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| 2. |
- Frisén, Lars, 1939, et al.
(författare)
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Detection of extracranial carotid stenosis by computed tomography.
- 1979
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Ingår i: Lancet (London, England). - 0140-6736. ; 1:8130, s. 1319-20
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Tidskriftsartikel (refereegranskat)abstract
- Ten patients with transient ischaemic attacks (T.I.A.) within the carotid territory were examined by aortic-arch angiography and computed tomography (C.T.) of the neck. C.T. showed evidence of carotid calcifications in all 17 angiographically abnormal arteries, and there was no evidence of calcification in the 3 normal arteries. C.T. of the neck promises to be a useful technique for the investigation of patients with T.I.A.s.
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| 3. |
- Frisén, Lars, 1939, et al.
(författare)
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Sectorial optic atrophy and homonymous, horizontal sectoranopia: a lateral choroidal artery syndrome?
- 1978
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Ingår i: Journal of neurology, neurosurgery, and psychiatry. - 0022-3050. ; 41:4, s. 374-80
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Tidskriftsartikel (refereegranskat)abstract
- Two patients with sectorial optic atrophy and homonymous, horizontal sectoranopia are described. Neuroradiological investigations localised the visual pathway lesion to the lateral geniculate body. The peculiar nature of the field defect and the optic atrophy appeared explicable in terms of ischaemia in the territory of the lateral choroidal artery.
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| 4. |
- Frisén, Lars, 1939, et al.
(författare)
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Spectrum of optic nerve hypoplasia.
- 1978
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Ingår i: The British journal of ophthalmology. - 0007-1161. ; 62:1, s. 7-15
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Tidskriftsartikel (refereegranskat)abstract
- Optic nerve hypoplasia is a non-progressive condition characterised by subnormal vision and a subnormal number of optic nerve axons. It may be unilateral or bilateral, isolated or combined with other defects. Analysis of fundus photographs from a series of 7 patients with a stationary abnormality of different degrees showed that the functional defects could be closely correlated with defects in the retinal nerve fibre layer. Our observations show that the condition has a wide range of both functional and anatomical defects and that a subnormal diameter of the optic disc is not a requisite for the diagnosis. Presumably, there is also a wide variety of causes, not only a primary failure of development of retinal ganglion cells. We suggest that optic nerve hypoplasia can be viewed as a non-specific manifestation of damage to the visual system, sustained any time before its full development.
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