| 1. |
- Andreyev, A. N., et al.
(författare)
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Signatures of the Z=82 shell closure in alpha-decay process
- 2013
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Ingår i: Physical Review Letters. - : American Physical Society (APS). - 0031-9007 .- 1079-7114. ; 110:24
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Tidskriftsartikel (refereegranskat)abstract
- In recent experiments at the velocity filter Separator for Heavy Ion reaction Products (SHIP) (GSI, Darmstadt), an extended and improved set of α-decay data for more than 20 of the most neutron-deficient isotopes in the region from lead to thorium was obtained. The combined analysis of this newly available α-decay data, of which the Po186 decay is reported here, allowed us for the first time to clearly show that crossing the Z=82 shell to higher proton numbers strongly accelerates the α decay. From the experimental data, the α-particle formation probabilities are deduced following the Universal Decay Law approach. The formation probabilities are discussed in the framework of the pairing force acting among the protons and the neutrons forming the α particle. A striking resemblance between the phenomenological pairing gap deduced from experimental binding energies and the formation probabilities is noted. These findings support the conjecture that both the N=126 and Z=82 shell closures strongly influence the α-formation probability.
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| 2. |
- Kleinberger, G., et al.
(författare)
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TREM2 mutations implicated in neurodegeneration impair cell surface transport and phagocytosis
- 2014
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Ingår i: Science Translational Medicine. - : American Association for the Advancement of Science (AAAS). - 1946-6234 .- 1946-6242. ; 6:243
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Tidskriftsartikel (refereegranskat)abstract
- Genetic variants in the triggering receptor expressed on myeloid cells 2 (TREM2) have been linked to Nasu-Hakola disease, Alzheimer's disease (AD), Parkinson's disease, amyotrophic lateral sclerosis, frontotemporal dementia (FTD), and FTD-like syndrome without bone involvement. TREM2 is an innate immune receptor preferentially expressed by microglia and is involved in inflammation and phagocytosis. Whether and how TREM2 missense mutations affect TREM2 function is unclear. We report that missense mutations associated with FTD and FTD-like syndrome reduce TREM2 maturation, abolish shedding by ADAM proteases, and impair the phagocytic activity of TREM2-expressing cells. As a consequence of reduced shedding, TREM2 is virtually absent in the cerebrospinal fluid (CSF) and plasma of a patient with FTD-like syndrome. A decrease in soluble TREM2 was also observed in the CSF of patients with AD and FTD, further suggesting that reduced TREM2 function may contribute to increased risk for two neurodegenerative disorders.
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| 3. |
- Qi, Chong, et al.
(författare)
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Abrupt changes in alpha-decay systematics as a manifestation of collective nuclear modes
- 2010
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Ingår i: Physical Review C. Nuclear Physics. - 0556-2813 .- 1089-490X. ; 81:6, s. 064319-
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Tidskriftsartikel (refereegranskat)abstract
- An abrupt change in alpha-decay systematics around the N = 126 neutron shell closure is discussed. It is explained as a sudden hindrance of the clustering of the nucleons that eventually form the a particle. This is because the clustering induced by the pairing mode acting upon the four nucleons is inhibited if the configuration space does not allow a proper manifestation of the pairing collectivity.
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| 4. |
- Qi, Chong, et al.
(författare)
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On the validity of the Geiger-Nuttall alpha-decay law and its microscopic basis
- 2014
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Ingår i: Physics Letters B. - : Elsevier BV. - 0370-2693 .- 1873-2445. ; 734, s. 203-206
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Tidskriftsartikel (refereegranskat)abstract
- The Geiger-Nuttall (GN) law relates the partial alpha-decay half-life with the energy of the escaping alpha particle and contains for every isotopic chain two experimentally determined coefficients. The expression is supported by several phenomenological approaches, however its coefficients lack a fully microscopic basis. In this paper we will show that: (1) the empirical coefficients that appear in the GN law have a deep physical meaning, and (2) the GN law is successful within the restricted experimental data sets available so far, but is not valid in general. We will show that, when the dependence of logarithm values of the alpha formation probability on the neutron number is not linear or constant, the GN law is broken. For the alpha decay of neutron-deficient nucleus Po-186, the difference between the experimental half-life and that predicted by the GN law is as large as one order of magnitude.
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