| 1. |
- Jiang, Z. K, et al.
(författare)
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Lifetimes of Rydberg Levels In the Perturbed 6snp P-1,3(1) Series of Ybi
- 1995
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Ingår i: Physics Letters. Section A: General, Atomic and Solid State Physics. - : Elsevier BV. - 0375-9601. ; 204:1, s. 49-53
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Tidskriftsartikel (refereegranskat)abstract
- Using two methods: (1) three-step excitation and (2) single UV photon excitation in an atomic beam with direct observation of the decay of the fluorescence light, we have measured the lifetimes of the Rydberg levels of YbI belonging to the perturbed series 6snp J = 1 levels. The measured values have been interpreted by the multichannel quantum defect theory (MQDT).
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| 2. |
- Baoren, Che, et al.
(författare)
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Aging effects of exercise training on erythrocyte
- 1995
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Ingår i: Journal of Shanghai Physical Education Institute. - 1000-5498. ; 19:S1, s. 102-104
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Tidskriftsartikel (refereegranskat)abstract
- 长时间剧烈运动能引起循环中的红细胞损伤和老化;而长期运动训练有可能导致运动性贫血的发生,据最近的一些研究表明,尽管一次长时间剧烈运动可引起红细胞被破坏增多,但长期运动训练引起的运动性贫血,在很多情况下属于血液稀释作用造成的。本文设计6周身体训练并在训练的不同阶段进行剧烈运动,测定反映红细咆老化状态的几个敏感指标:2,3—DPG、红细胞肌酸含量及红细胞生成素等,旨在确认不同强度的训练及剧烈运动对红细胞生成和老化的影响,为进一步完善运动性贫血理论提供依据。
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| 3. |
- Che, Baoren, et al.
(författare)
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Effects of Exercise training on Erythrocyte
- 1995
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Ingår i: Journal of Shanghai Physical Education Institute. - 1000-5498. ; :s1
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Tidskriftsartikel (refereegranskat)abstract
- 长时间剧烈运动能引起循环中的红细胞损伤和老化;而长期运动训练有可能导致运动性贫血的发生,据最近的一些研究表明,尽管一次长时间剧烈运动可引起红细胞被破坏增多,但长期运动训练引起的运动性贫血,在很多情况下属于血液稀释作用造成的。本文设计6周身体训练并在训练的不同阶段进行剧烈运动,测定反映红细咆老化状态的几个敏感指标:2,3—DPG、红细胞肌酸含量及红细胞生成素等,旨在确认不同强度的训练及剧烈运动对红细胞生成和老化的影响,为进一步完善运动性贫血理论提供依据。
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| 4. |
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| 5. |
- Ni, Jiun, et al.
(författare)
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Cystatin E is a novel human cysteine proteinase inhibitor with structural resemblance to family 2 cystatins
- 1997
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Ingår i: Journal of Biological Chemistry. - 1083-351X. ; 272:16, s. 10853-10858
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Tidskriftsartikel (refereegranskat)abstract
- A new member of the human cystatin superfamily, called cystatin E, has been found by expressed sequence tag (EST) sequencing in amniotic cell and fetal skin epithelial cell cDNA libraries. The sequence of a full-length amniotic cell cDNA clone contained an open reading frame encoding a putative 28-residue signal peptide and a mature protein of 121 amino acids, including four cysteine residues and motifs of importance for the inhibitory activity of Family 2 cystatins like cystatin C. Recombinant cystatin E was produced in a baculovirus expression system and isolated. An antiserum against the recombinant protein could be used for affinity purification of cystatin E from human urine, as confirmed by N-terminal sequencing. The mature recombinant protein processed by insect cells started at amino acid 4 (cystatin C numbering), and displayed reversible inhibition of papain and cathepsin B (Ki values of 0.39 and 32 nM, respectively), in competition with substrate. Cystatin E is thus a functional cysteine proteinase inhibitor despite relatively low amino acid sequence similarities with human cystatins (26-34% identity with sequences for the Family 2 cystatins C, D, S, SN, and SA; <30% with the Family 1 cystatins, A and B, and domains 2 and 3 of the Family 3 cystatin, kininogen). Unlike other human low Mr cystatins, cystatin E is a glycoprotein, carrying an N-linked carbohydrate chain at position 108. Northern blot analysis revealed that the cystatin E gene is expressed in most human tissues, with the highest mRNA amounts found in uterus and liver. A strikingly high incidence of cystatin E clones in cDNA libraries from fetal skin epithelium and amniotic membrane cells (>0.5% of clones sequenced) indicates a protective role of cystatin E during fetal development.
