| 1. |
- Andersson, Eva M., 1968, et al.
(författare)
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Road traffic noise, air pollution and cardiovascular events in a Swedish cohort
- 2020
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Ingår i: Environmental Research. - : Elsevier BV. - 0013-9351. ; 185
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Tidskriftsartikel (refereegranskat)abstract
- Urbanization and increasing road traffic cause exposure to both noise and air pollution. While the levels of air pollutants such as nitrogen oxides (NOx) have decreased in Sweden during the past decades, exposure to traffic noise has increased. The association with cardiovascular morbidity is less well established for noise than for air pollution, and most studies have only studied one of the two highly spatially correlated exposures. The Swedish Primary Prevention Study cohort consists of men aged 47 to 55 when first examined in 1970-1973. The cohort members were linked to the Swedish patient registry through their personal identity number and followed until first cardiovascular event 1970-2011. The address history during the entire study period was used to assign annual modelled residential exposure to road traffic noise and NOx. The Cox proportional hazards model with age on the time axis and time-varying exposures were used in the analysis. The results for 6304 men showed a non-significant increased risk of cardiovascular disease for long-term road traffic noise at the home address, after adjusting for air pollution. The hazard ratios were 1.08 (95% CI 0.90-1.28) for cardiovascular mortality, 1.14 (95% CI 0.96-1.36) for ischemic heart disease incidence and 1.07 (95% CI 0.85-1.36) for stroke incidence, for noise above 60 dB, compared to below 50 dB. This study found some support for cardiovascular health effects of long-term exposure to road traffic noise above 60 dB, after having accounted for exposure to air pollution.
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| 2. |
- Bartels, Susanne, et al.
(författare)
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The impact of nocturnal road traffic noise, bedroom window orientation, and work-related stress on subjective sleep quality: results of a cross-sectional study among working women.
- 2021
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Ingår i: International archives of occupational and environmental health. - : Springer Science and Business Media LLC. - 1432-1246 .- 0340-0131. ; 94, s. 1523-1536
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Tidskriftsartikel (refereegranskat)abstract
- To examine the effect of work-related stress and road noise exposure on self-rated sleep and potential additive interaction effects.Sleep and predictor variables were surveyed within two subsamples with 2191 and 1764 working women in a cross-sectional study. Sleep was assessed using a single question on general sleep quality and four questions on specific sleep problems and subsequently dichotomized (poor sleep vs. no poor sleep). Work-related stress was operationalized by job strain and effort-reward imbalance. Nocturnal exposure to road traffic noise was assessed as (a) the orientation of the bedroom window to a quiet façade vs. a low-, medium- or high-trafficked street and (b) energy-equivalent sound pressure levels for night-time modelled at the most exposed façade (Lnight). We distinguished between low (<45dB(A)), medium (45-50dB(A)) and high exposure (>50dB(A)).Poor sleep was associated with job strain and effort-reward imbalance. The prevalence of poor sleep did not increase with increasing Lnight, but bedroom window orientation showed a non-significant trend. A quiet façade had a protective effect on sleep in each Lnight category. We found a non-significant trend for an additive interaction between bedroom window orientation and job strain.Noise levels modelled for the most exposed façade likely overestimate the actual exposure and thus may not be a precise predictor of poor sleep. Bedroom window orientation seems more relevant. Potential additive interaction effects between bedroom window orientation and job strain should be considered when interpreting epidemiological study results on noise-induced sleep disturbances.
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| 3. |
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| 4. |
- Kilbo Edlund, Karl, et al.
(författare)
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Long-term exposure to air pollution, coronary artery calcification, and carotid artery plaques in the population-based Swedish SCAPIS Gothenburg cohort.
