| 1. |
- Kivimaeki, A., et al.
(författare)
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On the production of N-2(+) ions at the N 1s edge of the nitrogen molecule
- 2013
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Ingår i: Physica Scripta. - : IOP Publishing. - 0031-8949 .- 1402-4896. ; 87:6
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Tidskriftsartikel (refereegranskat)abstract
- The N-2(+) ion yield of the N-2 molecule has been measured at the N 1s -> Rydberg excitations. It displays Fano-type line shapes due to interference between direct outer-valence photoionization and participator decay of the core-excited Rydberg states. The N-2(+) ion yield is compared with the total intensity of the outer-valence photoelectron lines obtained recently with electron spectroscopy (Kivimaki et al 2012 Phys. Rev. A 86 012516). The increasing difference between the two curves at the higher core-to-Rydberg excitations is most likely due to soft x-ray emission processes that are followed by autoionization. The results also suggest that resonant Auger decay from the core-valence doubly excited states contributes to the N-2(+) ion yield at the photon energies that are located on both sides of the N 1s ionization limit.
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| 2. |
- Kivimaeki, A., et al.
(författare)
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Valence photoionization of the N-2 molecule in the region of the N 1s -> Rydberg excitations
- 2012
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Ingår i: Physical Review A (Atomic, Molecular and Optical Physics). - 1050-2947. ; 86:1
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Tidskriftsartikel (refereegranskat)abstract
- The intensities of the X and A valence photoelectron lines of N-2 have been found to display Fano line shapes as a function of photon energy around the N 1s -> Rydberg excitations. The vibrational intensity distributions of these photoelectron lines change at the N 1s -> 3s sigma and 3p pi resonances. These effects indicate interference between direct and resonant photoionization channels. Our numerical simulations reproduce quite well the experimental results.
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| 3. |
- Urpelainen, S., et al.
(författare)
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FINEST:A high performance branch-line for VUV photon energy range gas phase studies at MAX-lab
- 2010
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Ingår i: AIP Conference Proceedings. - : AIP. - 0094-243X. - 9780735407824 ; 1234, s. 411-414
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Konferensbidrag (refereegranskat)abstract
- We present a dedicated beamline branch for high flux and ultra-high resolution (R>100000) gas and vapor phase studies in the vacuum-ultra-violet (VUV) region of light on the undulator beamline I3, located on the 700 MeV MAX-III storage ring. The mechanical and optical design of the branch-line, the differential pumping setup as well as performance characteristics are presented.
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| 4. |
- Charlesworth, A., et al.
(författare)
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Epidermolysis bullosa simplex with PLEC mutations : new phenotypes and new mutations
- 2013
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Ingår i: British Journal of Dermatology. - : Oxford University Press (OUP). - 0007-0963 .- 1365-2133. ; 168:4, s. 808-814
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Tidskriftsartikel (refereegranskat)abstract
- Background Genetic mutations in the plectin gene (PLEC) cause autosomal recessive forms of epidermolysis bullosa simplex (EBS) associated with either muscular dystrophy (EBS-MD) or pyloric atresia (EBS-PA). Phenotype-genotype analysis has suggested that EBS-MD is due mostly to genetic mutations affecting the central rod domain of plectin, and EBS-PA to mutations outside this domain. Objectives This study aimed to describe new phenotypes of patients with EBS-MD and EBS-PA, to identify novel PLEC mutations and to establish genotype-phenotype correlations. Methods Seven patients with a suspicion of EBS linked to PLEC mutations were included. A standardized clinical questionnaire was sent to the physicians in charge of each patient. Immunofluorescence studies of skin biopsies followed by molecular analysis of PLEC were performed in all patients. Results We report the first case of nonlethal EBS-PA improving with age, the first multisystemic involvement in a patient with lethal EBS-PA, and the first patients with EBS-MD with involvement of either the bladder or oesophagus. Eleven novel PLEC mutations are also reported. Conclusions Our results confirm that EBS-PA is linked to mutations in the distal exons 1-30 and 32 of PLEC. Long-term survival is possible, with skin improvement, but a delayed onset of MD is probable. While EBS-MD is linked to PLEC mutations in all exons, in most cases one of the mutations affects exon 31. The precocity of MD seems to be linked to the type and localization of the PLEC mutation(s), but no correlation with mucosal involvement has been found.
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