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| 6. |
- Yu, Guo
(författare)
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Polyunsaturated fatty acids in relation to childhood and maternal allergic diseases
- 1998
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Doktorsavhandling (övrigt vetenskapligt/konstnärligt)abstract
- The aim of this thesis was to study polyunsaturated fatty acid (PUPA) in relation to the appem11nce of anergic diseases in children and mothers in late pregnancy and during the lactation period and to the development of atopy in children, as well as the influence of maternal PUF A on their babies.The levels of docosahexaenoic acid (DHA, C22:6n-3) and total n-3 long-chain polyunsaturated fatty acids (LCP) were lower (p = 0.01 and< 0.05) and the ratio of total n-6 to n-3 LCP was higher (p < 0.01) in serum phospholipids (PL) in 22 allergic school children than in 23 controls. The levels of docosapentaenoic acid (DPA, C22:5n-3) and the ratio of arachidonic acid (AA, C20:4n-6) to its precursor dihomo-y-linolenic acid (DHGLA, C20:3n-6) were also lower in 12 children with positive skin prick test (SPT), as compared to SPT negative children (both p < 0.05). Higher levels of C20:2n-6 and lower EPA were recorded in allergic children with serum IgE above the median level (56kU/L), as compared to those with lower IgE levels,The levels of DHGLA, eicosapentaenoic acid (EPA, C20:5n-3), DPA and DHA were all lower in milk total lipid (TL) obtained from atopic, as compared to non-atopic mothers after one month oflactation (all p < 0.05). Similarly, the lower levels of DHGLA, AA, EPA, DPA and DHA were also observed in serum 1L of the atopic mothers at the time of delivery (all P < 0.05), while the levels of a-linolenic acid and C20:2n-6 were higher. Several ratios of n-6 to n- 3 LCP in mature milk at 1 and 3 months were significantly higher in atopic than non-atopic mothers (all p < 0.05). An n-3 fatty acid C20:4n-3 was present in human milk, but not in the blood samples of the mothers and children.The levels of most of the individual PUFA con·elated well in blood of non-allergic children and in colostrum and mature milk of non-atopic mothers (r > 0.5, p < 0.01 as significant), but the correlations were largely absent in the atopic groups. Furthmore, a correlation between linoleic acid (LA, C18:2n-6) and AA levels was observed in serum samples of non-allergic (r = 0.64, p < 0.001), but not allergic mothers at delivery (r = 0.25, p > 0.05).There were some correlations between AA and its precursor DHGLA and metabolite C22:4n-6 and between several n-3 and n-6 LCP, i.e. DPA and C22:4n-6, DHA and DHGLA and AA in semm phospholipids from cord blood of children without a family history of allergy (FHA) and who did not develop allergic diseases during the first 6 years of life. These relationships between the LCP levels were not seen in children with a FHA and in those who developed allergic diseases in later life. Furthermore, an abnormal PUF A composition in maternal blood and breast milk was related to the appearance of allergy in their children.The LA levels correlated in 22 non-allergic mothers and their babies at the time of delivery (r = 0.53, p = 0.01). Furthermore, the serum levels of maternal DHGLA correlated with some LCP levels in cord serum of their babies, i.e. AA, C22:4 and DHA (all r= 0.65, p = 0.001). None of these relationships were observed in 25 allergic mothers and their babies.The findings confirmed an abnormal PUF A composition in allergic children, in allergic mothers at the time of delivery and early lactation period and in newborn infants who developed allergic diseases during the first 6 years of life. The latter finding indicates that an abnormality of PUF A composition may be primary in allergic diseases. An impaired 0-6-desaturase activity in allergic diseases could not be confirmed, The presence of n-3 series fatty acid C20:4n-3 in human milk but not blood samples may contribute to the anti-inflammatory effect of human milk,
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| 7. |
- Yu, Ji-Guo, et al.
(författare)
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Effects of exercise intensity on muscle tissue free radical metabolism and serum enzymes
- 1996
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Ingår i: Journal of Physical Education. - 1006-7116. ; 4:2, s. 20-22
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Tidskriftsartikel (refereegranskat)abstract
- 以二种不同强度运动,测定运动结束后不同时间内股四头肌MDA含量及SOD、GSH-PX活性,并测定了相应时间内的血清酶活性.结果发现长时间力竭性运动引起肌组织MDA含量增加的同时,SOD和GSH-PX活性下降,血清CK、LDH及Mb含量上升;短时间间歇运动后,尽管MDA变化不明显,但SOD和GSH-PX趋于上升,且血清酶活性也在运动后有上升表现.
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| 8. |
- Yu, Ji-Guo, et al.
(författare)
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Exercise training and Blood Volume
- 1995
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Ingår i: Journal of Shandong Physical Education Institute. - 1006-2076. ; 11:1, s. 31-35
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Forskningsöversikt (refereegranskat)abstract
- Both an acute sport and long-term exercise can result in changes in blood volume.Studying these changes, finding their changing patterns and knowing their effects on sport ability have been the problems of a lot of reserchers. In this review. previous works of other peoples were reviewed and analysed in order to provide a clue forfurther studies.
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| 9. |
- Yu, Ji-Guo, et al.
(författare)
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Exercise training induces increased lipid peroxidation in red blood cells
- 1997
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Ingår i: Chinese Journal of Sports Medicine. - 1000-6710. ; 16:2, s. 145-147
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Tidskriftsartikel (refereegranskat)abstract
- 本文采用２种不同的运动强度对受试者进行６周的运动训练，并在训练的不同阶段进行剧烈运动，测定了运动前后反映红细胞老化状态的各项指标：膜ＭＤＡ含量、Ｎａ，Ｋ－ＡＴＰａｓｅ活性、ＳＯＤ活性以及红细胞唾液酸含量。结果表明，不同强度的运动训练均引起血液系统中年轻红细胞比例上升，但影响的程度有所差异。不同训练阶段进行剧烈运动，由于血液系统对训练的适应状态不同，其影响也有所不同，但剧烈运动的结果仍引起红细胞老化加速。
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| 10. |
- Yu, Ji-Guo, et al.
(författare)
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Oxidative damage in red blood cells induced by exercise training
- 1996
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Ingår i: Yantai Teachers University Journal. - 1004-4930. ; 12:2, s. 138-141
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Tidskriftsartikel (refereegranskat)abstract
- 本文采用２种不同的运动强度对受试者进行６周的运动训练，并在训练的不同阶段进行剧烈运动，测定了运动前后反映红细胞老化状态的各项指标：膜ＭＤＡ含量、Ｎａ，Ｋ－ＡＴＰａｓｅ活性、ＳＯＤ活性以及红细胞唾液酸含量。结果表明，不同强度的运动训练均引起血液系统中年轻红细胞比例上升，但影响的程度有所差异。不同训练阶段进行剧烈运动，由于血液系统对训练的适应状态不同，其影响也有所不同，但剧烈运动的结果仍引起红细胞老化加速。
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