- 2022
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Ingår i: Environmental research. - : Elsevier BV. - 1096-0953 .- 0013-9351. ; 214:Pt 2
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Tidskriftsartikel (refereegranskat)abstract
- Long-term exposure to air pollution is associated with cardiovascular events. A main suggested mechanism is that air pollution accelerates the progression of atherosclerosis, yet current evidence is inconsistent regarding the association between air pollution and coronary artery and carotid artery atherosclerosis, which are well-established causes of myocardial infarction and stroke. We studied associations between low levels of long-term air pollution, coronary artery calcium (CAC) score, and the prevalence and area of carotid artery plaques, in a middle-aged population-based cohort. The Swedish CArdioPulmonary bioImage Study (SCAPIS) Gothenburg cohort was recruited during 2013-2017 and thoroughly examined for cardiovascular risk factors, including computed tomography of the heart and ultrasonography of the carotid arteries. In 5070 participants (age 50-64 years), yearly residential exposures to air pollution (PM2.5, PM10, PMcoarse, NOx, and exhaust-specific PM2.5 1990-2015) were estimated using high-resolution dispersion models. We used Poisson regression to examine associations between long-term (26 years' mean) exposure to air pollutants and CAC score, and prevalence of carotid artery plaques, adjusted for potential confounders. Among participants with carotid artery plaques, we also examined the association with plaque area using linear regression. Mean exposure to PM2.5 was low by international standards (8.5μg/m3). There were no consistent associations between long-term total PM2.5 exposure and CAC score or presence of carotid artery plaques, but an association between total PM2.5 and larger plaque area in participants with carotid plaques. Associations with traffic-related air pollutants were consistently positive for both a high CAC score and bilateral carotid artery plaques. These associations were independent of road traffic noise. We found stronger associations among men and participants with cardiovascular risk factors. The results lend some support to atherosclerosis as a main modifiable pathway between low levels of traffic-related ambient air pollution and cardiovascular disease, especially in vulnerable individuals.
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| 5. |
- Pyko, Andrei, et al.
(författare)
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Long-Term Exposure to Transportation Noise and Ischemic Heart Disease: A Pooled Analysis of Nine Scandinavian Cohorts.
- 2023
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Ingår i: Environmental health perspectives. - : Environmental Health Perspectives. - 1552-9924 .- 0091-6765. ; 131:1
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Tidskriftsartikel (refereegranskat)abstract
- Transportation noise may induce cardiovascular disease, but the public health implications are unclear.The study aimed to assess exposure-response relationships for different transportation noise sources and ischemic heart disease (IHD), including subtypes.Pooled analyses were performed of nine cohorts from Denmark and Sweden, together including 132,801 subjects. Time-weighted long-term exposure to road, railway, and aircraft noise, as well as air pollution, was estimated based on residential histories. Hazard ratios (HRs) were calculated using Cox proportional hazards models following adjustment for lifestyle and socioeconomic risk factors.A total of 22,459 incident cases of IHD were identified during follow-up from national patient and mortality registers, including 7,682 cases of myocardial infarction. The adjusted HR for IHD was 1.03 [95% confidence interval (CI) 1.00, 1.05] per 10 dB Lden for both road and railway noise exposure during 5 y prior to the event. Higher risks were indicated for IHD excluding angina pectoris cases, with HRs of 1.06 (95% CI: 1.03, 1.08) and 1.05 (95% CI: 1.01, 1.08) per 10 dB Lden for road and railway noise, respectively. Corresponding HRs for myocardial infarction were 1.02 (95% CI: 0.99, 1.05) and 1.04 (95% CI: 0.99, 1.08). Increased risks were observed for aircraft noise but without clear exposure-response relations. A threshold at around 55 dB Lden was suggested in the exposure-response relation for road traffic noise and IHD.Exposure to road, railway, and aircraft noise in the prior 5 y was associated with an increased risk of IHD, particularly after exclusion of angina pectoris cases, which are less well identified in the registries. https://doi.org/10.1289/EHP10745.
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| 6. |
- Roswall, Nina, et al.
(författare)
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Long-term exposure to traffic noise and risk of incident colon cancer : A pooled study of eleven Nordic cohorts
- 2023
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Ingår i: Environmental Research. - : Elsevier BV. - 0013-9351 .- 1096-0953. ; 224
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Tidskriftsartikel (refereegranskat)abstract
- BackgroundColon cancer incidence is rising globally, and factors pertaining to urbanization have been proposed involved in this development. Traffic noise may increase colon cancer risk by causing sleep disturbance and stress, thereby inducing known colon cancer risk-factors, e.g. obesity, diabetes, physical inactivity, and alcohol consumption, but few studies have examined this.ObjectivesThe objective of this study was to investigate the association between traffic noise and colon cancer (all, proximal, distal) in a pooled population of 11 Nordic cohorts, totaling 155,203 persons.MethodsWe identified residential address history and estimated road, railway, and aircraft noise, as well as air pollution, for all addresses, using similar exposure models across cohorts. Colon cancer cases were identified through national registries. We analyzed data using Cox Proportional Hazards Models, adjusting main models for harmonized sociodemographic and lifestyle data.ResultsDuring follow-up (median 18.8 years), 2757 colon cancer cases developed. We found a hazard ratio (HR) of 1.05 (95% confidence interval (CI): 0.99–1.10) per 10-dB higher 5-year mean time-weighted road traffic noise. In sub-type analyses, the association seemed confined to distal colon cancer: HR 1.06 (95% CI: 0.98–1.14). Railway and aircraft noise was not associated with colon cancer, albeit there was some indication in sub-type analyses that railway noise may also be associated with distal colon cancer. In interaction-analyses, the association between road traffic noise and colon cancer was strongest among obese persons and those with high NO2-exposure.DiscussionA prominent study strength is the large population with harmonized data across eleven cohorts, and the complete address-history during follow-up. However, each cohort estimated noise independently, and only at the most exposed façade, which may introduce exposure misclassification. Despite this, the results of this pooled study suggest that traffic noise may be a risk factor for colon cancer, especially of distal origin.
