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Träfflista för sökning "WFRF:(Wålinder Robert) srt2:(2000-2004)"

Sökning: WFRF:(Wålinder Robert) > (2000-2004)

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  • Mattsson, Magnus, et al. (författare)
  • Mixing and displacement ventilation compared in classrooms; distribution of particles, cat allergen and CO2
  • 2003
  • Ingår i: Proceedings from Healthy Buildings 2003. ; , s. 458-464
  • Konferensbidrag (refereegranskat)abstract
    • Mixing ventilation and displacement ventilation were compared in an intervention study in classrooms. Particles, cat allergen and CO2, were measured in classroom air at different levels above the floor, during regular lessons. With mixing ventilation, the particle concentration tended to decrease with height, with a stronger gradient occurring for larger particles. With displacement ventilation, the particle concentration increased with height, except for particles >25 µm. The displacement system thus tended to have a slight upward displacement effect on most of the particles. Significant correlations were found between concentrations of cat allergen and particles in the size fraction 1–10 µm. The particle and cat allergen concentration at breathing height did not, however, differ significantly between the two ventilation systems. CO2 was about 10% lower with displacement ventilation. A fairly high level of physical activity of the pupils is believed to have had significant dispersing effect on the airborne contaminants.
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3.
  • Wålinder, Robert, et al. (författare)
  • Nasal lavage biomarkers : the effect of water damage and microbial growth in an office building
  • 2001
  • Ingår i: Archives of environmental health. - 0003-9896. ; 56:1, s. 30-36
  • Tidskriftsartikel (refereegranskat)abstract
    • Selected nasal symptoms were studied in personnel who worked in a damp office building that had microbial growth (including Stachybotrys sp.) in mineral fiber insulation and gypsum board. There were also signs of dampness in the floor. Clinical examinations included nasal lavage and peak expiratory flow measurements in 12 subjects in the damp building; an additional 8 subjects in a control building (i.e., no signs of dampness or microbial growth) were also examined. Hygienic air measurements of microorganisms and volatile organic compounds were performed in both buildings. The concentrations of eosinophil cationic protein, myeloperoxidase, and albumin, and the number of subjects with eosinophils in lavage fluid, were higher among office workers in the damp building than among controls. The damp biiilding had greater amounts of total molds and bacteria in its construction than the building materials in nondamp buildings. In addition, an increase of 2-ethyl-1-hexanol in the indoor air was detected in the damp building-a sign of dampness-related alkaline degradation of diethyl-hexyl phthalate in polyvinyl chloride floor coatings. In conclusion, the results of this study indicate that exposures in a damp office building may cause an inflammatory nasal mucosal response. The results also support conclusions of earlier studies, indicating that building dampness is related to respiratory inflammation.
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