : Wiley. - 0022-4790 .- 1096-9098. ; 50:2, s. 70-6
  • Tidskriftsartikel (refereegranskat)abstract
    • The blood flow in an experimental adenocarcinoma in the rat liver was determined with the 133Xe-washout technique before and after hepatic artery ligation (HAL). There was an initial reduction of the washout of 50%. This was further reduced after 1 day by 50%, which was maintained for 7 days. Seven days after HAL or sham procedures the 133Xe-washout was of similar magnitude in the liver tumours, although after the sham procedure the tumours were larger (3.4 g vs. 1.5 g). The estimated tumour blood flow was then approximately 0.04 ml x min-1 x g-1. The influence on normal liver parenchyma of HAL was a reduction at 30 minutes, which was maintained for 7 days. Postacton--a synthetic vasopressin--did not influence the 133Xe-washout in normal liver parenchyma in non-tumour, as well as in tumour-bearing animals. There was no influence of Postacton on the 133Xe-washout in the liver tumours. Thirty minutes after HAL Postacton gave a reduction of blood flow in normal liver parenchyma of tumour-bearing animals, which is thus only from the portal vein. In tumours Postacton did not significantly reduce the tumour blood flow immediately after HAL.
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