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| 7. |
- Roswall, N., et al.
(författare)
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Long-Term Exposure to Transportation Noise and Risk of Incident Stroke: A Pooled Study of Nine Scandinavian Cohorts
- 2021
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Ingår i: Environmental Health Perspectives. - : Environmental Health Perspectives. - 0091-6765 .- 1552-9924. ; 129:10
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Tidskriftsartikel (refereegranskat)abstract
- BACKGROUND: Transportation noise is increasingly acknowledged as a cardiovascular risk factor, but the evidence base for an association with stroke is sparse. OBJECTIVE: We aimed to investigate the association between transportation noise and stroke incidence in a large Scandinavian population. METHODS: We harmonized and pooled data from nine Scandinavian cohorts (seven Swedish, two Danish), totaling 135,951 participants. We identified residential address history and estimated road, railway, and aircraft noise for all addresses. Information on stroke incidence was acquired through linkage to national patient and mortality registries. We analyzed data using Cox proportional hazards models, including socioeconomic and lifestyle confounders, and air pollution. RESULTS: During follow-up (median = 19.5 y), 11,056 stroke cases were identified. Road traffic noise (Lden) was associated with risk of stroke, with a hazard ratio (HR) of 1.06 [95% confidence interval (CI): 1.03, 1.08] per 10-dB higher 5-y mean time-weighted exposure in analyses adjusted for individual- and area-level socioeconomic covariates. The association was approximately linear and persisted after adjustment for air pollution [particulate matter (PM) with an aerodynamic diameter of <= 2.5 mu m (PM2.5) and NO2]. Stroke was associated with moderate levels of 5-y aircraft noise exposure (40-50 vs. <= 40 dB) (HR = 1.12; 95% CI: 0.99, 1.27), but not with higher exposure (>= 50 dB, HR = 0.94; 95% CI: 0.79, 1.11). Railway noise was not associated with stroke. DISCUSSION: In this pooled study, road traffic noise was associated with a higher risk of stroke. This finding supports road traffic noise as an important cardiovascular risk factor that should be included when estimating the burden of disease due to traffic noise.
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| 8. |
- Seidler, A., et al.
(författare)
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Health effects of railway-induced vibration combined with railway noise-A systematic review with exposure-effect curves
- 2023
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Ingår i: Environmental Research. - 0013-9351. ; 233
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Tidskriftsartikel (refereegranskat)abstract
- Background: The combined health impact of concurrent railway noise and railway vibration exposure is not yet well understood. Objectives: This systematic review gives an overview of epidemiological studies on health effects from railway vibration, aiming to quantify this association with exposure-effect curves. Moreover, the combined health effects of vibration and concurrent noise were investigated.Methods: We converted the vibration metric to an equivalent noise level and calculated an overall noise level by energetically summing the equivalent and railway noise level. The combined health effect was determined by using published evidence-based exposure-effect formulas.Results: Studies included in this systematic review predominately investigated annoyance and self-reported sleep disturbances; no studies on manifest diseases were identified. For the combined effects of vibration and noise on "total" annoyance, the results based on the pooled analysis of CargoVibes project are recommended as conservative approach.Discussion: Converting railway vibration into equivalent noise levels in dB may offer a pragmatic approach to assess the combined health effects of railway noise and railway vibration exposure. Future studies should include cardiovascular and mental diseases in addition to vibration-induced annoyance and sleep disturbances. Furthermore, future studies should include in-depth investigations of the interaction between railway noise and railway vibration to allow for a more accurate assessment of the railway-induced burden of disease.
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| 9. |
- Smith, Michael, et al.
