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- Papazafeiropoulos, Anastasios K., et al.
(författare)
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Performance of Massive MIMO Uplink With Zero-Forcing Receivers Under Delayed Channels
- 2017
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Ingår i: IEEE Transactions on Vehicular Technology. - : IEEE-INST ELECTRICAL ELECTRONICS ENGINEERS INC. - 0018-9545 .- 1939-9359. ; 66:4, s. 3158-3169
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Tidskriftsartikel (refereegranskat)abstract
- In this paper, we analyze the performance of the up-link communication of massive multicell multiple-input multiple-output (MIMO) systems under the effects of pilot contamination and delayed channels because of terminal mobility. The base stations (BSs) estimate the channels through the uplink training and then use zero-forcing (ZF) processing to decode the transmit signals from the users. The probability density function (pdf) of the signal-to-interference-plus-noise ratio (SINR) is derived for any finite number of antennas. From this pdf, we derive an achievable ergodic rate with a finite number of BS antennas in closed form. Insights into the impact of the Doppler shift (due to terminal mobility) at the low signal-to-noise ratio (SNR) regimes are exposed. In addition, the effects on the outage probability are investigated. Furthermore, the power scaling law and the asymptotic performance result by infinitely increasing the numbers of antennas and terminals (while their ratio is fixed) are provided. The numerical results demonstrate the performance loss for various Doppler shifts. Among the interesting observations revealed is that massive MIMO is favorable even under channel aging conditions.
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- Vornhagen, J., et al.
(författare)
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Group B streptococcus exploits vaginal epithelial exfoliation for ascending infection
- 2018
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Ingår i: Journal of Clinical Investigation. - : American Society for Clinical Investigation. - 0021-9738 .- 1558-8238. ; 128:5, s. 1985-1999
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Tidskriftsartikel (refereegranskat)abstract
- Thirteen percent of pregnancies result in preterm birth or stillbirth, accounting for fifteen million preterm births and three and a half million deaths annually. A significant cause of these adverse pregnancy outcomes is in utero infection by vaginal microorganisms. To establish an in utero infection, vaginal microbes enter the uterus by ascending infection; however, the mechanisms by which this occurs are unknown. Using both in vitro and murine models of vaginal colonization and ascending infection, we demonstrate how a vaginal microbe, group B streptococcus (GBS), which is frequently associated with adverse pregnancy outcomes, uses vaginal exfoliation for ascending infection. GBS induces vaginal epithelial exfoliation by activation of integrin and beta-catenin signaling. However, exfoliation did not diminish GBS vaginal colonization as reported for other vaginal microbes. Rather, vaginal exfoliation increased bacterial dissemination and ascending GBS infection, and abrogation of exfoliation reduced ascending infection and improved pregnancy outcomes. Thus, for some vaginal bacteria, exfoliation promotes ascending infection rather than preventing colonization. Our study provides insight into mechanisms of ascending infection by vaginal microbes.
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