(författare)
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A laboratory study on the effects of wind turbine noise on sleep: Results of the polysomnographic WiTNES study
- 2020
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Ingår i: Sleep. - : Oxford University Press (OUP). - 1550-9109 .- 0161-8105. ; 43:9, s. 1-14
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Tidskriftsartikel (refereegranskat)abstract
- Study Objectives: Assess the physiologic and self-reported effects of wind turbine noise (WTN) on sleep. Methods: Laboratory sleep study (n = 50 participants: n = 24 living close to wind turbines and n = 26 as a reference group) using polysomnography, electrocardiography, salivary cortisol, and questionnaire endpoints. Three consecutive nights (23:00-07:00): one habituation followed by a randomized quiet Control and an intervention night with synthesized 32 dB LAEq WTN. Noise in WTN nights simulated closed and ajar windows and low and high amplitude modulation depth. Results: There was a longer rapid eye movement (REM) sleep latency (+16.8 min) and lower amount of REM sleep (-11.1 min, -2.2%) in WTN nights. Other measures of objective sleep did not differ significantly between nights, including key indicators of sleep disturbance (sleep efficiency: Control 86.6%, WTN 84.2%; wakefulness after sleep onset: Control 45.2 min, WTN 52.3 min; awakenings: Control n = 11.4, WTN n = 11.5) or the cortisol awakening response. Self-reported sleep was consistently rated as worse following WTN nights, and individuals living close to wind turbines had worse self-reported sleep in both the Control and WTN nights than the reference group. Conclusions: Amplitude-modulated continuous WTN may impact on self-assessed and some aspects of physiologic sleep. Future studies are needed to generalize these findings outside of the laboratory and should include more exposure nights and further examine possible habituation or sensitization.
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| 10. |
- Thacher, Jesse D., et al.
(författare)
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Occupational noise exposure and risk of incident stroke: a pooled study of five Scandinavian cohorts
- 2022
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Ingår i: OCCUPATIONAL AND ENVIRONMENTAL MEDICINE. - : BMJ. - 1351-0711 .- 1470-7926. ; 79:9, s. 594-601
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Tidskriftsartikel (refereegranskat)abstract
- Objectives To investigate the association between occupational noise exposure and stroke incidence in a pooled study of five Scandinavian cohorts (NordSOUND). Methods We pooled and harmonised data from five Scandinavian cohorts resulting in 78 389 participants. We obtained job data from national registries or questionnaires and recoded these to match a job-exposure matrix developed in Sweden, which specified the annual average daily noise exposure in five exposure classes (L-Aeq8h): <70, 70-74, 75-79, 80-84, >= 85 dB(A). We identified residential address history and estimated 1-year average road traffic noise at baseline. Using national patient and mortality registers, we identified 7777 stroke cases with a median follow-up of 20.2 years. Analyses were conducted using Cox proportional hazards models adjusting for individual and area-level potential confounders. Results Exposure to occupational noise at baseline was not associated with overall stroke in the fully adjusted models. For ischaemic stroke, occupational noise was associated with HRs (95% CI) of 1.08 (0.98 to 1.20), 1.09 (0.97 to 1.24) and 1.06 (0.92 to 1.21) in the 75-79, 80-84 and >= 85 dB(A) exposure groups, compared with <70 dB(A), respectively. In subanalyses using time-varying occupational noise exposure, we observed an indication of higher stroke risk among the most exposed (>= 85 dB(A)), particularly when restricting analyses to people exposed to occupational noise within the last year (HR: 1.27; 95% CI: 0.99 to 1.63). Conclusions We found no association between occupational noise and risk of overall stroke after adjustment for confounders. However, the non-significantly increased risk of ischaemic stroke warrants further investigation.
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| 11. |
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| 12. |
- Wallas, A. E., et al.
(författare)
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Noise exposure and childhood asthma up to adolescence
- 2020
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Ingår i: Environmental Research. - : Elsevier BV. - 0013-9351 .- 1096-0953. ; 185
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Tidskriftsartikel (refereegranskat)abstract
- Objective: Increasing evidence indicates aggravation of immune-mediated diseases due to physiological and psychological stress. Noise is a stressor, however, little is known about its effects on children's respiratory health. This study investigates the association between pre- or postnatal road traffic or occupational noise exposure and asthma as well as related symptoms from infancy to adolescence. Methods: The study was conducted in the Swedish birth cohort BAMSE, including over 4000 participants followed with repeated questionnaires and clinical tests until 16 years of age. Pre- and postnatal residential road traffic noise was assessed by estimating time-weighted average noise levels at the most exposed facade. Maternal occupational noise exposure during pregnancy was evaluated using a job-exposure-matrix. The associations between noise exposure and asthma-related outcomes were explored using logistic regression and generalised estimating equations. Results: We observed non-significant associations for asthma ever up to 16 years with residential road traffic noise exposure in infancy >= 55 dB(Lden) (adjusted OR = 1.22; 95% CI 0.90-1.65), as well as prenatal occupational noise exposure >= 80 dB(LAeq,8h) (1.18, 0.85-1.62). In longitudinal analyses, however, no clear associations between pre- or postnatal exposure to residential road traffic noise, or average exposure to noise since birth, were detected in relation to asthma or wheeze until 16 years. Conclusion: We did not find a clear overall association between exposure to noise during different time periods and asthma or wheeze up to adolescence.
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| 13. |
- Winroth, Julia, et al.
(författare)
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Child-Centred Room Acoustic Parameters of Public Preschools in Sweden
- 2023
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Ingår i: Buildings. - 2075-5309. ; 13:11
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Tidskriftsartikel (refereegranskat)abstract
- Preschool should promote children’s well-being and development, but the indoor sound environment is commonly problematic. The aim of our research project Supportive Preschool ACoustic Environment (SPACE) is to identify acoustic quality factors resulting in a supportive sound environment for children. This paper presents the first phase of the project where acoustic conditions were measured in unoccupied preschool rooms and analysed in terms of reverberation time, early decay time, sound strength, speech clarity, unoccupied sound pressure levels, and several room features. The results were compared with current target values, building year, and socioeconomic status of the preschool. A child perspective on room acoustics was, in addition, applied and it was revealed that children may be exposed to a lower sound strength than adults, and that adults may have better speech intelligibility conditions than children. Rooms in newer buildings had a longer reverberation time in the 125 Hz band, lower unoccupied levels, and lower sound strength. These differences could be explained by the trend towards larger rooms and porous acoustic ceilings in newer buildings. We found no significant correlations with the socioeconomic status. Ongoing work will facilitate an analysis of the correlation between the room acoustic parameters, the sound environment and children’s perception.
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| 14. |
- Ögren, Mikael, 1972-, et al.
(författare)
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Beräkning av maximal bullernivå med CNOSSOS-EU
- 2021
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Rapport (övrigt vetenskapligt/konstnärligt)abstract
- När man beräknar buller från väg-, spår- och flygtrafik i Sverige så har man sedan lång tid tillbaka tagit fram två mått, den ekvivalenta och den maximala nivån. Den ekvivalenta ljudtrycksnivån är ett mått på den totala trafiken och är ett slags medelvärde, medan den maximala ljudtrycksnivån är ett mått på den högsta nivån som uppstår. Det är relativt enkelt att definiera vad den ekvivalenta nivån är, och det finns inte olika definitioner eller konkurrerande beskrivningar från olika aktörer. För maximalnivån är det dock mera komplext, exempelvis definieras ofta maximalnivån från vägtrafik som den maximala nivån som man beräknar om trafiken passerar ett fordon i taget med konstant hastighet och med tom väg i övrigt. För mätning av maximal nivå krävs viss manuell hantering av mätdata, speciellt på platser med hög trafik. Samma frågeställning är inte så viktig för tåg- och flygtrafik, eftersom det är ovanligt med möten eller samtidiga passager. För strategiska kartläggningar skall den nya EU-gemensamma beräkningsmetoden Cnossos-EU användas från och med 2022 års kartläggning. Denna beräkningsmetod kan endast beräkna ekvivalenta mått och har inga indata eller metodbeskrivningar för beräkningar av maximal nivå. I denna rapport redovisas genom ett antal beräkningsexempel för väg- och spårtrafik hur man utgående från Cnossos-EU ändå kan beräkna den maximala nivån om man lägger till indata och modifierar beräkningsmetodiken.
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| 15. |
- Ögren, Mikael, 1972-, et al.
(författare)
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Bullerkartläggning inom den hälsorelaterade miljöövervakningen (HÄMI)
- 2022
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Rapport (övrigt vetenskapligt/konstnärligt)abstract
- Inom Naturvårdsverkets hälsorelaterade miljöövervakning (HÄMI) har flera nationella kartläggningar av hur Sveriges befolkning exponeras för buller från flyg-, väg- och spårtrafik genomförts sedan år 2000, med något olika metoder och underlag. Vart femte år genomförs också en kartläggning av bullerexponering i delar av landet som rapporteras till EU via omgivningsbullerdirektivet. Under 2022 kommer dessa beräkningar genomföras nära högtrafikerade vägar, flygplatser och spår samt heltäckande inom de 18 största kommunerna, av olika utförare.Att på regelbunden basis göra nationella kartläggningar av trafikbuller och dess hälsopåverkan med en standardiserad och reproducerbar metodik är viktigt som underlag för bedömningar av trafikbuller som folkhälsoproblem. Vi rekommenderar att detta utförs minst vart femte år. Beräknade bullerindikatorer bör i första hand vara de internationellt gångbara Lden och Lnight, men även de indikatorer som används i Sverige (LAeq,24h, Lmax och FBN) kan beräknas. Trots utmaningarna med trafikdata och beräkningstid är vår bedömning att en fullständig beräkning baserad på antingen Nord2000 eller Cnossos-EU bör ligga till grund för framtida nationella kartläggningar av exponering för buller inom HÄMI. Erfarenheter i Danmark visar att det är möjligt, och det ger det bästa underlaget i termer av noggrannhet och ger störst möjligheter till exempelvis analyser av hälsoeffekter i befolkningen. Om det bedöms som för arbetskrävande att samla in trafikuppgifter i den skala som krävs i Sverige kan schabloner och beräknade trafikflöden användas, förutsatt att de genomförs med samma metodik för alla delar av landet och på samma sätt över tid så att det blir möjligt att följa tidstrender och effekter av förändringar i samhället. Därför bör också kommande kartläggningar spara använd indata och dokumentera genomförandet så att metodiken kan upprepas vid nästa tillfälle, och framtida utvecklingar av beräknings- och analysmetoder kan nyttjas till fullo även bakåt i tiden.
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| 16. |
- Ögren, Mikael, 1972, et al.
(författare)
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Ground and Building Vibration Estimation for Health Impact Research
- 2021
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Ingår i: Notes on Numerical Fluid Mechanics and Multidisciplinary Design. - Cham : Springer Nature. - 1612-2909 .- 1860-0824. ; , s. 487-495
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Bokkapitel (refereegranskat)abstract
- Building vibration from railway traffic is relatively common in Sweden. The vibration causes annoyance and interferes with sleep. Less is known about long term health effects of living in dwellings exposed to vibration, which is why a research project named EpiVib has been started where more than 6000 persons living close to railways have been recruited into a health cohort. Therefore we need to estimate the vibration exposure. However, the number of dwellings makes it difficult to use measurements for all, and limits how much data on geology can be collected. Instead, a semi-empirical model was developed based on 829 measurements in the area and geology data from official maps. The resulting method divides all soil types in the area into three classes and then estimates the vibration for different distances to the railway, and different main layer depths.
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| 17. |
- Ögren, Mikael, 1972-, et al.
(författare)
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Nord2000 spår : Beräkning av spårkorrektion från ytråhet på rälen för bullerberäkningar
- 2024
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Rapport (övrigt vetenskapligt/konstnärligt)abstract
- I samband med att nya emissionsdata för svenska tåg togs fram för den EU-gemensamma beräkningsmetoden Cnossos-EU och för Nord2000 skapades en korrektion för varje mätplats utifrån den uppmätta ytråheten på rälen. Detta PM beskriver en förenklad metod för att beräkna motsvarande korrektion baserad på den uppmätta ytråheten som sedan går att använda både för beräkningar med den Nordiska metoden reviderad 1996 och med Nord2000.
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| 18. |
- Ögren, Mikael, 1972-, et al.
(författare)
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Nord2000 spår : Svenska emissionsdata för beräkning av buller från spårburen trafik med hjälp av Nord 2000
- 2023
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Rapport (övrigt vetenskapligt/konstnärligt)abstract
- Denna rapport beskriver emissionsdata för beräkningav buller från spårtrafik i Sverige med den Nordiskaberäkningsmetoden Nord2000. Emissionsdata ärframtagna för olika tågtyper utifrån mer än 700uppmätta tågpassager fördelade över 9 mätplatser,samt från mätningar av ytråheten på rälhuvudet pådessa platser. Totalt presenteras indataparametrar för8 motorvagnar (inklusive X2) och godståg medgjutjärnsblock skapade genom statistisk analys avmätresultaten i enlighet med ett utkast tilleuropastandard (prEN 17936). För motorvagnen ER1,godståg med skivbroms eller K-block och för lokdragnapersontåg var mätunderlaget för litet och deframtagna indata är istället baserade på bådeuppmätta passa ger och/eller äldre indata.
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